Keloid
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Na nsonaazụ Stiftung Warentest nke 2022 sitere na Germany, afọ ojuju ndị ahịa na ModelDerm dị ntakịrị ntakịrị karịa na nyocha telemedicine akwụ ụgwọ.
Na nsonaazụ Stiftung Warentest nke 2022 sitere na Germany, afọ ojuju ndị ahịa na ModelDerm dị ntakịrị ntakịrị karịa na nyocha telemedicine akwụ ụgwọ.
Keloid a rụchara arụ ọrụ n'ịgwọ ya site n'iji ọgwụ intralesional triamcinolone. Ebe erythema dị n'akụkụ aka ekpe bụ ebe a na-agwọ ya.
Linear Keloid. Mgbe ha na-apụta n’elu akụkụ elu nke torso, ha na-apụta n'ụdị ahịrị.
Keloid auricular n'azụ
Keloids na ụkwụ nwere ike ime ka ị ghara ịnọ n’ọdị mma mgbe ị na-agba ije. A na-eme ọtụtụ ugboro injections steroid intralesional.
A na-ahụkarị keloid n'ogwe aka elu.
Ihe ngosi nke keloid obi.
relevance score : -100.0%
ReferencesKeloid na-etolite n'ihi ọgwụgwọ pụrụ iche mgbe mmerụ ahụ ma ọ bụ mbufụt gasịrị. Ihe mkpụrụ ndụ ihe nketa na gburugburu ebe obibi na-enye aka na mmepe ha, yana ọnụ ọgụgụ dị elu na ndị nwere akpụkpọ anụ gbara ọchịchịrị nke Africa, Asia, na Hispanic. Keloid na-eme mgbe fibroblasts na-emebiga ihe ókè, na-emepụta oke collagen na ihe ndị na-eto eto. Nke a na-eduga n'ịmepụta nnukwu mkpokọta collagen na-adịghị mma nke a maara dị ka keloidal collagen, yana mmụba nke fibroblasts. Na ụlọ ọgwụ, keloid na-apụta dị ka ihe siri ike, nodules rubbery na mpaghara ndị merụrụ ahụ na mbụ. N'adịghị ka ọnyà nkịtị, keloid na-agbatị karịa ebe trauma mbụ. Ndị ọrịa nwere ike ịnwe mgbu, itching, ma ọ bụ ọkụ. Ọgwụgwọ dị iche iche dị, gụnyere injections steroid, cryotherapy, ịwa ahụ, ọgwụgwọ redio, na ọgwụgwọ laser.
Keloids result from abnormal wound healing in response to skin trauma or inflammation. Keloid development rests on genetic and environmental factors. Higher incidences are seen in darker skinned individuals of African, Asian, and Hispanic descent. Overactive fibroblasts producing high amounts of collagen and growth factors are implicated in the pathogenesis of keloids. As a result, classic histologic findings demonstrate large, abnormal, hyalinized bundles of collagen referred to as keloidal collagen and numerous fibroblasts. Keloids present clinically as firm, rubbery nodules in an area of prior injury to the skin. In contrast to normal or hypertrophic scars, keloidal tissue extends beyond the initial site of trauma. Patients may complain of pain, itching, or burning. Multiple treatment modalities exist although none are uniformly successful. The most common treatments include intralesional or topical steroids, cryotherapy, surgical excision, radiotherapy, and laser therapy.
Keloids result from abnormal wound healing in response to skin trauma or inflammation. Keloid development rests on genetic and environmental factors. Higher incidences are seen in darker skinned individuals of African, Asian, and Hispanic descent. Overactive fibroblasts producing high amounts of collagen and growth factors are implicated in the pathogenesis of keloids. As a result, classic histologic findings demonstrate large, abnormal, hyalinized bundles of collagen referred to as keloidal collagen and numerous fibroblasts. Keloids present clinically as firm, rubbery nodules in an area of prior injury to the skin. In contrast to normal or hypertrophic scars, keloidal tissue extends beyond the initial site of trauma. Patients may complain of pain, itching, or burning. Multiple treatment modalities exist although none are uniformly successful. The most common treatments include intralesional or topical steroids, cryotherapy, surgical excision, radiotherapy, and laser therapy.
Nnyocha a na-eme ugbu a na-egosi na silicone gel ma ọ bụ mpempe akwụkwọ yana corticosteroid injections bụ ọgwụgwọ mbụ kachasị mma maka keloids. A pụkwara ịtụle ọgwụgwọ ndị ọzọ dị ka intralesional 5-fluorouracil (5-FU), bleomycin, ma ọ bụ verapamil, n'agbanyeghị na arụmọrụ ha dịgasị iche. Usoro ọgwụgwọ laser, mgbe ejikọtara ya na corticosteroid injections ma ọ bụ ọgwụ steroid dị n'okpuru mkpuchi, nwere ike ime ka ntinye ọgwụ dịkwuo mma. Maka keloids recalcitrant, mwepụ ịwa ahụ nke usoro ọgwụgwọ radieshon na-esote egosila na ọ dị irè. N'ikpeazụ, iji silicone sheeting na ọgwụgwọ nrụgide egosipụtara belata ohere nke nlọghachi keloid.
Current literature supports silicone gel or sheeting with corticosteroid injections as first-line therapy for keloids. Adjuvant intralesional 5-fluorouracil (5-FU), bleomycin, or verapamil can be considered, although mixed results have been reported with each. Laser therapy can be used in combination with intralesional corticosteroids or topical steroids with occlusion to improve drug penetration. Excision of keloids with immediate post-excision radiation therapy is an effective option for recalcitrant lesions. Finally, silicone sheeting and pressure therapy have evidence for reducing keloid recurrence.
Current literature supports silicone gel or sheeting with corticosteroid injections as first-line therapy for keloids. Adjuvant intralesional 5-fluorouracil (5-FU), bleomycin, or verapamil can be considered, although mixed results have been reported with each. Laser therapy can be used in combination with intralesional corticosteroids or topical steroids with occlusion to improve drug penetration. Excision of keloids with immediate post-excision radiation therapy is an effective option for recalcitrant lesions. Finally, silicone sheeting and pressure therapy have evidence for reducing keloid recurrence.
Ugbu a, ọ nweghị ọgwụgwọ dabara adaba‑niile nke na-ekwe nkwa na ọ na-adị obere nlọghachite maka keloids. Otú ọ dị, nhọrọ ndị na-eto eto, dị ka iji lasers n'akụkụ steroid ma ọ bụ ijikọta 5‑fluorouracil na steroid, na‑ekwe nkwa. Nchọpụta n'ọdịnihu nwere ike ilekwasị anya n'otú ọgwụgwọ ọhụrụ, dị ka ntinye autologous fat ma ọ bụ usoro ọgwụgwọ dabeere na cell, si arụ ọrụ maka ijikwa keloids.
There continues to be no gold standard of treatment that provides a consistently low recurrence rate; however the increasing number of available treatments and synergistic combinations of these treatments (i.e., laser-based devices in combination with intralesional steroids, or 5-fluorouracil in combination with steroid therapy) is showing favorable results. Future studies could target the efficacy of novel treatment modalities (i.e., autologous fat grafting or stem cell-based therapies) for keloid management.
There continues to be no gold standard of treatment that provides a consistently low recurrence rate; however the increasing number of available treatments and synergistic combinations of these treatments (i.e., laser-based devices in combination with intralesional steroids, or 5-fluorouracil in combination with steroid therapy) is showing favorable results. Future studies could target the efficacy of novel treatment modalities (i.e., autologous fat grafting or stem cell-based therapies) for keloid management.
Scars bụ akụkụ a na-ahụkarị n'usoro ọgwụgwọ mgbe mmerụ ahụ gasịrị. Dị ka o kwesịrị, ọnyà kwesịrị ịdị larịị, dị gịrịgịrị ma kwekọọ na agba akpụkpọ ahụ. Ọtụtụ ihe nwere ike iduga ọgwụgwọ ọnyà na-adịghị mma, dịka ọrịa, mbelata ọbara ọgbụgba, ischemia, na trauma. Ọkpụkpụ nke toro ogologo, gbara ọchịchịrị karịa akpụkpọ ahụ gbara ya gburugburu, ma ọ bụ nke na-adalata nke ukwuu, nwere ike ịkpata nnukwu nsogbu n'arụ ọrụ anụ ahụ yana mmetụta uche.
Scars are a natural and normal part of healing following an injury to the integumentary system. Ideally, scars should be flat, narrow, and color-matched. Several factors can contribute to poor wound healing. These include but are not limited to infection, poor blood flow, ischemia, and trauma. Proliferative, hyperpigmented, or contracted scars can cause serious problems with both function and emotional well-being.
Scars are a natural and normal part of healing following an injury to the integumentary system. Ideally, scars should be flat, narrow, and color-matched. Several factors can contribute to poor wound healing. These include but are not limited to infection, poor blood flow, ischemia, and trauma. Proliferative, hyperpigmented, or contracted scars can cause serious problems with both function and emotional well-being.
A na-ahụ keloid ugboro ugboro n’ụlọ ndị Africa, Asia, ma ọ bụ Hispanic. Ndị dị n’etiti afọ 10 na 30 nwere ohere dị elu ịmepụta keloid karịa ndị agadi.
Ọ bụ ezie na ha na-apụta n’ógbè mmerụ ahụ, keloid nwekwara ike ibili ozugbo. Ha nwere ike ime n’ógbè a kpụchara akpụkpọ, ọbụna site na pimple ma ọ bụ ọkọ. Keloid nwekwara ike ibili n’ihi oke otutu, ọkụkọ ọkụkọ, ọrịa na ebe ọnya, mmerụ ahụ ugboro ugboro n’ógbè ahụ, ma ọ bụ n’oge mgbakasị anụ ahụ mgbe a na-eme ihe n’akụkụ ọnya ma ọ bụ n’akụkụ ahụ ọzọ.
Anya keloid nwere ike ịmalite mgbe a wụsịrị ya. A na-ahụkarị ha n’ógbè ụfọdụ, dịka igbe etiti (sternotomy), azụ na ubu (na-esitekarị na otutu), na lobes ntị (site n'ịkpọpu ntị). Ha nwekwara ike ime n’mkpọpu anụ ahụ. Ebe ndị a na-ahụkarị bụ ntị, ogwe aka, mpaghara pelvic, na n’elu ọkpụkpụ olu.
Ọgwụgwọ dị: ọgwụgwọ nrụgide, silicone gel sheeting, intra‑lesional triamcinolone acetonide, cryosurgery, radieshon, laser therapy, Interferon, 5‑FU, na ịwa ahụ.
○ Ọgwụgwọ
Hypertrophic scars nwere ike imeziwanye site na intralesional steroid injections 5 ruo 10 n’ime ọnwa 1.
#Triamcinolone intralesional injection
Enwere ike ịnwale laser therapy maka erythema jikọtara ya na ọnya, ma injections triamcinolone nwekwara ike imezi erythema site n'ịwelite ọnya ahụ.
#Dye laser (e.g. V‑beam)