Postinflammatory hyperpigmentation
Postinflammatory hyperpigmentation je stanje kože koje karakterizira povećani pigment u koži nakon upale kože. Postinflammatory hyperpigmentation može biti uzrokovana produljenim izlaganjem suncu, upalom ili drugim ozljedama kože, uključujući one povezane s aknama. Ljudi s tamnijim tonovima kože obično su skloniji hiperpigmentaciji zbog pretjeranog izlaganja suncu.
☆ U rezultatima Stiftung Warentest iz Njemačke za 2022., zadovoljstvo potrošača s ModelDermom bilo je samo malo niže nego s plaćenim konzultacijama o telemedicini.
References
Postinflammatory hyperpigmentation (PIH) je čest kožni problem koji se javlja nakon upale ili ozljede kože. Obično traje dugo i gore je kod ljudi tamnije kože (Fitzpatrick skin types III–VI) . Iako često bude bolje samo od sebe, to može potrajati, pa je često potrebno dugotrajno liječenje. Kombinacija različitih tretmana najbolje djeluje.
Postinflammatory hyperpigmentation (PIH) is a common acquired cutaneous disorder occurring after skin inflammation or injury. It is chronic and is more common and severe in darker-skinned individuals (Fitzpatrick skin types III–VI). While the condition typically improves spontaneously, this process can take months to years, necessitating prolonged treatment. Combination therapy is the most effective.
Postinflammatory hyperpigmentation (PIH) is a common acquired cutaneous disorder occurring after skin inflammation or injury. It is chronic and is more common and severe in darker-skinned individuals (Fitzpatrick skin types III–VI). While the condition typically improves spontaneously, this process can take months to years, necessitating prolonged treatment. Combination therapy is the most effective.
Postinflammatory hyperpigmentation je uobičajena posljedica upale kože. Ima tendenciju da ozbiljnije i češće utječe na osobe tamnije puti. Studije pokazuju da su problemi poput postinflammatory hyperpigmentation među glavnim razlozima zašto ljudi tamnije kože traže dermatološku njegu. Rano liječenje ključno je za rješavanje postinflammatory hyperpigmentation i obično počinje svladavanjem početnog upalnog stanja. Prva linija liječenja obično uključuje korištenje lokalnih sredstava koja posvjetljuju kožu zajedno s kremom za sunčanje za zaštitu. Ova sredstva, poput hydroquinone, azelaic acid, kojic acid, arbutin, licorice extracts , mogu učinkovito smanjiti pretjeranu pigmentaciju. Osim toga, retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, soy se također koriste kao sredstva za depigmentaciju, s pojavom novih tretmana. Dok su lokalni tretmani obično učinkoviti za hiperpigmentaciju na površinskoj razini, postupci (laser, chemical peel) mogu biti potrebni za tvrdokorne slučajeve. Važno je biti oprezan s ovim tretmanima kako biste izbjegli iritaciju i pogoršanje postinflammatory hyperpigmentation.
Postinflammatory hyperpigmentation is a common sequelae of inflammatory dermatoses that tends to affect darker skinned patients with greater frequency and severity. Epidemiological studies show that dyschromias, including postinflammatory hyperpigmentation, are among the most common reasons darker racial/ethnic groups seek the care of a dermatologist. The treatment of postinflammatory hyperpigmentation should be started early to help hasten its resolution and begins with management of the initial inflammatory condition. First-line therapy typically consists of topical depigmenting agents in addition to photoprotection including a sunscreen. Topical tyrosinase inhibitors, such as hydroquinone, azelaic acid, kojic acid, arbutin, and certain licorice extracts, can effectively lighten areas of hypermelanosis. Other depigmenting agents include retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, and soy with a number of emerging therapies on the horizon. Topical therapy is typically effective for epidermal postinflammatory hyperpigmentation; however, certain procedures, such as chemical peeling and laser therapy, may help treat recalcitrant hyperpigmentation. It is also important to use caution with all of the above treatments to prevent irritation and worsening of postinflammatory hyperpigmentation.
Postinflammatory hyperpigmentation is a common sequelae of inflammatory dermatoses that tends to affect darker skinned patients with greater frequency and severity. Epidemiological studies show that dyschromias, including postinflammatory hyperpigmentation, are among the most common reasons darker racial/ethnic groups seek the care of a dermatologist. The treatment of postinflammatory hyperpigmentation should be started early to help hasten its resolution and begins with management of the initial inflammatory condition. First-line therapy typically consists of topical depigmenting agents in addition to photoprotection including a sunscreen. Topical tyrosinase inhibitors, such as hydroquinone, azelaic acid, kojic acid, arbutin, and certain licorice extracts, can effectively lighten areas of hypermelanosis. Other depigmenting agents include retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, and soy with a number of emerging therapies on the horizon. Topical therapy is typically effective for epidermal postinflammatory hyperpigmentation; however, certain procedures, such as chemical peeling and laser therapy, may help treat recalcitrant hyperpigmentation. It is also important to use caution with all of the above treatments to prevent irritation and worsening of postinflammatory hyperpigmentation.
