Impetigo - Enpètigohttps://en.wikipedia.org/wiki/Impetigo
Enpètigo (Impetigo) se yon enfeksyon bakteri ki afekte po sipèfisyèl la. Prezantasyon ki pi komen se kwout jòn sou figi, bra, oswa janm. Blesi yo ka douloure oswa grate, men lafyèv pa komen.

enpètigo (impetigo) anjeneral se akòz Staphylococcus aureus oswa Streptococcus pyogenes. Li ka gaye pa kontak dirèk ant moun, espesyalman ant timoun, frè ak sè.

Tretman an tipikman se krèm antibyotik tankou mupirocin oswa asid fusidik. Antibyotik oral, tankou cefalexin, ka itilize si gwo zòn afekte.

enpètigo (impetigo) te afekte anviwon 140 milyon moun (2 % popilasyon mondyal la) an 2010. Li ka rive nenpòt laj, men li pi komen nan timoun piti. Konplikasyon yo ka gen ladan selulit oswa glomerulonefrit poststreptococcal.

Tretman - Medikaman OTC
* Paske enpètigo se yon maladi enfektye, esteroyid pa dwe itilize. Si ou pa sèten pou distenge blesi enpètigo ak ekzema, pran yon antihistamin OTC san w pa itilize esteroyid.
#OTC antihistamine

* Tanpri aplike antibyotik OTC sou blesi a.
#Bacitracin
#Polysporin
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  • Yon ka enpètigo sou manton an. Enpètigo ta dwe sispèk si yon timoun piti pa gen okenn istwa blesi, men blesi yo gaye.
  • Li sipoze se yon enfeksyon segondè ki rive sou pasyan ki gen dèrmatoz atopik.
  • Kontrèman ak dermatit atopik, enpètigo mande tretman antibyotik epi li ka vin pi mal ak itilizasyon estewoyid.
  • Imaj la montre aparans apre ti anpoul bullous impetigo yo te pete.
  • Li ka mal dyagnostike kòm dèrmatoz atopik.
  • Bullous impetigo ― Lè li prezante ak ti anpoul mens, frajil, yo dyagnostike li kòm bullous impetigo.
References Impetigo: Diagnosis and Treatment 25250996
Impetigo, enfeksyon po bakteri ki pi komen nan timoun ki gen laj ant 0 ak 5 an, vini nan de kalite prensipal: non‑bullous (70 % ka) ak bullous (30 % ka). Staphylococcus aureus oswa Streptococcus pyogenes se kòz Impetigo ki pa bullous. Li karakterize pa kouch ki gen koulè siwo myèl sou figi a ak sou branch yo, epi li souvan vize po ki gen ekzem, mòde ensèk, oswa blesi èpètik. Impetigo bullous, ki koze sèlman pa S. aureus, pwodui gwo blòk likid (bullae) epi souvan afekte zòn kote po fwote ansanm. Tou de kalite anjeneral geri nan de a twa semèn san sikatriz; konplikasyon yo ra, eksepte glomerulonefrit poststreptococcal ki pi grav. Tretman an gen ladan antibyotik topikal (mupirocin, retapamulin, fusidic acid). Antibyotik oral ka nesesè pou Impetigo ak gwo bullae oswa lè tretman topikal pa posib. Plizyè antibyotik oral (amoxicillin/clavulanate, dicloxacillin, cephalexin, clindamycin, doxycycline, minocycline, trimethoprim/sulfamethoxazole, macrolides) se opsyon; penicilin pa efikas. Dezinfektan topikal pa tèlman efikas ke antibyotik epi yo dwe evite. Fusidic acid, mupirocin, retapamulin efikas kont methicillin‑sansib S. aureus ak enfeksyon streptokok. Clindamycin itil pou sispèk enfeksyon methicillin‑resistant S. aureus. Trimethoprim/sulfamethoxazole travay kont S. aureus ki reziste methicillin, men li pa ase pou enfeksyon streptokok.
Impetigo, the most common bacterial skin infection in children aged two to five, comes in two main types: nonbullous (70% of cases) and bullous (30% of cases). Nonbullous impetigo is typically caused by Staphylococcus aureus or Streptococcus pyogenes. It's recognized by honey-colored crusts on the face and limbs and mainly targets the skin or can infect insect bites, eczema, or herpetic lesions. Bullous impetigo, caused solely by S. aureus, leads to large, flaccid bullae and often affects areas where skin rubs together. Both types usually clear up within two to three weeks without scarring, and complications are rare, with poststreptococcal glomerulonephritis being the most severe. Treatment involves topical antibiotics (mupirocin, retapamulin, fusidic acid). Oral antibiotics might be necessary for impetigo with large bullae or when topical treatment isn't feasible. While several oral antibiotics (amoxicillin/clavulanate, dicloxacillin, cephalexin, clindamycin, doxycycline, minocycline, trimethoprim/sulfamethoxazole, macrolides) are options, penicillin isn't effective. Topical disinfectants aren't as good as antibiotics and should be avoided. Fusidic acid, mupirocin, retapamulin are effective against methicillin-susceptible S. aureus and streptococcal infections. Clindamycin is useful for suspected methicillin-resistant S. aureus infections. Trimethoprim/sulfamethoxazole works against methicillin-resistant S. aureus, but isn't enough for streptococcal infection.
 Impetigo 28613693 
NIH
Impetigo se yon enfeksyon po ki komen, ki koze pa sèten bakteri epi ki fasil gaye atravè kontak. Li souvan parèt kòm plak wouj ki kouvri ak yon kwout jòn, epi li ka lakòz demanjezon oswa doulè. Enfeksyon sa a pi souvan rive lakay timoun ki viv nan zòn cho ak imid. Li ka manifeste kòm ti anpoul oswa krust. Pandan ke li souvan afekte figi a, li ka parèt nenpòt kote ki gen po ki domaje. Dyagnostik la depann sitou sou sentòm yo ak aparans li. Tretman anjeneral gen ladan antibyotik, tou de topikal ak oral, ansanm ak jesyon sentòm yo.
Impetigo is a common infection of the superficial layers of the epidermis that is highly contagious and most commonly caused by gram-positive bacteria. It most commonly presents as erythematous plaques with a yellow crust and may be itchy or painful. The lesions are highly contagious and spread easily. Impetigo is a disease of children who reside in hot humid climates. The infection may be bullous or nonbullous. The infection typically affects the face but can also occur in any other part of the body that has an abrasion, laceration, insect bite or other trauma. Diagnosis is typically based on the symptoms and clinical manifestations alone. Treatment involves topical and oral antibiotics and symptomatic care.