Amyloidosis
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Ing asil Stiftung Warentest 2022 saka Jerman, kepuasan konsumen karo ModelDerm mung luwih murah tinimbang konsultasi telemedicine sing dibayar.
Fitur rai klasik saka Amyloidosis
relevance score : -100.0%
ReferencesWong wadon umur 26 taun teka ing klinik kita amarga gatel ing sikile sing wis suwé 10 taun. Sanajan wis nggunakake krim steroid lan krim tazarotene, ruam ora mari. Dheweke ora duwe riwayat kulawarga sing relevan. Nalika dipriksa, ditemokake bintik‑bintik kasar ing ngarep sikile, sing cocog karo lichen amyloidosis.
A 26-year-old woman presented to our clinic with an itchy rash on her legs that had persisted for 10 years. The rash had previously been treated with topical steroids and tazarotene cream, with no improvement. The patient’s family history was noncontributory. A physical examination showed discrete and coalescing hyperkeratotic tan-brown papules on the pretibial surfaces, consistent with lichen amyloidosis.
A 26-year-old woman presented to our clinic with an itchy rash on her legs that had persisted for 10 years. The rash had previously been treated with topical steroids and tazarotene cream, with no improvement. The patient’s family history was noncontributory. A physical examination showed discrete and coalescing hyperkeratotic tan-brown papules on the pretibial surfaces, consistent with lichen amyloidosis.
Lichen Amyloidosis yaiku kondisi kulit langka sing ana gandhengane karo gatel sing ora dingerteni sababé. Biasane katon minangka klompok bintik-bintik sing menonjol, berwarna berubah pada permukaan luar kulit. Lichen Amyloidosis biasane katon ing wong umur 50 nganti 60 taun lan sayangé, ora ana tamba kanggo kondisi iki. Pengobatan sing kasedhiya saiki biasane ora efektif.
Lichen Amyloidosis (LA) is an uncommon, primary cutaneous amyloidosis associated with chronic, idiopathic pruritus. Clinical presentation of LA includes skin colored to hyperpigmented, papules coalescing into plaques with a rippled appearance on the extensors.1 LA most commonly presents in the fifth to sixth decade of life and has no curative treatments. Overall response to current therapies is poor.
Lichen Amyloidosis (LA) is an uncommon, primary cutaneous amyloidosis associated with chronic, idiopathic pruritus. Clinical presentation of LA includes skin colored to hyperpigmented, papules coalescing into plaques with a rippled appearance on the extensors.1 LA most commonly presents in the fifth to sixth decade of life and has no curative treatments. Overall response to current therapies is poor.
Lichen amyloidosis yaiku kondisi kulit gatel sing tahan suwe. Dikenal amarga klompok benjolan sing kenthel, utamane ditemokake ing punggung, balung, pupu, lan lengen. Nalika diteliti ing mikroskop, Lichen amyloidosis nuduhake akumulasi amiloid ing lapisan ndhuwur kulit bebarengan karo penebalan lan pembesaran lapisan kulit njaba. Sanadyan panyebab sing tepat saka Lichen amyloidosis durung dimangerteni kanthi lengkap, panliten sadurunge wis nggandhengake faktor kaya gesekan utawa iritasi ing kulit, pati sel, infeksi virus, lan liya‑liyane. Lichen amyloidosis katon ana hubungane karo sawetara kondisi kulit liyane (atopic dermatitis, lichen planus, mycosis fungoides).
Lichen amyloidosis (LA) is a chronic pruritic skin disorder characterized by multiple grouped hyperkeratotic papules, predominantly located on the back, shins, thighs, and arms. Histological analysis of LA shows amyloid deposition in the papillary dermis and hyperkeratosis and acanthosis of the epidermis. The exact pathogenesis of LA has not yet been elucidated; however, prior reports have implicated frictional epidermal damage, apoptosis, viral infection, and many other triggers. LA is reportedly associated with several skin disorders, including atopic dermatitis (AD), lichen planus, and mycosis fungoides.
Lichen amyloidosis (LA) is a chronic pruritic skin disorder characterized by multiple grouped hyperkeratotic papules, predominantly located on the back, shins, thighs, and arms. Histological analysis of LA shows amyloid deposition in the papillary dermis and hyperkeratosis and acanthosis of the epidermis. The exact pathogenesis of LA has not yet been elucidated; however, prior reports have implicated frictional epidermal damage, apoptosis, viral infection, and many other triggers. LA is reportedly associated with several skin disorders, including atopic dermatitis (AD), lichen planus, and mycosis fungoides.
○ Diagnosis lan Perawatan
#Electrophoresis of blood or urine
#Skin biopsy