Impetigo
https://en.wikipedia.org/wiki/Impetigo
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References
Impetigo: Diagnosis and Treatment 25250996Impetigo , ọrịa na-efe efe na-efe efe na-efe efe nke ụmụaka dị afọ abụọ ruo ise na-abịa n'ụdị isi abụọ: nonbulous (70% nke ikpe) na bullous (30% nke ikpe) . A na-ebutekarị impetigo na-enweghị mgbaasị Staphylococcus aureus ma ọ bụ Streptococcus pyogenes. A na-amata ya site na crusts ndị na-acha mmanụ aṅụ na ihu na aka na nke na-elekwasị anya n'akpụkpọ ahụ ma ọ bụ nwee ike ibunye ahụhụ ahụhụ, eczema, ma ọ bụ ọnya herpetic. Bullous impetigo, kpatara naanị S. Aureus, na-eduga na nnukwu, flaccid bullae na-emetụtakarị ebe akpụkpọ ahụ na-etekọta ọnụ. Ụdị abụọ a na-ekpochapụ n'ime izu abụọ ma ọ bụ atọ na-enweghị ọnyà, na nsogbu dị ụkọ, na poststreptococcal glomerulonephritis bụ nke kachasị njọ. Ọgwụgwọ na-agụnye ọgwụ nje ndị dị n'elu (mupirocin, retapamulin, fusidic acid) . Ọgwụ nje nwere ike ịdị mkpa maka impetigo nwere nnukwu bulla ma ọ bụ mgbe ọgwụgwọ n'elu anaghị ekwe omume. Ọ bụ ezie na ọtụtụ ọgwụ mgbochi ọnụ (amoxicillin/clavulanate, dicloxacillin, cephalexin, clindamycin, doxycycline, minocycline, trimethoprim/sulfamethoxazole, macrolides) bụ nhọrọ, penicillin adịghị arụ ọrụ. Ngwọta ndị dị n'elu adịghị mma dị ka ọgwụ nje ma ekwesịrị izere ya. Fusidic acid, mupirocin, retapamulin dị irè megide methicillin-susceptible S. Aureus na streptococcal ọrịa. Clindamycin bara uru maka ọrịa a na-enyo enyo methicillin-resistant S. Aureus ọrịa. Trimethoprim/sulfamethoxazole # na-arụ ọrụ megide methicillin na-eguzogide S. Aureus, mana ezughị maka ọrịa streptococcal.
Impetigo, the most common bacterial skin infection in children aged two to five, comes in two main types: nonbullous (70% of cases) and bullous (30% of cases). Nonbullous impetigo is typically caused by Staphylococcus aureus or Streptococcus pyogenes. It's recognized by honey-colored crusts on the face and limbs and mainly targets the skin or can infect insect bites, eczema, or herpetic lesions. Bullous impetigo, caused solely by S. aureus, leads to large, flaccid bullae and often affects areas where skin rubs together. Both types usually clear up within two to three weeks without scarring, and complications are rare, with poststreptococcal glomerulonephritis being the most severe. Treatment involves topical antibiotics (mupirocin, retapamulin, fusidic acid). Oral antibiotics might be necessary for impetigo with large bullae or when topical treatment isn't feasible. While several oral antibiotics (amoxicillin/clavulanate, dicloxacillin, cephalexin, clindamycin, doxycycline, minocycline, trimethoprim/sulfamethoxazole, macrolides) are options, penicillin isn't effective. Topical disinfectants aren't as good as antibiotics and should be avoided. Fusidic acid, mupirocin, retapamulin are effective against methicillin-susceptible S. aureus and streptococcal infections. Clindamycin is useful for suspected methicillin-resistant S. aureus infections. Trimethoprim/sulfamethoxazole works against methicillin-resistant S. aureus, but isn't enough for streptococcal infection.
Impetigo 28613693 NIH
Impetigo bụ ọrịa akpụkpọ anụ nke ụfọdụ nje bacteria na-ebute, na-agbasa ngwa ngwa site na kọntaktị. Ọ na-apụtakarị dị ka patches na-acha uhie uhie kpuchie ya na eriri na-acha odo odo ma nwee ike ịkpata itching ma ọ bụ mgbu. Ọrịa a na-adịkarị n'ime ụmụaka bi n'ebe ọkụ na iru mmiri. Ọ nwere ike ịpụta dị ka ọnya ma ọ bụ na-enweghị ha. Ọ bụ ezie na ọ na-emetụtakarị ihu, ọ nwere ike ime ebe ọ bụla nkwụsịtụ na akpụkpọ ahụ dị. Nchọpụta nchọpụta na-adabere na mgbaàmà yana ka ọ dị. Ọgwụgwọ na-agụnyekarị ọgwụ nje, ma n'elu ma nke ọnụ, yana njikwa akara.
Impetigo is a common infection of the superficial layers of the epidermis that is highly contagious and most commonly caused by gram-positive bacteria. It most commonly presents as erythematous plaques with a yellow crust and may be itchy or painful. The lesions are highly contagious and spread easily. Impetigo is a disease of children who reside in hot humid climates. The infection may be bullous or nonbullous. The infection typically affects the face but can also occur in any other part of the body that has an abrasion, laceration, insect bite or other trauma. Diagnosis is typically based on the symptoms and clinical manifestations alone. Treatment involves topical and oral antibiotics and symptomatic care.
Impetigo na-abụkarị n'ihi Staphylococcus aureus ma ọ bụ Streptococcus pyogenes. Site na kọntaktị ọ nwere ike gbasaa gburugburu ma ọ bụ n'etiti ndị mmadụ. N'ihe banyere ụmụaka, ọ na-efesa ụmụnne ha.
Ọgwụgwọ na-ejikarị ude ọgwụ nje dị ka mupirocin ma ọ bụ fusidic acid. Enwere ike iji ọgwụ nje site n'ọnụ, dị ka cefalexin, ma ọ bụrụ na emetụta akụkụ buru ibu.
Impetigo metụtara ihe dị ka nde mmadụ 140 (2% nke ndị bi n'ụwa) na 2010. Ọ nwere ike ime n'afọ ọ bụla, mana ọ na-adịkarị na ụmụaka. Mgbagwoju anya nwere ike ịgụnye cellulitis ma ọ bụ poststreptococcal glomerulonephritis.
○ Ọgwụgwọ - Ọgwụ OTC
* Ebe ọ bụ na impetigo bụ ọrịa na-efe efe, e kwesịghị iji mmanụ aṅụ steroid mee ihe. Ọ bụrụ na ị nwere nsogbu ịmata ọdịiche nke ọnya impetigo na eczemas, biko were ọgwụ antihistamines OTC na-ejighị mmanụ steroid.
#OTC antihistamine
* Biko tinye ude ọgwụ OTC na ọnya ahụ.
#Bacitracin
#Polysporin