Amyloidosis - Amyloidoz
☆ AI Dermatology — Free Service
Nan rezilta Stiftung Warentest 2022 ki soti nan Almay, satisfaksyon konsomatè yo ak ModelDerm te sèlman yon ti kras pi ba pase ak konsiltasyon telemedsin peye.
Nan rezilta Stiftung Warentest 2022 ki soti nan Almay, satisfaksyon konsomatè yo ak ModelDerm te sèlman yon ti kras pi ba pase ak konsiltasyon telemedsin peye.
Karakteristik fasial klasik nan Amyloidoz (Amyloidosis)
relevance score : -100.0%
ReferencesYon fanm 26 an te vin nan klinik nou an pou li plenyen sou yon gratèl sou janm li ki te prezan depi 10 ane. Malgre li te itilize krèm esteroyid ak krèm tazarotèn, gratèl la pa t vin pi bon. Li pa t gen okenn istwa fanmi ki enpòtan. Lè nou te egzamine li, nou te jwenn tach ki leve sou devan janm li, ki koresponn ak yon maladi ki rele lichen amyloidosis.
A 26-year-old woman presented to our clinic with an itchy rash on her legs that had persisted for 10 years. The rash had previously been treated with topical steroids and tazarotene cream, with no improvement. The patient’s family history was noncontributory. A physical examination showed discrete and coalescing hyperkeratotic tan-brown papules on the pretibial surfaces, consistent with lichen amyloidosis.
A 26-year-old woman presented to our clinic with an itchy rash on her legs that had persisted for 10 years. The rash had previously been treated with topical steroids and tazarotene cream, with no improvement. The patient’s family history was noncontributory. A physical examination showed discrete and coalescing hyperkeratotic tan-brown papules on the pretibial surfaces, consistent with lichen amyloidosis.
Lichen Amyloidosis se yon maladi po ki ra, ki lye ak demanjezon ki pèsistan epi ki gen kòz enkoni. Li souvan parèt kòm ti grenn ki leve, plak ki dekolore sou sifas po a. Lichen Amyloidosis anjeneral parèt lakay moun ki gen laj ant 50 ak 60 ane, e malerezman pa gen okenn remèd pou li. Tretman ki disponib kounye a pa souvan efikas.
Lichen Amyloidosis (LA) is an uncommon, primary cutaneous amyloidosis associated with chronic, idiopathic pruritus. Clinical presentation of LA includes skin colored to hyperpigmented, papules coalescing into plaques with a rippled appearance on the extensors.1 LA most commonly presents in the fifth to sixth decade of life and has no curative treatments. Overall response to current therapies is poor.
Lichen Amyloidosis (LA) is an uncommon, primary cutaneous amyloidosis associated with chronic, idiopathic pruritus. Clinical presentation of LA includes skin colored to hyperpigmented, papules coalescing into plaques with a rippled appearance on the extensors.1 LA most commonly presents in the fifth to sixth decade of life and has no curative treatments. Overall response to current therapies is poor.
Lichen amyloidosis se yon kondisyon po ki grate epi ki dire lontan. Li karakterize pa aparisyon boul epè sou do, tib, kwis, ak bra. Lè yo egzamine po a anba mikwoskòp, Lichen amyloidosis montre akimilasyon amiloid nan kouch siperyè po a ansanm ak epesman ak elajisman kouch po ekstèn lan. Menmsi kòz egzak Lichen amyloidosis poko fin konprann, etid anvan yo te lye li ak faktè tankou fwotman oswa friksyon sou po a, lanmò selil, enfeksyon viral, pami lòt faktè. Lichen amyloidosis sanble lye ak plizyè lòt kondisyon po (atopic dermatitis, lichen planus, mycosis fungoides).
Lichen amyloidosis (LA) is a chronic pruritic skin disorder characterized by multiple grouped hyperkeratotic papules, predominantly located on the back, shins, thighs, and arms. Histological analysis of LA shows amyloid deposition in the papillary dermis and hyperkeratosis and acanthosis of the epidermis. The exact pathogenesis of LA has not yet been elucidated; however, prior reports have implicated frictional epidermal damage, apoptosis, viral infection, and many other triggers. LA is reportedly associated with several skin disorders, including atopic dermatitis (AD), lichen planus, and mycosis fungoides.
Lichen amyloidosis (LA) is a chronic pruritic skin disorder characterized by multiple grouped hyperkeratotic papules, predominantly located on the back, shins, thighs, and arms. Histological analysis of LA shows amyloid deposition in the papillary dermis and hyperkeratosis and acanthosis of the epidermis. The exact pathogenesis of LA has not yet been elucidated; however, prior reports have implicated frictional epidermal damage, apoptosis, viral infection, and many other triggers. LA is reportedly associated with several skin disorders, including atopic dermatitis (AD), lichen planus, and mycosis fungoides.
○ Dyagnostik ak Tretman
#Electrophoresis of blood or urine
#Skin biopsy