Keloid
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Keloid a rụchara arụ ọrụ na nkwojiaka nke ejiri ọgwụ intralesional triamcinolone gwọọ ya. Ebe erythema dara dara n'akụkụ aka ekpe bụ ebe a na-agwọ ya.
Linear Keloid. Mgbe ha na-eme n'ihu elu nke torso, ha na-apụtakarị n'ụdị ahịrị.
Keloid auricular azụ
Keloids na ọbụ ụkwụ nwere ike ị gaghị ahụ ahụ iru ala ịga ije. A na-emekarị injections steroid intralesional ọtụtụ ugboro.
A na-ahụkarị Keloid na ogwe aka elu.
Ihe ngosi nke keloid obi.
relevance score : -100.0%
ReferencesKeloid na-etolite n'ihi ọgwụgwọ pụrụ iche mgbe mmerụ ahụ ma ọ bụ mbufụt gasịrị. Ihe mkpụrụ ndụ ihe nketa na gburugburu ebe obibi na-enye aka na mmepe ha, yana ọnụ ọgụgụ dị elu na ndị nwere akpụkpọ ahụ gbara ọchịchịrị nke Africa, Asia, na Hispanic. Keloid na-eme mgbe fibroblasts na-emebiga ihe ókè, na-emepụta oke collagen na ihe ndị na-eto eto. Nke a na-eduga n'ịmepụta nnukwu mkpokọta collagen na-adịghị mma nke a maara dị ka keloidal collagen, yana mmụba nke fibroblasts. Na ụlọ ọgwụ, keloid na-apụta dị ka ihe siri ike, rubbery nodules na mpaghara ndị merụrụ ahụ na mbụ. N'adịghị ka ọnyà nkịtị, keloid na-agbatị karịa ebe trauma mbụ. Ndị ọrịa nwere ike ịnwe mgbu, itching, ma ọ bụ ọkụ. Ọgwụgwọ dị iche iche dị, gụnyere steroid injections, cryotherapy, surgical excision, radiation therapy, na laser therapy.
Keloids result from abnormal wound healing in response to skin trauma or inflammation. Keloid development rests on genetic and environmental factors. Higher incidences are seen in darker skinned individuals of African, Asian, and Hispanic descent. Overactive fibroblasts producing high amounts of collagen and growth factors are implicated in the pathogenesis of keloids. As a result, classic histologic findings demonstrate large, abnormal, hyalinized bundles of collagen referred to as keloidal collagen and numerous fibroblasts. Keloids present clinically as firm, rubbery nodules in an area of prior injury to the skin. In contrast to normal or hypertrophic scars, keloidal tissue extends beyond the initial site of trauma. Patients may complain of pain, itching, or burning. Multiple treatment modalities exist although none are uniformly successful. The most common treatments include intralesional or topical steroids, cryotherapy, surgical excision, radiotherapy, and laser therapy.
Keloids result from abnormal wound healing in response to skin trauma or inflammation. Keloid development rests on genetic and environmental factors. Higher incidences are seen in darker skinned individuals of African, Asian, and Hispanic descent. Overactive fibroblasts producing high amounts of collagen and growth factors are implicated in the pathogenesis of keloids. As a result, classic histologic findings demonstrate large, abnormal, hyalinized bundles of collagen referred to as keloidal collagen and numerous fibroblasts. Keloids present clinically as firm, rubbery nodules in an area of prior injury to the skin. In contrast to normal or hypertrophic scars, keloidal tissue extends beyond the initial site of trauma. Patients may complain of pain, itching, or burning. Multiple treatment modalities exist although none are uniformly successful. The most common treatments include intralesional or topical steroids, cryotherapy, surgical excision, radiotherapy, and laser therapy.
Nnyocha a na-eme ugbu a na-egosi na silicone gel ma ọ bụ silicone sheeting yana corticosteroid injections bụ ọgwụgwọ mbụ kachasị mma maka keloids. A pụkwara ịtụle ọgwụgwọ ndị ọzọ dị ka intralesional 5-fluorouracil (5-FU), bleomycin, ma ọ bụ verapamil, n'agbanyeghị na arụmọrụ ha dịgasị iche. Laser therapy, mgbe ejikọtara ya na corticosteroid injections ma ọ bụ topical steroids n'okpuru mkpuchi, nwere ike ịkwalite ntinye ọgwụ. Maka recalcitrant keloids, excision with immediate post‑excision radiation therapy egosila na ọ dị irè. N'ikpeazụ, iji silicone sheeting na pressure therapy egosipụtara iji belata ohere nke nlọghachi nke keloid.
Current literature supports silicone gel or sheeting with corticosteroid injections as first-line therapy for keloids. Adjuvant intralesional 5-fluorouracil (5-FU), bleomycin, or verapamil can be considered, although mixed results have been reported with each. Laser therapy can be used in combination with intralesional corticosteroids or topical steroids with occlusion to improve drug penetration. Excision of keloids with immediate post-excision radiation therapy is an effective option for recalcitrant lesions. Finally, silicone sheeting and pressure therapy have evidence for reducing keloid recurrence.
Current literature supports silicone gel or sheeting with corticosteroid injections as first-line therapy for keloids. Adjuvant intralesional 5-fluorouracil (5-FU), bleomycin, or verapamil can be considered, although mixed results have been reported with each. Laser therapy can be used in combination with intralesional corticosteroids or topical steroids with occlusion to improve drug penetration. Excision of keloids with immediate post-excision radiation therapy is an effective option for recalcitrant lesions. Finally, silicone sheeting and pressure therapy have evidence for reducing keloid recurrence.
Ugbu a, ọ nweghị ọgwụgwọ dabara adaba‑niile nke na-ekwe nkwa na ọ na-adị obere nlọghachite maka keloids. Otú ọ dị, nhọrọ ndị na‑eto eto, dị ka iji lasers n'akụkụ steroid ma ọ bụ ijikọta 5‑fluorouracil na steroid, na‑ekwe nkwa. Nchọpụta n'ọdịnihu nwere ike ilekwasị anya n'otú ọgwụgwọ ọhụrụ, dị ka ntinye abụba autologous ma ọ bụ usoro ọgwụgwọ dabeere na cell, si arụ ọrụ maka ijikwa keloids.
There continues to be no gold standard of treatment that provides a consistently low recurrence rate; however the increasing number of available treatments and synergistic combinations of these treatments (i.e., laser-based devices in combination with intralesional steroids, or 5-fluorouracil in combination with steroid therapy) is showing favorable results. Future studies could target the efficacy of novel treatment modalities (i.e., autologous fat grafting or stem cell-based therapies) for keloid management.
There continues to be no gold standard of treatment that provides a consistently low recurrence rate; however the increasing number of available treatments and synergistic combinations of these treatments (i.e., laser-based devices in combination with intralesional steroids, or 5-fluorouracil in combination with steroid therapy) is showing favorable results. Future studies could target the efficacy of novel treatment modalities (i.e., autologous fat grafting or stem cell-based therapies) for keloid management.
Scars bụ akụkụ a na-ahụkarị nke usoro ọgwụgwọ mgbe mmerụ ahụ gasịrị. Dị ka o kwesịrị, ọnyà kwesịrị ịdị larịị, dị gịrịgịrị ma kwekọọ na agba akpụkpọ ahụ. Ọtụtụ ihe nwere ike iduga ọgwụgwọ ọnya na-adịghị mma, dị ka ọrịa, mbelata ọbara ọgbụgba, ischemia, na trauma. Akpụkpọ na-etolite (proliferative), akpụkpọ nwere pigment dị elu (hyperpigmented), ma ọ bụ akpụkpọ akpọrọ (contracted) nwere ike ịkpata nnukwu nsogbu na arụ ọrụ anụ ahụ yana mmetụta uche.
Scars are a natural and normal part of healing following an injury to the integumentary system. Ideally, scars should be flat, narrow, and color-matched. Several factors can contribute to poor wound healing. These include but are not limited to infection, poor blood flow, ischemia, and trauma. Proliferative, hyperpigmented, or contracted scars can cause serious problems with both function and emotional well-being.
Scars are a natural and normal part of healing following an injury to the integumentary system. Ideally, scars should be flat, narrow, and color-matched. Several factors can contribute to poor wound healing. These include but are not limited to infection, poor blood flow, ischemia, and trauma. Proliferative, hyperpigmented, or contracted scars can cause serious problems with both function and emotional well-being.
A na-ahụ ọnyà keloid ugboro ugboro na ndị African, Asian, ma ọ bụ ndị Hispanic. Ndị nọ n'agbata afọ 10 na 30 nwere ọchịchọ dị elu nke ịmepụta keloid karịa ndị agadi.
Ọ bụ ezie na ha na-emekarị na saịtị nke mmerụ ahụ, keloid nwekwara ike ibili ozugbo. Ha nwere ike ime na saịtị nke ịkpọpu ya na ọbụna site na ihe dị mfe dị ka pimple ma ọ bụ ọkọ. Ha nwere ike ime n'ihi acne ma ọ bụ chickenpox, infection na ebe ọnya, mmerụ ahụ ugboro ugboro na mpaghara, skin tension n'oge mmechi ọnya ma ọ bụ foreign body na ọnya.
Anya keloid nwere ike ịmalite mgbe a surgery. A na-ahụkarị ha na saịtị ụfọdụ, dị ka central chest (site na sternotomy), back and shoulders (na-esitekarị na acne), na lobes ntị (site n'ịkpọpu ntị). Ha nwekwara ike ime na mkpọpu anụ ahụ. Ebe ndị a na-ahụkarị bụ ntị, ogige aka, mpaghara pelvic, na n'elu collarbone.
Ọgwụgwọ dị bụ pressure therapy, silicone gel sheeting, intra‑lesional triamcinolone acetonide, cryosurgery, radiation, laser therapy, Interferon, 5‑FU na surgical excision.
○ Ọgwụgwọ
Ọkpụkpụ hypertrophic nwere ike imeziwanye site na intralesional steroid injections 5 ruo 10 1 ọnwa. #Triamcinolone intralesional injection
Enwere ike ịnwale ọgwụgwọ laser maka erythema jikọtara ya na ọnyà, mana triamcinolone injections nwekwara ike imezi erythema site n'iwetu ọnya ahụ. #Dye laser (e.g. V-beam)