Staphylococcal scalded skin syndrome - Staphylococcal Scalded Skin Syndroom
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In berntsje mei Staphylococcal Scalded Skin Syndroom (Staphylococcal scalded skin syndrome)
References
Staphylococcal Scalded Skin Syndrome is in betingst wêryn de hûd ferdwynt troch gifstoffen produsearre troch bepaalde soarten Staphylococcus-baktearjes. It is ûngewoan yn bern oer seis. It kin ek foarkomme yn folwoeksenen mei ferswakke ymmúnsysteem of serieuze nierproblemen. It wichtichste skaaimerk is wiidweidige skin peeling nei ûntstekking. De earnst farieart fan in pear blieren oant wiidferspraat hûdferlies, dat kin slimme drippen yn lichemstemperatuer en instabiliteit yn bloeddruk feroarsaakje.
Staphylococcal Scalded Skin Syndrome is a disease characterized by denudation of the skin caused by exotoxin producing strains of the Staphylococcus species, typically from a distant site. It usually presents 48 hours after birth and is rare in children older than six years. It may also present in immunocompromised adults or those with severe renal disease. The disorder is characterized by significant exfoliation of skin following cellulitis. The severity may vary from a few blisters to system exfoliation leading to marked hypothermia and hemodynamic instability.
Staphylococcal Scalded Skin Syndrome is a disease characterized by denudation of the skin caused by exotoxin producing strains of the Staphylococcus species, typically from a distant site. It usually presents 48 hours after birth and is rare in children older than six years. It may also present in immunocompromised adults or those with severe renal disease. The disorder is characterized by significant exfoliation of skin following cellulitis. The severity may vary from a few blisters to system exfoliation leading to marked hypothermia and hemodynamic instability.
Staphylococcal scalded skin syndrome (SSSS) and bullous impetigo are infections caused by Staphylococcus aureus. Bullous impetigo is due to the local release of these toxins and thus, often presents with localized skin findings, whereas SSSS is from the systemic spread of these toxins, resulting in a more generalized rash and severe presentation. Both conditions are treated with antibiotics that target S. aureus. These conditions can sometimes be confused with other conditions that result in superficial blistering.
In jonge fan 10 moanne kaam binnen mei in rinnende noas en iet net goed. Dokters seine dat it in ynfeksje fan 'e boppeste luchtwegen wie. Twa dagen letter kaam er werom om't er net better waard en nije problemen hie as in swollen gesicht en yrritearre hûd om 'e mûle. De kommende twa dagen waard de jonge slimmer. Syn earms en skonken swollen omheech en syn hûd begon los te kommen. Werom yn it sikehûs fernaam de dokters in reade útslach op syn gesicht en yn 'e plooien fan syn hûd, dy't blanchearre by it oanreitsjen. Se diagnostearre him mei staphylococcal scalded skin syndrome (SSSS) en begûn him op antibiotika troch in ader.
A 10-month-old male presented with rhinorrhea and decreased oral intake and was diagnosed with an upper respiratory infection. Two days later, he returned to the clinic due to a lack of improvement and the onset of new symptoms, including facial edema and perioral skin irritation. That evening, he became febrile at 100.4 °F and went to the emergency department at the local children's hospital. No further workup was done and the parents were instructed to continue with the current treatment regimen. Over the next 48 hours, the patient's symptoms worsened with the new onset of bilateral extremity edema and desquamation. The patient was returned to the emergency department. A physical exam was notable for a blanching, desquamating, erythematous rash on the face and creases of the arms, legs, and groin. A positive Nikolsky sign was reported. A clinical diagnosis of staphylococcal scalded skin syndrome (SSSS) was made, and the patient was started on intravenous clindamycin. This case illustrates a severe presentation of SSSS in a pediatric patient, demonstrating the challenges it poses to diagnosis and treatment.
A 10-month-old male presented with rhinorrhea and decreased oral intake and was diagnosed with an upper respiratory infection. Two days later, he returned to the clinic due to a lack of improvement and the onset of new symptoms, including facial edema and perioral skin irritation. That evening, he became febrile at 100.4 °F and went to the emergency department at the local children's hospital. No further workup was done and the parents were instructed to continue with the current treatment regimen. Over the next 48 hours, the patient's symptoms worsened with the new onset of bilateral extremity edema and desquamation. The patient was returned to the emergency department. A physical exam was notable for a blanching, desquamating, erythematous rash on the face and creases of the arms, legs, and groin. A positive Nikolsky sign was reported. A clinical diagnosis of staphylococcal scalded skin syndrome (SSSS) was made, and the patient was started on intravenous clindamycin. This case illustrates a severe presentation of SSSS in a pediatric patient, demonstrating the challenges it poses to diagnosis and treatment.
In famke fan 2 jier ferskynde mei in útslach oer har hiele lichem dy't mear dan 48 oeren ûntwikkele hie, nei ynsektebeten op har gesicht de dei derfoar. Tidens it ûndersyk hie se in wiidferspraat útslach makke fan lytse bulten dy't gearfoege, en doe't licht wrijven, toande har hûd tekens fan Nikolsky-teken. D'r wiene gjin tekens fan 'e útslach dy't har slijmvliezen beynfloede. Tests op har bloed lieten gjin tekens fan ynfeksje sjen.
A 2-year-old girl presented a generalised rash with 48 h of evolution, in the context of insect bites on the face on the day before. At observation, she had a generalised micropapular rash with confluent areas and Nikolsky sign. There was no mucosal area affected. Blood cultures were negative.
A 2-year-old girl presented a generalised rash with 48 h of evolution, in the context of insect bites on the face on the day before. At observation, she had a generalised micropapular rash with confluent areas and Nikolsky sign. There was no mucosal area affected. Blood cultures were negative.
It syndroom wurdt feroarsake troch epidermolytyske exotoxinen (exfoliatin)A en B, dy't troch S. aureus frijlitten wurde. De prognoaze fan staphylococcal scalded skin syndroom (staphylococcal scalded skin syndrome) by bern is poerbêst, mei folsleine resolúsje binnen 10 dagen fan behanneling, en sûnder signifikante littekens. Lykwols, staphylococcal scalded skin syndroom (staphylococcal scalded skin syndrome) moat foarsichtich ûnderskieden wurde fan toxyske epidermale nekrolyse, dy't in minne prognoaze draacht.
○ Diagnose en behanneling
Misdiagnoaze as eczema-lêsjes lykas atopyske dermatitis, en tapassing fan steroide salve fergruttet leezjes. Sykje asjebleaft medyske oandacht sa gau as mooglik by it oanbringen fan antibiotika-salf.
#Bacitracin
#First-generation cephalosporins (e.g. Cefradine)
#Bacterial culture
#Third-generation cephalosporins (e.g. Cefditoren Pivoxil)