Staphylococcal scalded skin syndrome - Syndrom Croen Sgaldio Staphylococcal
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Babanod gyda Syndrom Croen Sgaldio Staphylococcal (Staphylococcal scalded skin syndrome)
References
Mae Staphylococcal Scalded Skin Syndrome yn gyflwr lle mae'r croen yn gollwng oherwydd tocsinau a gynhyrchir gan rai mathau o facteria Staphylococcus. Mae'n anghyffredin ymhlith plant dros chwech. Gall hefyd ddigwydd mewn oedolion sydd â systemau imiwnedd gwan neu broblemau difrifol gyda'r arennau. Y brif nodwedd yw pilio croen helaeth yn dilyn llid. Mae difrifoldeb yn amrywio o ychydig o bothelli i golli croen eang, a all achosi cwympiadau difrifol yn nhymheredd y corff ac ansefydlogrwydd mewn pwysedd gwaed.
Staphylococcal Scalded Skin Syndrome is a disease characterized by denudation of the skin caused by exotoxin producing strains of the Staphylococcus species, typically from a distant site. It usually presents 48 hours after birth and is rare in children older than six years. It may also present in immunocompromised adults or those with severe renal disease. The disorder is characterized by significant exfoliation of skin following cellulitis. The severity may vary from a few blisters to system exfoliation leading to marked hypothermia and hemodynamic instability.
Staphylococcal Scalded Skin Syndrome is a disease characterized by denudation of the skin caused by exotoxin producing strains of the Staphylococcus species, typically from a distant site. It usually presents 48 hours after birth and is rare in children older than six years. It may also present in immunocompromised adults or those with severe renal disease. The disorder is characterized by significant exfoliation of skin following cellulitis. The severity may vary from a few blisters to system exfoliation leading to marked hypothermia and hemodynamic instability.
Staphylococcal scalded skin syndrome (SSSS) and bullous impetigo are infections caused by Staphylococcus aureus. Bullous impetigo is due to the local release of these toxins and thus, often presents with localized skin findings, whereas SSSS is from the systemic spread of these toxins, resulting in a more generalized rash and severe presentation. Both conditions are treated with antibiotics that target S. aureus. These conditions can sometimes be confused with other conditions that result in superficial blistering.
Daeth bachgen 10 mis oed i mewn gyda thrwyn yn rhedeg ac nid oedd yn bwyta'n dda. Dywedodd meddygon ei fod yn haint anadlol uwch. Dau ddiwrnod yn ddiweddarach, daeth yn ôl oherwydd nad oedd yn gwella ac roedd ganddo broblemau newydd fel wyneb chwyddedig a chroen llidiog o amgylch y geg. Dros y ddau ddiwrnod nesaf, gwaethygodd y bachgen. Chwyddodd ei freichiau a'i goesau a dechreuodd ei groen ddod i ffwrdd. Yn ôl yn yr ysbyty, sylwodd y meddygon ar frech goch ar ei wyneb ac ym mhlygiadau ei groen, a oedd yn blancio wrth gyffwrdd. Gwnaethant ddiagnosis staphylococcal scalded skin syndrome (SSSS) iddo a'i gychwyn ar wrthfiotigau trwy wythïen.
A 10-month-old male presented with rhinorrhea and decreased oral intake and was diagnosed with an upper respiratory infection. Two days later, he returned to the clinic due to a lack of improvement and the onset of new symptoms, including facial edema and perioral skin irritation. That evening, he became febrile at 100.4 °F and went to the emergency department at the local children's hospital. No further workup was done and the parents were instructed to continue with the current treatment regimen. Over the next 48 hours, the patient's symptoms worsened with the new onset of bilateral extremity edema and desquamation. The patient was returned to the emergency department. A physical exam was notable for a blanching, desquamating, erythematous rash on the face and creases of the arms, legs, and groin. A positive Nikolsky sign was reported. A clinical diagnosis of staphylococcal scalded skin syndrome (SSSS) was made, and the patient was started on intravenous clindamycin. This case illustrates a severe presentation of SSSS in a pediatric patient, demonstrating the challenges it poses to diagnosis and treatment.
A 10-month-old male presented with rhinorrhea and decreased oral intake and was diagnosed with an upper respiratory infection. Two days later, he returned to the clinic due to a lack of improvement and the onset of new symptoms, including facial edema and perioral skin irritation. That evening, he became febrile at 100.4 °F and went to the emergency department at the local children's hospital. No further workup was done and the parents were instructed to continue with the current treatment regimen. Over the next 48 hours, the patient's symptoms worsened with the new onset of bilateral extremity edema and desquamation. The patient was returned to the emergency department. A physical exam was notable for a blanching, desquamating, erythematous rash on the face and creases of the arms, legs, and groin. A positive Nikolsky sign was reported. A clinical diagnosis of staphylococcal scalded skin syndrome (SSSS) was made, and the patient was started on intravenous clindamycin. This case illustrates a severe presentation of SSSS in a pediatric patient, demonstrating the challenges it poses to diagnosis and treatment.
Ymddangosodd merch 2 oed â brech ar hyd ei chorff a oedd wedi datblygu dros 48 awr, yn dilyn brathiadau pryfed ar ei hwyneb y diwrnod cynt. Yn ystod archwiliad, roedd ganddi frech eang yn cynnwys bumps bach a oedd yn uno â'i gilydd, a phan oedd wedi'i rwbio'n ysgafn, roedd ei chroen yn dangos arwyddion o arwydd Nikolsky. Nid oedd unrhyw arwyddion bod y frech yn effeithio ar ei philenni mwcaidd. Ni ddangosodd profion ar ei gwaed unrhyw arwyddion o haint.
A 2-year-old girl presented a generalised rash with 48 h of evolution, in the context of insect bites on the face on the day before. At observation, she had a generalised micropapular rash with confluent areas and Nikolsky sign. There was no mucosal area affected. Blood cultures were negative.
A 2-year-old girl presented a generalised rash with 48 h of evolution, in the context of insect bites on the face on the day before. At observation, she had a generalised micropapular rash with confluent areas and Nikolsky sign. There was no mucosal area affected. Blood cultures were negative.
Mae'r syndrom yn cael ei achosi gan ecsotocsinau epidermolytig (exfoliatin)A a B, sy'n cael eu rhyddhau gan S. awrëws. Mae prognosis syndrom croen sgaldio staphylococcal (staphylococcal scalded skin syndrome) mewn plant yn ardderchog, gyda datrysiad cyflawn o fewn 10 diwrnod o driniaeth, a heb greithiau sylweddol. Fodd bynnag, rhaid gwahaniaethu'n ofalus rhwng syndrom croen sgaldio staphylococcal (staphylococcal scalded skin syndrome) a necrolysis epidermaidd gwenwynig, sy'n dangos prognosis gwael.
○ Diagnosis a Thriniaeth
Mae camddiagnosis fel briwiau ecsema fel dermatitis atopig, a defnyddio eli steroid yn gwaethygu briwiau. Ceisiwch sylw meddygol cyn gynted â phosibl wrth ddefnyddio eli gwrthfiotig.
#Bacitracin
#First-generation cephalosporins (e.g. Cefradine)
#Bacterial culture
#Third-generation cephalosporins (e.g. Cefditoren Pivoxil)