Herpes simplex
☆ A cikin sakamakon Stiftung Warentest na 2022 daga Jamus, gamsuwar mabukaci tare da ModelDerm ya ɗan yi ƙasa kaɗan fiye da biyan shawarwarin telemedicine.
Herpes simplex akan lebe.
Herpes simplex ― Cutar sankarau ta Herpes simplex a cikin yatsu ya fi yawa a kananan yara fiye da manya.
References
HSV-1 kamuwa da cuta yana ci gaba ta hanyar kamuwa da cuta ta farko na sel epithelial, sannan kuma latency, yawanci a cikin neurons, da sake kunnawa. HSV-1 yakan haifar da fashewar vesicular na farko da maimaituwa, galibi akan baki da mucosa na al'aura. Bayyanar cututtuka sun fito ne daga herpes na orolabial zuwa yanayi daban-daban kamar herpetic folliculitis, cututtuka na fata, shigar da ido, da kuma lokuta masu tsanani kamar herpes encephalitis. Maganin rigakafi yana taimakawa sarrafa kamuwa da cutar HSV.
Herpes simplex virus type 1 (HSV-1) is a member of the Alphaherpesviridae subfamily. Its structure is composed of linear dsDNA, an icosahedral capsid that is 100 to 110 nm in diameter, with a spikey envelope. In general, the pathogenesis of HSV-1 infection follows a cycle of primary infection of epithelial cells, latency primarily in neurons, and reactivation. HSV-1 is responsible for establishing primary and recurrent vesicular eruptions, primarily in the orolabial and genital mucosa. HSV-1 infection has a wide variety of presentations, including orolabial herpes, herpetic sycosis (HSV folliculitis), herpes gladiatorum, herpetic whitlow, ocular HSV infection, herpes encephalitis, Kaposi varicelliform eruption (eczema herpeticum), and severe or chronic HSV infection. Antiviral therapy limits the course of HSV infection.
Herpes simplex virus type 1 (HSV-1) is a member of the Alphaherpesviridae subfamily. Its structure is composed of linear dsDNA, an icosahedral capsid that is 100 to 110 nm in diameter, with a spikey envelope. In general, the pathogenesis of HSV-1 infection follows a cycle of primary infection of epithelial cells, latency primarily in neurons, and reactivation. HSV-1 is responsible for establishing primary and recurrent vesicular eruptions, primarily in the orolabial and genital mucosa. HSV-1 infection has a wide variety of presentations, including orolabial herpes, herpetic sycosis (HSV folliculitis), herpes gladiatorum, herpetic whitlow, ocular HSV infection, herpes encephalitis, Kaposi varicelliform eruption (eczema herpeticum), and severe or chronic HSV infection. Antiviral therapy limits the course of HSV infection.
Herpes simplex virus type 2 (HSV-2) cuta ce mai yaduwa, tana shafar kusan kashi 22% na manya masu shekaru 12 zuwa sama, jimlar manya miliyan 45 a Amurka. Yayin da HSV-1 yakan haifar da ciwon baki, yana iya haifar da raunuka na al'aura. Duk da haka, lokacin da marasa lafiya suna da raunin al'aura, HSV-2 yawanci shine babban damuwa. Alamun cututtukan HSV-2 sau da yawa ba su da tabbas, kamar itching da haushi, wanda zai iya jinkirta ganewar asali da magani. Wannan jinkirin na iya haifar da ƙarin watsawa ga mutanen da basu kamu da cutar ba.
Herpes simplex virus type 2 (HSV-2) continues to be a common infection, affecting approximately 22% of adults ages 12 and older, representing 45 million adults in the United States alone. While HSV-1 often affects the perioral region and can be known to cause genital lesions, HSV-2 is more commonly the consideration when patients present with genital lesions. Despite this, most outbreaks of the infection will present with nonspecific symptoms such as genital itching, irritation, and excoriations, which may cause diagnosis and treatment to be delayed. As a result, further exposure to uninfected individuals may occur.
Herpes simplex virus type 2 (HSV-2) continues to be a common infection, affecting approximately 22% of adults ages 12 and older, representing 45 million adults in the United States alone. While HSV-1 often affects the perioral region and can be known to cause genital lesions, HSV-2 is more commonly the consideration when patients present with genital lesions. Despite this, most outbreaks of the infection will present with nonspecific symptoms such as genital itching, irritation, and excoriations, which may cause diagnosis and treatment to be delayed. As a result, further exposure to uninfected individuals may occur.
Herpes simplex virus (HSV) yawanci yana haifar da cututtuka kamar cututtukan al'aura da ciwon sanyi ga matasa da manya. Lokacin da HSV ya cutar da jariri a cikin makonni 4-6 na farko na rayuwa, zai iya haifar da rashin lafiya mai tsanani tare da sakamako mai tsanani. Gaggawa da sauri gano kamuwa da cutar HSV na jarirai yana da mahimmanci don dakatar da cutar daga yin muni, hana al'amuran jijiyoyin jiki (har ma da mutuwa) .
Herpes simplex virus (HSV), a member of the Herpesviridae family, is a well-known cause of infections including genital herpes and herpes labialis in the adolescent and adult population. Transmission of HSV infection to an infant during the first 4-6 weeks of life can lead to devastating disease with the potential for poor outcomes. Early diagnosis is imperative when evaluating neonatal HSV infection in order to prevent further disease progression, neurological complications, and even death.
Herpes simplex virus (HSV), a member of the Herpesviridae family, is a well-known cause of infections including genital herpes and herpes labialis in the adolescent and adult population. Transmission of HSV infection to an infant during the first 4-6 weeks of life can lead to devastating disease with the potential for poor outcomes. Early diagnosis is imperative when evaluating neonatal HSV infection in order to prevent further disease progression, neurological complications, and even death.
Herpes simplex kamuwa da cuta yana da yawa kuma yana iya kamuwa da mata masu juna biyu zuwa jariransu. Wannan ƙwayar cuta na iya haifar da mummunan lamuran lafiya ko ma mutuwa a cikin jarirai. Duk da yake yana da wuya a lokacin daukar ciki kanta, yakan faru a lokacin haihuwa. Haɗarin ya fi girma idan mahaifiyar ta kamu da cutar a cikin matakai na gaba na ciki. Duk da haka, ana iya rage wannan haɗarin ta hanyar amfani da magungunan rigakafi ko zaɓin sashin C a wasu yanayi.
Infection with herpes simplex is one of the most common sexually transmitted infections. Because the infection is common in women of reproductive age it can be contracted and transmitted to the fetus during pregnancy and the newborn. Herpes simplex virus is an important cause of neonatal infection, which can lead to death or long-term disabilities. Rarely in the uterus, it occurs frequently during the transmission delivery. The greatest risk of transmission to the fetus and the newborn occurs in case of an initial maternal infection contracted in the second half of pregnancy. The risk of transmission of maternal-fetal-neonatal herpes simplex can be decreased by performing a treatment with antiviral drugs or resorting to a caesarean section in some specific cases.
Infection with herpes simplex is one of the most common sexually transmitted infections. Because the infection is common in women of reproductive age it can be contracted and transmitted to the fetus during pregnancy and the newborn. Herpes simplex virus is an important cause of neonatal infection, which can lead to death or long-term disabilities. Rarely in the uterus, it occurs frequently during the transmission delivery. The greatest risk of transmission to the fetus and the newborn occurs in case of an initial maternal infection contracted in the second half of pregnancy. The risk of transmission of maternal-fetal-neonatal herpes simplex can be decreased by performing a treatment with antiviral drugs or resorting to a caesarean section in some specific cases.
Herpes simplex virus (HSV) nau'in 1 da 2 sun kamu da mutane da yawa a duniya. Yawanci, kwayar cutar takan zauna shiru a cikin sel jijiya bayan ta cutar da fata, amma tana iya sake kunnawa daga baya, ta haifar da mura. Wani lokaci, yana haifar da matsaloli masu tsanani kamar ciwon ido, kumburin kwakwalwa, ko yanayi masu haɗari a cikin jarirai da mutanen da ke da raunin tsarin rigakafi. Duk da yake magungunan na yanzu suna taimakawa wajen sarrafa cututtuka, haɗarin juriya na miyagun ƙwayoyi da illar illa ya kasance abin damuwa. Muna buƙatar sabbin magunguna don cutar da kwayar cutar da kyau.
Infection with herpes simplex virus (HSV) types 1 and 2 is ubiquitous in the human population. Most commonly, virus replication is limited to the epithelia and establishes latency in enervating sensory neurons, reactivating periodically to produce localized recurrent lesions. However, these viruses can also cause severe disease such as recurrent keratitis leading potentially to blindness, as well as encephalitis, and systemic disease in neonates and immunocompromised patients. Although antiviral therapy has allowed continual and substantial improvement in the management of both primary and recurrent infections, resistance to currently available drugs and long-term toxicity pose a current and future threat that should be addressed through the development of new antiviral compounds directed against new targets.
Infection with herpes simplex virus (HSV) types 1 and 2 is ubiquitous in the human population. Most commonly, virus replication is limited to the epithelia and establishes latency in enervating sensory neurons, reactivating periodically to produce localized recurrent lesions. However, these viruses can also cause severe disease such as recurrent keratitis leading potentially to blindness, as well as encephalitis, and systemic disease in neonates and immunocompromised patients. Although antiviral therapy has allowed continual and substantial improvement in the management of both primary and recurrent infections, resistance to currently available drugs and long-term toxicity pose a current and future threat that should be addressed through the development of new antiviral compounds directed against new targets.
Akwai nau'i biyu na cutar herpes simplex , nau'in 1 (HSV-1) da nau'in 2 (HSV-2). HSV-1 ya fi haifar da cututtuka a kusa da baki yayin da HSV-2 ya fi haifar da cututtuka na al'ada. Ana yada su ta hanyar hulɗa kai tsaye tare da wanda ya kamu da cutar. An rarraba herpes a cikin al'aura azaman kamuwa da cuta ta hanyar jima'i. Ana iya yada shi ga jariri yayin haihuwa. Bayan kamuwa da cuta, ana jigilar ƙwayoyin cuta tare da jijiyoyi masu azanci zuwa ga jikin kwayoyin halitta, inda suke rayuwa tsawon rai. Dalilan sake dawowa na iya haɗawa da: raguwar aikin rigakafi, damuwa, da hasken rana.
A mafi yawan lokuta, ana shan magungunan antiviral ne kawai lokacin da alamun cututtuka suka yi tsanani. Za a iya rubuta magungunan rigakafin yau da kullun ga wanda ke da kamuwa da cuta akai-akai. Babu maganin rigakafi da ake samu kuma maganin shingles baya hana herpes simplex. Jiyya tare da maganin rigakafi kamar aciclovir ko valaciclovir na iya rage girman alamun bayyanar cututtuka.
Ƙididdigar duniya na ko dai HSV-1 ko HSV-2 suna tsakanin 60% da 95% a cikin manya. HSV-1 yawanci ana kamuwa da ita a lokacin ƙuruciya. Kimanin mutane miliyan 536 a duk duniya (16% na yawan jama'a) sun kamu da HSV-2 kamar na 2003 tare da mafi girma tsakanin mata da waɗanda ke cikin ƙasashe masu tasowa. Yawancin mutanen da ke da HSV-2 ba su gane cewa sun kamu da cutar ba.
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