Plis enfomasyon ― Kreyòl ayisyen

Impetigo , enfeksyon po bakteri ki pi komen nan timoun ki gen laj de a senk an, vini nan de kalite prensipal: non-bullou (70% nan ka) ak boulous (30% nan ka) . Se Staphylococcus aureus oswa Streptococcus pyogenes ki lakòz enpetigo ki pa boulous. Li rekonèt pa kwout ki gen koulè siwo myèl sou figi a ak branch yo epi sitou vize po a oswa li ka enfekte mòde ensèk, ekzema, oswa blesi èpètik. Enpetigo boulous, ki te koze sèlman pa S. Aureus, mennen nan gwo, flask bullae e souvan afekte zòn kote po fwote ansanm. Tou de kalite anjeneral klè nan de a twa semèn san yo pa sikatris, ak konplikasyon yo ra, ak glomerulonefrit poststreptococcal ki pi grav la. Tretman enplike antibyotik aktualite (mupirocin, retapamulin, fusidic acid) . Antibyotik oral ta ka nesesè pou enpètigo ak gwo bullae oswa lè tretman aktualite pa posib. Pandan ke plizyè antibyotik oral (amoxicillin/clavulanate, dicloxacillin, cephalexin, clindamycin, doxycycline, minocycline, trimethoprim/sulfamethoxazole, macrolides) se opsyon, penisilin pa efikas. Dezenfektan aktualite yo pa osi bon ke antibyotik epi yo ta dwe evite. Fusidic acid, mupirocin, retapamulin yo efikas kont methicillin-sansib S. Aureus ak enfeksyon strèptokok. Clindamycin se itil pou sispèk methicillin-resistant S. Aureus enfeksyon. Trimethoprim/sulfamethoxazole travay kont S. Aureus ki reziste methicillin, men li pa ase pou enfeksyon strèptokok.
Impetigo, the most common bacterial skin infection in children aged two to five, comes in two main types: nonbullous (70% of cases) and bullous (30% of cases). Nonbullous impetigo is typically caused by Staphylococcus aureus or Streptococcus pyogenes. It's recognized by honey-colored crusts on the face and limbs and mainly targets the skin or can infect insect bites, eczema, or herpetic lesions. Bullous impetigo, caused solely by S. aureus, leads to large, flaccid bullae and often affects areas where skin rubs together. Both types usually clear up within two to three weeks without scarring, and complications are rare, with poststreptococcal glomerulonephritis being the most severe. Treatment involves topical antibiotics (mupirocin, retapamulin, fusidic acid). Oral antibiotics might be necessary for impetigo with large bullae or when topical treatment isn't feasible. While several oral antibiotics (amoxicillin/clavulanate, dicloxacillin, cephalexin, clindamycin, doxycycline, minocycline, trimethoprim/sulfamethoxazole, macrolides) are options, penicillin isn't effective. Topical disinfectants aren't as good as antibiotics and should be avoided. Fusidic acid, mupirocin, retapamulin are effective against methicillin-susceptible S. aureus and streptococcal infections. Clindamycin is useful for suspected methicillin-resistant S. aureus infections. Trimethoprim/sulfamethoxazole works against methicillin-resistant S. aureus, but isn't enough for streptococcal infection.

Impetigo se yon enfeksyon po komen ki koze pa sèten bakteri, fasil gaye nan kontak. Li anjeneral parèt tankou plak wouj ki kouvri ak yon kwout jòn epi li ka lakòz demanjezon oswa doulè. Enfeksyon sa a pi komen nan timoun k ap viv nan zòn cho ak imid. Li ka parèt tankou ti anpoul oswa san yo. Pandan ke li souvan afekte figi a, li ka rive nenpòt kote gen yon repo nan po a. Dyagnostik sitou depann sou sentòm yo ak fason li sanble. Tretman anjeneral gen ladan antibyotik, tou de aktualite ak oral, ansanm ak jesyon sentòm yo.
Impetigo is a common infection of the superficial layers of the epidermis that is highly contagious and most commonly caused by gram-positive bacteria. It most commonly presents as erythematous plaques with a yellow crust and may be itchy or painful. The lesions are highly contagious and spread easily. Impetigo is a disease of children who reside in hot humid climates. The infection may be bullous or nonbullous. The infection typically affects the face but can also occur in any other part of the body that has an abrasion, laceration, insect bite or other trauma. Diagnosis is typically based on the symptoms and clinical manifestations alone. Treatment involves topical and oral antibiotics and symptomatic care.