Poikiloderma - 多形皮膚萎縮症
多形皮膚萎縮症 (Poikiloderma) は、色素沈着低下、色素沈着過剰、毛細血管拡張症、および萎縮の領域で構成される皮膚疾患です。 多形皮膚萎縮症 (poikiloderma) は胸や首に最もよく見られ、一般に日光による損傷と関連する皮膚の赤い色素を特徴とします。
References
Poikiloderma of Civatte は、主に首と顔に現れる一般的な皮膚疾患で、特に色白の閉経後の女性に多く見られます。赤い線、黒い斑点、薄い皮膚が混在して現れます。通常、顔、首、胸など、太陽にさらされた領域に影響を与えますが、日陰の領域には影響しません。 Poikiloderma of Civatte は、赤み、黒ずみ、またはその両方の混合という主な特徴に基づいて分類できます。正確な原因は完全には解明されていませんが、日光への曝露、ホルモンの変化、香水や化粧品への反応、加齢などの要因が関与していると考えられています。 Poikiloderma of Civatte は時間の経過とともにゆっくりと悪化する傾向があります。
Poikiloderma of Civatte (PC) is a rather common benign dermatosis of the neck and face, mainly affecting fair-skinned individuals, especially postmenopausal females. It is characterized by a combination of a reticular pattern of linear telangiectasia, mottled hyperpigmentation and superficial atrophy. Clinically, it involves symmetrically sun-exposed areas of the face, the neck, and the V-shaped area of the chest, invariably sparing the anatomically shaded areas. Depending on the prevalent clinical feature, PC can be classified into erythemato-telangiectatic, pigmented, and mixed clinical types. The etiopathogenesis of PC is incompletely understood. Exposure to ultraviolet radiation, hormonal changes of menopause, contact sensitization to perfumes and cosmetics, and normal ageing have been incriminated. The diagnosis is usually clinical and can be confirmed by histology, which is characteristic, but not pathognomonic. The course is slowly progressive and irreversible, often causing significant cosmetic disfigurement.
Poikiloderma of Civatte (PC) is a rather common benign dermatosis of the neck and face, mainly affecting fair-skinned individuals, especially postmenopausal females. It is characterized by a combination of a reticular pattern of linear telangiectasia, mottled hyperpigmentation and superficial atrophy. Clinically, it involves symmetrically sun-exposed areas of the face, the neck, and the V-shaped area of the chest, invariably sparing the anatomically shaded areas. Depending on the prevalent clinical feature, PC can be classified into erythemato-telangiectatic, pigmented, and mixed clinical types. The etiopathogenesis of PC is incompletely understood. Exposure to ultraviolet radiation, hormonal changes of menopause, contact sensitization to perfumes and cosmetics, and normal ageing have been incriminated. The diagnosis is usually clinical and can be confirmed by histology, which is characteristic, but not pathognomonic. The course is slowly progressive and irreversible, often causing significant cosmetic disfigurement.
