Keloid
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Yon keloid ki te parèt apre operasyon sou ponyèt la te trete ak piki intralesional triamcinolone. Zòn ki gen eritem sou bò gòch la se zòn ki te trete.
Keloid lineyè. Lè yo rive sou devan anwo nan tò a, yo souvan parèt kòm yon fòm lineyè.
Posyèr aurikilè Keloid
Keloid sou plant pye yo ka fè mache alèz. Piki esteroyid intralesional yo anjeneral fèt plizyè fwa.
Keloid yo souvan obsève sou bra anwo yo.
Manifestasyon tipik keloid sou pwatrin.
relevance score : -100.0%
ReferencesKeloid yo fòme akoz gerizon ki pa nòmal apre blesi po oswa enflamasyon. Faktè jenetik ak anviwònman kontribye nan devlopman yo, epi pi gwo pousantaj la obsève lakay moun ki gen po nwa ki gen desandan Afriken, Azyatik oswa Panyòl. Keloid rive lè fibroblast yo vin twò aktif, yo pwodui twòp kolagen ak faktè kwasans. Sa mennen a fòmasyon gwo pakèt kolagen ki pa nòmal, yo rele kolagen keloidal, ansanm ak yon ogmantasyon fibroblast. Klinikman, keloid parèt kòm nòd fòm kawoutchou sou zòn ki te blese anvan. Kontrèman ak blesi nòmal, keloid pwolonje pi lwen pase limit orijinal la. Pasyan yo ka santi doulè, gratèl, oswa boule. Gen plizyè tretman ki disponib, tankou piki steroid, cryotherapy, operasyon, radiotherapy, ak laser therapy.
Keloids result from abnormal wound healing in response to skin trauma or inflammation. Keloid development rests on genetic and environmental factors. Higher incidences are seen in darker skinned individuals of African, Asian, and Hispanic descent. Overactive fibroblasts producing high amounts of collagen and growth factors are implicated in the pathogenesis of keloids. As a result, classic histologic findings demonstrate large, abnormal, hyalinized bundles of collagen referred to as keloidal collagen and numerous fibroblasts. Keloids present clinically as firm, rubbery nodules in an area of prior injury to the skin. In contrast to normal or hypertrophic scars, keloidal tissue extends beyond the initial site of trauma. Patients may complain of pain, itching, or burning. Multiple treatment modalities exist although none are uniformly successful. The most common treatments include intralesional or topical steroids, cryotherapy, surgical excision, radiotherapy, and laser therapy.
Keloids result from abnormal wound healing in response to skin trauma or inflammation. Keloid development rests on genetic and environmental factors. Higher incidences are seen in darker skinned individuals of African, Asian, and Hispanic descent. Overactive fibroblasts producing high amounts of collagen and growth factors are implicated in the pathogenesis of keloids. As a result, classic histologic findings demonstrate large, abnormal, hyalinized bundles of collagen referred to as keloidal collagen and numerous fibroblasts. Keloids present clinically as firm, rubbery nodules in an area of prior injury to the skin. In contrast to normal or hypertrophic scars, keloidal tissue extends beyond the initial site of trauma. Patients may complain of pain, itching, or burning. Multiple treatment modalities exist although none are uniformly successful. The most common treatments include intralesional or topical steroids, cryotherapy, surgical excision, radiotherapy, and laser therapy.
Rechèch aktyèl la sijere ke jèl silikon oswa fèy ansanm ak piki kortikoterapi se premye tretman ki pi pito pou keloid. Lòt tretman tankou intralesyonèl 5-fluorouracil (5-FU), bleomycin (bleomicin) oswa verapamil ka konsidere tou, byenke efikasite yo varye. Terapi lazè, lè li konbine avèk piki kortikoterapi oswa estewoyid ki aplike anba oklizyon, ka amelyore pénétrasyon dwòg la. Pou keloid ki rekòmanse, retire chirijikal ki swiv pa terapi radyasyon imedyat te montre efikas. Finalman, lè yo itilize jèl silikon oswa fèy ak terapi presyon, sa pwouve li diminye chans pou rekòmansman keloid.
Current literature supports silicone gel or sheeting with corticosteroid injections as first-line therapy for keloids. Adjuvant intralesional 5-fluorouracil (5-FU), bleomycin, or verapamil can be considered, although mixed results have been reported with each. Laser therapy can be used in combination with intralesional corticosteroids or topical steroids with occlusion to improve drug penetration. Excision of keloids with immediate post-excision radiation therapy is an effective option for recalcitrant lesions. Finally, silicone sheeting and pressure therapy have evidence for reducing keloid recurrence.
Current literature supports silicone gel or sheeting with corticosteroid injections as first-line therapy for keloids. Adjuvant intralesional 5-fluorouracil (5-FU), bleomycin, or verapamil can be considered, although mixed results have been reported with each. Laser therapy can be used in combination with intralesional corticosteroids or topical steroids with occlusion to improve drug penetration. Excision of keloids with immediate post-excision radiation therapy is an effective option for recalcitrant lesions. Finally, silicone sheeting and pressure therapy have evidence for reducing keloid recurrence.
Kounye a, pa gen okenn sèl tretman ki adapte tout gwosè ki garanti yon pousantaj repete ki toujou ba pou keloid. Sepandan, opsyon ki ap devlope, tankou itilizasyon laser ansanm ak steroid oswa konbine 5‑fluorouracil ak steroid, montre pwomès. Rechèch nan lavni ta ka konsantre sou kijan nouvo tretman, tankou graft grès oswa terapi ki baze sou selil souch, ka ede jere keloid.
There continues to be no gold standard of treatment that provides a consistently low recurrence rate; however the increasing number of available treatments and synergistic combinations of these treatments (i.e., laser-based devices in combination with intralesional steroids, or 5-fluorouracil in combination with steroid therapy) is showing favorable results. Future studies could target the efficacy of novel treatment modalities (i.e., autologous fat grafting or stem cell-based therapies) for keloid management.
There continues to be no gold standard of treatment that provides a consistently low recurrence rate; however the increasing number of available treatments and synergistic combinations of these treatments (i.e., laser-based devices in combination with intralesional steroids, or 5-fluorouracil in combination with steroid therapy) is showing favorable results. Future studies could target the efficacy of novel treatment modalities (i.e., autologous fat grafting or stem cell-based therapies) for keloid management.
Mak yo se yon pati komen nan pwosesis gerizon apre blesi po a. Idealman, mak yo ta dwe plat, mens, epi matche ak koulè po a. Anpil faktè ka mennen nan gerizon pòv blesi, tankou enfeksyon, sikilasyon san redwi, iskemi, ak chòk. Sikatris ki epè, ki pi fonse pase po ki antoure a, oswa ki retresi twòp, ka lakòz pwoblèm enpòtan sou fonksyon fizik ak sante emosyonèl.
Scars are a natural and normal part of healing following an injury to the integumentary system. Ideally, scars should be flat, narrow, and color-matched. Several factors can contribute to poor wound healing. These include but are not limited to infection, poor blood flow, ischemia, and trauma. Proliferative, hyperpigmented, or contracted scars can cause serious problems with both function and emotional well-being.
Scars are a natural and normal part of healing following an injury to the integumentary system. Ideally, scars should be flat, narrow, and color-matched. Several factors can contribute to poor wound healing. These include but are not limited to infection, poor blood flow, ischemia, and trauma. Proliferative, hyperpigmented, or contracted scars can cause serious problems with both function and emotional well-being.
Sikatris keloid yo wè pi souvan lakòz moun ki gen desandan Afriken, Azyatik oswa Panyòl. Moun ki gen laj ant 10 ak 30 an gen plis tandans pou devlope keloid pase granmoun aje.
Malgre ke yo souvan parèt apre yon blesi, keloid ka leve tou espontane. Yo ka parèt sou kote yon piercing, oswa menm apre yon bouton oswa grafouyen. Yo ka rive kòm rezilta akne grav oswa sikatris varisèl, enfeksyon sou yon blesi, chòk repete sou yon zòn, twòp tansyon po pandan fèmen blesi, oswa prezans yon kò etranje nan blesi a.
Mak keloid ka devlope apre operasyon. Yo pi komen sou kèk kote, tankou pwatrin santral (ki soti apre sternotomi), do ak zepòl (anjeneral ki soti apre akne), ak lòb zòrèy (soti apre piercing). Yo ka parèt tou sou piercing kò. Zòn ki pi souvan afekte yo se lòb zòrèy, bra, rejyon basen, ak sou kolye a.
Tretman ki disponib yo se terapi presyon, fèy jèl silicone, Triamcinolone acetonide intralesional, kriochiriji, radyasyon, terapi lazè, entèferon, 5‑FU, ak eksizyon chirijikal.
○ Tretman
Mak ipètrofik yo ka amelyore ak 5 a 10 piki esteroyid intralesional chak 1 mwa.
#Triamcinolone intralesional injection
Tretman lazè ka eseye pou eritem ki asosye ak sikatris, men piki triamcinolone kapab tou amelyore eritem sou mak la.
#Dye laser (e.g. V-beam)