Herpes simplex
☆ Yn 'e 2022 Stiftung Warentest-resultaten út Dútslân wie de konsuminttefredenheid mei ModelDerm mar wat leger dan mei betelle telemedisynkonsultaasjes.
Herpes simplex op 'e lippe.
Herpes simplex ― Herpes simplex-ynfeksje yn fingers komt faker foar by jonge bern dan by folwoeksenen.
References
HSV-1-ynfeksje giet troch primêre ynfeksje fan epitheliale sellen, folge troch latency, benammen yn neuroanen, en reaktivaasje. HSV-1 feroarsaket gewoan inisjele en weromkommende vesikulêre útbarstings, benammen op 'e mûle en genital mucosa. Syn manifestaasjes fariearje fan orolabiale herpes oant ferskate betingsten lykas herpetyske folliculitis, hûdynfeksjes, okulêre belutsenens, en slimme gefallen lykas herpes-encephalitis. Antivirale terapy helpt by it behearen fan HSV-ynfeksje.
Herpes simplex virus type 1 (HSV-1) is a member of the Alphaherpesviridae subfamily. Its structure is composed of linear dsDNA, an icosahedral capsid that is 100 to 110 nm in diameter, with a spikey envelope. In general, the pathogenesis of HSV-1 infection follows a cycle of primary infection of epithelial cells, latency primarily in neurons, and reactivation. HSV-1 is responsible for establishing primary and recurrent vesicular eruptions, primarily in the orolabial and genital mucosa. HSV-1 infection has a wide variety of presentations, including orolabial herpes, herpetic sycosis (HSV folliculitis), herpes gladiatorum, herpetic whitlow, ocular HSV infection, herpes encephalitis, Kaposi varicelliform eruption (eczema herpeticum), and severe or chronic HSV infection. Antiviral therapy limits the course of HSV infection.
Herpes simplex virus type 1 (HSV-1) is a member of the Alphaherpesviridae subfamily. Its structure is composed of linear dsDNA, an icosahedral capsid that is 100 to 110 nm in diameter, with a spikey envelope. In general, the pathogenesis of HSV-1 infection follows a cycle of primary infection of epithelial cells, latency primarily in neurons, and reactivation. HSV-1 is responsible for establishing primary and recurrent vesicular eruptions, primarily in the orolabial and genital mucosa. HSV-1 infection has a wide variety of presentations, including orolabial herpes, herpetic sycosis (HSV folliculitis), herpes gladiatorum, herpetic whitlow, ocular HSV infection, herpes encephalitis, Kaposi varicelliform eruption (eczema herpeticum), and severe or chronic HSV infection. Antiviral therapy limits the course of HSV infection.
Herpes simplex virus type 2 (HSV-2) is in wiidferspraat ynfeksje, beynfloedet sawat 22% fan folwoeksenen fan 12 en âlder, yn totaal 45 miljoen folwoeksenen yn 'e Feriene Steaten. Wylst HSV-1 typysk mûnlinge wûnen feroarsaket, kin it ek liede ta genital-lesjes. As pasjinten lykwols genital-lesjes hawwe, is HSV-2 normaal de wichtichste soarch. De symptomen fan HSV-2-útbraken binne faak vague, lykas genitale jeuk en yrritaasje, dy't diagnoaze en behanneling kinne fertrage. Dizze fertraging kin resultearje yn fierdere oerdracht oan net-ynfekteare persoanen.
Herpes simplex virus type 2 (HSV-2) continues to be a common infection, affecting approximately 22% of adults ages 12 and older, representing 45 million adults in the United States alone. While HSV-1 often affects the perioral region and can be known to cause genital lesions, HSV-2 is more commonly the consideration when patients present with genital lesions. Despite this, most outbreaks of the infection will present with nonspecific symptoms such as genital itching, irritation, and excoriations, which may cause diagnosis and treatment to be delayed. As a result, further exposure to uninfected individuals may occur.
Herpes simplex virus type 2 (HSV-2) continues to be a common infection, affecting approximately 22% of adults ages 12 and older, representing 45 million adults in the United States alone. While HSV-1 often affects the perioral region and can be known to cause genital lesions, HSV-2 is more commonly the consideration when patients present with genital lesions. Despite this, most outbreaks of the infection will present with nonspecific symptoms such as genital itching, irritation, and excoriations, which may cause diagnosis and treatment to be delayed. As a result, further exposure to uninfected individuals may occur.
Herpes simplex virus (HSV) feroarsake faak ynfeksjes lykas genitale herpes en koartswjirren by teeners en folwoeksenen. As HSV in poppe ynfektearret binnen de earste 4-6 wiken fan it libben, kin it liede ta slimme sykte mei serieuze gefolgen. Snelle diagnoaze fan neonatale HSV-ynfeksje is krúsjaal om te stopjen dat de sykte slimmer wurdt, it foarkommen fan neurologyske problemen (sels de dea) .
Herpes simplex virus (HSV), a member of the Herpesviridae family, is a well-known cause of infections including genital herpes and herpes labialis in the adolescent and adult population. Transmission of HSV infection to an infant during the first 4-6 weeks of life can lead to devastating disease with the potential for poor outcomes. Early diagnosis is imperative when evaluating neonatal HSV infection in order to prevent further disease progression, neurological complications, and even death.
Herpes simplex virus (HSV), a member of the Herpesviridae family, is a well-known cause of infections including genital herpes and herpes labialis in the adolescent and adult population. Transmission of HSV infection to an infant during the first 4-6 weeks of life can lead to devastating disease with the potential for poor outcomes. Early diagnosis is imperative when evaluating neonatal HSV infection in order to prevent further disease progression, neurological complications, and even death.
Herpes simplex ynfeksje is heul gewoan en kin wurde trochjûn fan swangere froulju op har poppen. Dit firus kin serieuze sûnensproblemen feroarsaakje of sels de dea by pasgeborenen. Wylst it seldsum is yn 'e swierens sels, bart it faaks by de berte. It risiko is it heechst as de mem yn 'e lettere stadia fan' e swangerskip ynfekteare wurdt. Dit risiko kin lykwols wurde fermindere troch antivirale medisinen te brûken of te kiezen foar in C-seksje yn bepaalde situaasjes.
Infection with herpes simplex is one of the most common sexually transmitted infections. Because the infection is common in women of reproductive age it can be contracted and transmitted to the fetus during pregnancy and the newborn. Herpes simplex virus is an important cause of neonatal infection, which can lead to death or long-term disabilities. Rarely in the uterus, it occurs frequently during the transmission delivery. The greatest risk of transmission to the fetus and the newborn occurs in case of an initial maternal infection contracted in the second half of pregnancy. The risk of transmission of maternal-fetal-neonatal herpes simplex can be decreased by performing a treatment with antiviral drugs or resorting to a caesarean section in some specific cases.
Infection with herpes simplex is one of the most common sexually transmitted infections. Because the infection is common in women of reproductive age it can be contracted and transmitted to the fetus during pregnancy and the newborn. Herpes simplex virus is an important cause of neonatal infection, which can lead to death or long-term disabilities. Rarely in the uterus, it occurs frequently during the transmission delivery. The greatest risk of transmission to the fetus and the newborn occurs in case of an initial maternal infection contracted in the second half of pregnancy. The risk of transmission of maternal-fetal-neonatal herpes simplex can be decreased by performing a treatment with antiviral drugs or resorting to a caesarean section in some specific cases.
Herpes simplex virus (HSV) typen 1 en 2 ynfektearje in protte minsken wrâldwiid. Meastentiids bliuwt it firus rêstich yn senuwsellen nei it ynfektearjen fan 'e hûd, mar it kin letter opnij aktivearje, wêrtroch't kâlde wûnen feroarsaakje. Soms liedt it ta slimme problemen lykas eachynfeksjes, harsensûntstekking, of gefaarlike omstannichheden by pasgeborenen en minsken mei swak ymmúnsysteem. Wylst hjoeddeistige medisinen helpe ynfeksjes te kontrolearjen, bliuwt it risiko fan drugsresistinsje en side-effekten in soarch. Wy hawwe nije medisinen nedich om it firus better te rjochtsjen.
Infection with herpes simplex virus (HSV) types 1 and 2 is ubiquitous in the human population. Most commonly, virus replication is limited to the epithelia and establishes latency in enervating sensory neurons, reactivating periodically to produce localized recurrent lesions. However, these viruses can also cause severe disease such as recurrent keratitis leading potentially to blindness, as well as encephalitis, and systemic disease in neonates and immunocompromised patients. Although antiviral therapy has allowed continual and substantial improvement in the management of both primary and recurrent infections, resistance to currently available drugs and long-term toxicity pose a current and future threat that should be addressed through the development of new antiviral compounds directed against new targets.
Infection with herpes simplex virus (HSV) types 1 and 2 is ubiquitous in the human population. Most commonly, virus replication is limited to the epithelia and establishes latency in enervating sensory neurons, reactivating periodically to produce localized recurrent lesions. However, these viruses can also cause severe disease such as recurrent keratitis leading potentially to blindness, as well as encephalitis, and systemic disease in neonates and immunocompromised patients. Although antiviral therapy has allowed continual and substantial improvement in the management of both primary and recurrent infections, resistance to currently available drugs and long-term toxicity pose a current and future threat that should be addressed through the development of new antiviral compounds directed against new targets.
Der binne twa soarten herpes simplex firus, type 1 (HSV-1) en type 2 (HSV-2). HSV-1 feroarsake faker ynfeksjes om 'e mûle, wylst HSV-2 faker genitale ynfeksjes feroarsaket. Se wurde oerdroegen troch direkt kontakt mei it ynfekteare yndividu. Genital herpes wurdt klassifisearre as in seksueel oerdraachbere ynfeksje. It kin ferspraat wurde nei in poppe by de berte. Nei ynfeksje wurde de firussen lâns sintúchlike nerven ferfierd nei de nerve-sellichems, wêr't se libbenslang wenje. De oarsaken fan werhelling kinne omfetsje: fermindere ymmúnfunksje, stress en bleatstelling oan sinneljocht.
Yn 'e measte gefallen wurde antivirale medisinen allinich nommen as symptomen serieus binne. Deistich antivirale medikaasje kin wurde foarskreaun oan immen dy't hat hiel faak ynfeksje. D'r is gjin faksin beskikber en it faksin foar gordelroos foarkomt herpes simplex net. Behannelingen mei antivirale medikaasje lykas aciclovir of valaciclovir kinne de earnst fan symptomen ferminderje.
Wrâldwiid tariven fan of HSV-1 of HSV-2 binne tusken 60% en 95% yn folwoeksenen. HSV-1 wurdt normaal ynfekteare yn 'e jeugd. Sawat 536 miljoen minsken wrâldwiid (16% fan 'e befolking) waarden ynfekteare mei HSV-2 fanôf 2003 mei gruttere tariven ûnder froulju en dy yn 'e ûntwikkelingswrâld. De measte minsken mei HSV-2 realisearje net dat se ynfekteare binne.
○ Behanneling ― OTC Drugs
Foarkom elk fysyk kontakt, lykas it tútsjen fan in bern, wylst de blieren oanwêzich binne, om't kontakt de ynfeksje kin ferspriede nei oare minsken. Jo moatte rêste sûnder alkohol te drinken.
#Acyclovir cream