Poikilodermahttps://en.wikipedia.org/wiki/Poikiloderma
Poikiloderma yaiku kondisi kulit sing kasusun saka area hipopigmentasi, hiperpigmentasi, telangiektasis, lan atrofi. Poikiloderma paling kerep katon ing dhadha utawa gulu, ditandai dening pigmen werna abang ing kulit sing umum digandhengake karo kerusakan sinar matahari.

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      References Diagnosis and Differential Diagnosis of Poikiloderma of Civatte: A Dermoscopy Cohort Study 36892344 
      NIH
      Poikiloderma of Civatte minangka kondisi kulit sing umum lan biasane katon ing gulu lan rai, utamane ing wanita postmenopausal kanthi kulit putih. Kondisi iki muncul minangka campuran garis abang, bintik peteng, lan kulit tipis. Biasane mengaruhi wilayah sing kena sinar srengenge, kayata rai, gulu, lan dada, nanging ora katon ing wilayah sing peteng. Poikiloderma of Civatte bisa dikategorikaké adhedhasar fitur utama: abang, bintik peteng, utawa campuran saka loro‑lorone. Penyebab sing tepat durung dimangerteni kanthi lengkap, nanging faktor kayata cahya srengenge, owah‑owahan hormonal, reaksi parfum utawa kosmetik, lan penuaan dianggep nduwèni peran. Poikiloderma of Civatte cenderung nambah parah saka wektu ke wektu.
      Poikiloderma of Civatte (PC) is a rather common benign dermatosis of the neck and face, mainly affecting fair-skinned individuals, especially postmenopausal females. It is characterized by a combination of a reticular pattern of linear telangiectasia, mottled hyperpigmentation and superficial atrophy. Clinically, it involves symmetrically sun-exposed areas of the face, the neck, and the V-shaped area of the chest, invariably sparing the anatomically shaded areas. Depending on the prevalent clinical feature, PC can be classified into erythemato-telangiectatic, pigmented, and mixed clinical types. The etiopathogenesis of PC is incompletely understood. Exposure to ultraviolet radiation, hormonal changes of menopause, contact sensitization to perfumes and cosmetics, and normal ageing have been incriminated. The diagnosis is usually clinical and can be confirmed by histology, which is characteristic, but not pathognomonic. The course is slowly progressive and irreversible, often causing significant cosmetic disfigurement.