Karin bayani ― Hausa

Of the 61 patients studied, hypersensitivity vasculitis (HSV) [23 (37.7%)] and Henoch Schonlein purpura (HSP) [16 (26.2%)] were the two most common forms. Systemic involvement was seen in 32 (52.45%) patients. Drugs were implicated in 12 (19.7%) cases, infections in 7 (11.4%) and connective tissue disorders in 4 (6.5%) cases. No association was seen between history of drug intake and tissue eosinophilia and also between histologically severe vasculitis and clinical severity.

Leukocytoclastic vasculitis wani nau'in kumburin fata ne da ke shafar kananan magudanar jini a cikin zurfin fatar jiki. Yana iya faruwa ba tare da sanin dalili ba ko kuma a haɗa shi da cututtuka, ciwace-ciwace, cututtuka na autoimmune, ko magunguna. Alamu na yau da kullun sun haɗa da tabo ja ko shunayya a ƙafafu, shigar da ƙananan jiragen ruwa, kuma a cikin kusan kashi 30 na lokuta, sauran sassan jiki ma suna shafar. Yawancin lokuta suna bayyana da kansu a cikin 'yan makonni zuwa watanni. Magani ya bambanta dangane da yadda yake da tsanani, daga sannu a hankali rage corticosteroids na baka zuwa amfani da wasu magungunan da ke rage kumburi ba tare da steroids ba.
Leukocytoclastic vasculitis is a cutaneous, small-vessel vasculitis of the dermal capillaries and venules. This condition can be idiopathic or can be associated with infections, neoplasms, autoimmune disorders, and drugs. Key clinical features of leukocytoclastic vasculitis include palpable purpura on the lower extremity, small vessel involvement, and, in about 30 percent of individuals, extracutaneous involvement. Most cases of idiopathic cutaneous, small vessel vasculitis are self-limited with 90 percent of cases resolving in weeks to months of onset. Otherwise, treatment depends on the severity of disease and can range from an oral corticosteroid taper to various steroid-sparing immunosuppressive agents.