Postinflammatory hyperpigmentation - 염증 후 색소 침착
염증 후 색소 침착 (Postinflammatory hyperpigmentation) 은 피부 염증에 따라 피부의 색소가 증가하는 것을 특징으로 하는 피부 질환입니다. 염증 후 색소 침착 (postinflammatory hyperpigmentation) 은 장기간의 햇빛 노출, 염증 또는 여드름과 관련된 손상을 포함한 기타 피부 손상으로 인해 발생할 수 있습니다. 피부색이 어두운 사람은 일반적으로 햇빛에 과도하게 노출되면 과다 색소 침착이 발생하기 쉽습니다.
References
Postinflammatory hyperpigmentation (PIH) 은 피부 염증이나 부상 후에 발생하는 빈번한 피부 문제입니다. 이는 오래 지속되는 경향이 있으며 피부색이 어두운 사람에게 더 심합니다 (Fitzpatrick skin types III–VI) . 저절로 좋아지는 경우가 많지만, 시간이 오래 걸릴 수 있어 장기간 치료가 필요한 경우가 많습니다. 다양한 치료법을 결합하는 것이 가장 효과적입니다.
Postinflammatory hyperpigmentation (PIH) is a common acquired cutaneous disorder occurring after skin inflammation or injury. It is chronic and is more common and severe in darker-skinned individuals (Fitzpatrick skin types III–VI). While the condition typically improves spontaneously, this process can take months to years, necessitating prolonged treatment. Combination therapy is the most effective.
Postinflammatory hyperpigmentation (PIH) is a common acquired cutaneous disorder occurring after skin inflammation or injury. It is chronic and is more common and severe in darker-skinned individuals (Fitzpatrick skin types III–VI). While the condition typically improves spontaneously, this process can take months to years, necessitating prolonged treatment. Combination therapy is the most effective.
Postinflammatory hyperpigmentation 은 피부 염증의 흔한 후유증입니다. 피부색이 어두운 개인에게 더 심각하고 자주 영향을 미치는 경향이 있습니다. 연구에 따르면 postinflammatory hyperpigmentation 과 같은 문제는 피부색이 어두운 사람들이 피부과 진료를 찾는 주된 이유 중 하나입니다. Postinflammatory hyperpigmentation 을 해결하려면 조기 치료가 중요하며 일반적으로 초기 염증 상태를 관리하는 것부터 시작됩니다. 치료의 첫 번째 라인은 일반적으로 보호를 위한 자외선 차단제와 함께 피부를 밝게 해주는 국소 제제를 사용하는 것입니다. Hydroquinone, azelaic acid, kojic acid, arbutin, licorice extracts 과 같은 이러한 제제는 과도한 색소 침착을 효과적으로 줄일 수 있습니다. 또한 retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, soy 은 탈색제로도 사용되며 새로운 치료법이 등장하고 있습니다. 국소 치료는 일반적으로 표면 수준의 과다 색소 침착에 효과적이지만, 완치되는 경우에는 시술 (laser, chemical peel) 이 필요할 수 있습니다. Postinflammatory hyperpigmentation 의 자극과 악화를 피하기 위해 이러한 치료법을 사용할 때 주의를 기울이는 것이 중요합니다.
Postinflammatory hyperpigmentation is a common sequelae of inflammatory dermatoses that tends to affect darker skinned patients with greater frequency and severity. Epidemiological studies show that dyschromias, including postinflammatory hyperpigmentation, are among the most common reasons darker racial/ethnic groups seek the care of a dermatologist. The treatment of postinflammatory hyperpigmentation should be started early to help hasten its resolution and begins with management of the initial inflammatory condition. First-line therapy typically consists of topical depigmenting agents in addition to photoprotection including a sunscreen. Topical tyrosinase inhibitors, such as hydroquinone, azelaic acid, kojic acid, arbutin, and certain licorice extracts, can effectively lighten areas of hypermelanosis. Other depigmenting agents include retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, and soy with a number of emerging therapies on the horizon. Topical therapy is typically effective for epidermal postinflammatory hyperpigmentation; however, certain procedures, such as chemical peeling and laser therapy, may help treat recalcitrant hyperpigmentation. It is also important to use caution with all of the above treatments to prevent irritation and worsening of postinflammatory hyperpigmentation.
Postinflammatory hyperpigmentation is a common sequelae of inflammatory dermatoses that tends to affect darker skinned patients with greater frequency and severity. Epidemiological studies show that dyschromias, including postinflammatory hyperpigmentation, are among the most common reasons darker racial/ethnic groups seek the care of a dermatologist. The treatment of postinflammatory hyperpigmentation should be started early to help hasten its resolution and begins with management of the initial inflammatory condition. First-line therapy typically consists of topical depigmenting agents in addition to photoprotection including a sunscreen. Topical tyrosinase inhibitors, such as hydroquinone, azelaic acid, kojic acid, arbutin, and certain licorice extracts, can effectively lighten areas of hypermelanosis. Other depigmenting agents include retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, and soy with a number of emerging therapies on the horizon. Topical therapy is typically effective for epidermal postinflammatory hyperpigmentation; however, certain procedures, such as chemical peeling and laser therapy, may help treat recalcitrant hyperpigmentation. It is also important to use caution with all of the above treatments to prevent irritation and worsening of postinflammatory hyperpigmentation.
