Postinflammatory hyperpigmentation - 염증 후 색소 침착
염증 후 색소 침착 (Postinflammatory hyperpigmentation)은 피부 염증 후 색소가 증가하는 현상을 의미하는 피부 상태입니다. 염증 후 색소 침착 (postinflammatory hyperpigmentation)은 장기간 햇빛 노출, 염증, 여드름 등으로 인한 피부 손상으로도 발생할 수 있습니다. 피부톤이 어두운 사람은 햇빛에 과도하게 노출될 경우 색소 침착이 더 쉽게 나타납니다.
relevance score : -100.0%
ReferencesPostinflammatory hyperpigmentation (PIH)은 피부 염증이나 부상 후에 발생하는 흔한 피부 문제입니다. 이는 오래 지속되는 경향이 있으며, 피부색이 어두운 사람에게 더 심하게 나타납니다 (Fitzpatrick skin types III–VI). 자연스럽게 호전되는 경우가 많지만 회복에 시간이 오래 걸릴 수 있어 장기간 치료가 필요한 경우가 많습니다. 다양한 치료법을 병합하는 것이 가장 효과적입니다.
Postinflammatory hyperpigmentation (PIH) is a common acquired cutaneous disorder occurring after skin inflammation or injury. It is chronic and is more common and severe in darker-skinned individuals (Fitzpatrick skin types III–VI). While the condition typically improves spontaneously, this process can take months to years, necessitating prolonged treatment. Combination therapy is the most effective.
Postinflammatory hyperpigmentation (PIH) is a common acquired cutaneous disorder occurring after skin inflammation or injury. It is chronic and is more common and severe in darker-skinned individuals (Fitzpatrick skin types III–VI). While the condition typically improves spontaneously, this process can take months to years, necessitating prolonged treatment. Combination therapy is the most effective.
Postinflammatory hyperpigmentation은 피부 염증의 흔한 후유증으로, 피부색이 어두운 사람에게 더 심각하고 자주 나타납니다. 연구에 따르면 Postinflammatory hyperpigmentation과 같은 문제는 피부색이 어두운 사람들이 피부과 진료를 찾는 주요 이유 중 하나입니다. Postinflammatory hyperpigmentation을 해결하려면 조기 치료가 중요하며, 일반적으로 초기 염증 상태를 관리하는 것부터 시작합니다. 치료의 첫 번째 단계는 자외선 차단제로 피부를 보호하고, 피부를 밝게 해주는 국소 제제를 사용하는 것입니다. Hydroquinone, azelaic acid, kojic acid, arbutin, licorice extracts와 같은 제제는 과도한 색소 침착을 효과적으로 감소시킬 수 있습니다. 또한 retinoids, mequinol, ascorbic acid, niacinamide, N‑acetyl glucosamine, soy도 탈색제로 사용됩니다. 새로운 치료법이 계속 등장하고 있습니다. 국소 치료는 표면 수준의 과다 색소 침착에 효과적이지만, 완치가 어려운 경우 레이저나 화학적 필링과 같은 시술이 필요할 수 있습니다. Postinflammatory hyperpigmentation의 자극과 악화를 방지하기 위해 이러한 치료법을 사용할 때 주의를 기울이는 것이 중요합니다.
Postinflammatory hyperpigmentation is a common sequelae of inflammatory dermatoses that tends to affect darker skinned patients with greater frequency and severity. Epidemiological studies show that dyschromias, including postinflammatory hyperpigmentation, are among the most common reasons darker racial/ethnic groups seek the care of a dermatologist. The treatment of postinflammatory hyperpigmentation should be started early to help hasten its resolution and begins with management of the initial inflammatory condition. First-line therapy typically consists of topical depigmenting agents in addition to photoprotection including a sunscreen. Topical tyrosinase inhibitors, such as hydroquinone, azelaic acid, kojic acid, arbutin, and certain licorice extracts, can effectively lighten areas of hypermelanosis. Other depigmenting agents include retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, and soy with a number of emerging therapies on the horizon. Topical therapy is typically effective for epidermal postinflammatory hyperpigmentation; however, certain procedures, such as chemical peeling and laser therapy, may help treat recalcitrant hyperpigmentation. It is also important to use caution with all of the above treatments to prevent irritation and worsening of postinflammatory hyperpigmentation.
Postinflammatory hyperpigmentation is a common sequelae of inflammatory dermatoses that tends to affect darker skinned patients with greater frequency and severity. Epidemiological studies show that dyschromias, including postinflammatory hyperpigmentation, are among the most common reasons darker racial/ethnic groups seek the care of a dermatologist. The treatment of postinflammatory hyperpigmentation should be started early to help hasten its resolution and begins with management of the initial inflammatory condition. First-line therapy typically consists of topical depigmenting agents in addition to photoprotection including a sunscreen. Topical tyrosinase inhibitors, such as hydroquinone, azelaic acid, kojic acid, arbutin, and certain licorice extracts, can effectively lighten areas of hypermelanosis. Other depigmenting agents include retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, and soy with a number of emerging therapies on the horizon. Topical therapy is typically effective for epidermal postinflammatory hyperpigmentation; however, certain procedures, such as chemical peeling and laser therapy, may help treat recalcitrant hyperpigmentation. It is also important to use caution with all of the above treatments to prevent irritation and worsening of postinflammatory hyperpigmentation.
