Herpes simplex
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Herpes simplex li ser lêv.
Herpes simplex - Enfeksiyona herpes simplex di tiliyan de li zarokên piçûk ji mezinan pirtir e.
relevance score : -100.0%
ReferencesEnfeksiyona HSV-1 bi enfeksiyona seretayî ya hûcreyên epitelî dest pê dike, piştî derengbûnê bi giranî di nêuronan de, û ji nû ve aktîvkiriê pêşve diçe. HSV-1 bi gelemperî sedema teqînên vaskuler ên destpêkê û dûbare dibe, bi giranî li ser dev û mukoza zayendî. Nîşanên wê ji herpesê orolabial heta rewşên cihêreng ên wekî foliculitis herpetic, enfeksiyonên çermî, tevlêbûna çavan, û rewşên giran ên mîna herpes encephalitis diguhere. Tedawiya antiviral alîkariya birêvebirina enfeksiyona HSV dike.
Herpes simplex virus type 1 (HSV-1) is a member of the Alphaherpesviridae subfamily. Its structure is composed of linear dsDNA, an icosahedral capsid that is 100 to 110 nm in diameter, with a spikey envelope. In general, the pathogenesis of HSV-1 infection follows a cycle of primary infection of epithelial cells, latency primarily in neurons, and reactivation. HSV-1 is responsible for establishing primary and recurrent vesicular eruptions, primarily in the orolabial and genital mucosa. HSV-1 infection has a wide variety of presentations, including orolabial herpes, herpetic sycosis (HSV folliculitis), herpes gladiatorum, herpetic whitlow, ocular HSV infection, herpes encephalitis, Kaposi varicelliform eruption (eczema herpeticum), and severe or chronic HSV infection. Antiviral therapy limits the course of HSV infection.
Herpes simplex virus type 1 (HSV-1) is a member of the Alphaherpesviridae subfamily. Its structure is composed of linear dsDNA, an icosahedral capsid that is 100 to 110 nm in diameter, with a spikey envelope. In general, the pathogenesis of HSV-1 infection follows a cycle of primary infection of epithelial cells, latency primarily in neurons, and reactivation. HSV-1 is responsible for establishing primary and recurrent vesicular eruptions, primarily in the orolabial and genital mucosa. HSV-1 infection has a wide variety of presentations, including orolabial herpes, herpetic sycosis (HSV folliculitis), herpes gladiatorum, herpetic whitlow, ocular HSV infection, herpes encephalitis, Kaposi varicelliform eruption (eczema herpeticum), and severe or chronic HSV infection. Antiviral therapy limits the course of HSV infection.
Herpes simplex virus type 2 (HSV-2) enfeksiyek berbelav e, li dora 22% ji kesên ku di nav 12 salî de ne, û bandora jorîn dike, bi tevahî 45 mîlyon kes li Dewletên Yekbûyî. HSV-1 bi gelemperî sedema birînên devkî dibe, lê ew dikare sedema birînên zayendî jî bibe. Lêbelê, dema ku birînên genital hene, HSV-2 bi gelemperî xemgîniya sereke ye. Nîşaneyên derketinê yên HSV-2 bi gelemperî nezelal in, mîna xurbûn û acizbûna organên zayendî, ku dikarin tespît û dermankirinê dereng bikin. Ev dereng dikare sedema veguheztina bêtir ji kesên nevegirtî bibe.
Herpes simplex virus type 2 (HSV-2) continues to be a common infection, affecting approximately 22% of adults ages 12 and older, representing 45 million adults in the United States alone. While HSV-1 often affects the perioral region and can be known to cause genital lesions, HSV-2 is more commonly the consideration when patients present with genital lesions. Despite this, most outbreaks of the infection will present with nonspecific symptoms such as genital itching, irritation, and excoriations, which may cause diagnosis and treatment to be delayed. As a result, further exposure to uninfected individuals may occur.
Herpes simplex virus type 2 (HSV-2) continues to be a common infection, affecting approximately 22% of adults ages 12 and older, representing 45 million adults in the United States alone. While HSV-1 often affects the perioral region and can be known to cause genital lesions, HSV-2 is more commonly the consideration when patients present with genital lesions. Despite this, most outbreaks of the infection will present with nonspecific symptoms such as genital itching, irritation, and excoriations, which may cause diagnosis and treatment to be delayed. As a result, further exposure to uninfected individuals may occur.
Herpes simplex virus (HSV) bi gelemperî di ciwan û mezinan de sedema enfeksiyonên mîna herpesên genital û birînên sermayê dibe. Dema ku HSV di nav 4‑6 hefteyên pêşîn a jiyanê de pitikek bikişîne, dikare sedema nexweşiyek giran û encamên giran bibe. Zû zû teşhîskirina enfeksiyona HSV ya neonatal pir girîng e da ku nexweşî nebe xerabtir, û pêşî li pirsgirêkên neurolojîk (tevî mirinê) bigire.
Herpes simplex virus (HSV), a member of the Herpesviridae family, is a well-known cause of infections including genital herpes and herpes labialis in the adolescent and adult population. Transmission of HSV infection to an infant during the first 4-6 weeks of life can lead to devastating disease with the potential for poor outcomes. Early diagnosis is imperative when evaluating neonatal HSV infection in order to prevent further disease progression, neurological complications, and even death.
Herpes simplex virus (HSV), a member of the Herpesviridae family, is a well-known cause of infections including genital herpes and herpes labialis in the adolescent and adult population. Transmission of HSV infection to an infant during the first 4-6 weeks of life can lead to devastating disease with the potential for poor outcomes. Early diagnosis is imperative when evaluating neonatal HSV infection in order to prevent further disease progression, neurological complications, and even death.
Infeksiyona Herpes simplex pir berbelav e û ji jinên ducanî derbasî zarokên wan dibe. Ev vîrus dikare bibe sedema pirsgirêkên tenduristiyê yên ciddî an jî mirina di zarokên nûbûyî de. Dema ku ew di dema ducanî de xwe kêm dike, pir caran di dema zayînê de dibe. Ger dêûbêj di qonaxên paşerojê yên ducanî de bi vegirtinê tê de, xetereya herî zêde ye. Lêbelê, ev xetere dikare bi karanîna dermanên antiviral an bijartina beşa C‑ê di hin rewşan de kêm bibe.
Infection with herpes simplex is one of the most common sexually transmitted infections. Because the infection is common in women of reproductive age it can be contracted and transmitted to the fetus during pregnancy and the newborn. Herpes simplex virus is an important cause of neonatal infection, which can lead to death or long-term disabilities. Rarely in the uterus, it occurs frequently during the transmission delivery. The greatest risk of transmission to the fetus and the newborn occurs in case of an initial maternal infection contracted in the second half of pregnancy. The risk of transmission of maternal-fetal-neonatal herpes simplex can be decreased by performing a treatment with antiviral drugs or resorting to a caesarean section in some specific cases.
Infection with herpes simplex is one of the most common sexually transmitted infections. Because the infection is common in women of reproductive age it can be contracted and transmitted to the fetus during pregnancy and the newborn. Herpes simplex virus is an important cause of neonatal infection, which can lead to death or long-term disabilities. Rarely in the uterus, it occurs frequently during the transmission delivery. The greatest risk of transmission to the fetus and the newborn occurs in case of an initial maternal infection contracted in the second half of pregnancy. The risk of transmission of maternal-fetal-neonatal herpes simplex can be decreased by performing a treatment with antiviral drugs or resorting to a caesarean section in some specific cases.
Herpes simplex virus (HSV) cureyên 1 û 2 li çaraliyê cîhanê gelek mirovan vedigirin. Bi gelemperî, vîrus piştî vegirtina çermê di hûcreyên nervê de bêdeng dimîne, lê ew dikare paşê ji nû ve çalak bibe û sedema birînên sar bibe. Carinan, ew sedema pirsgirêkên giran dibe, mîna enfeksiyonên çav, iltîhaba mêjî, an şert û mercên xeternak di nûdayik û mirovên xwedan pergalên berevaniya qels de. Digel ku dermanên heyî alîkariya kontrolkirina enfeksiyonan dikin, xetera berxwedana derman û bandorên alî xemir dimîne. Pêdivîya me dermanên nû hene ku vîrusê çêtir hedef bikin.
Infection with herpes simplex virus (HSV) types 1 and 2 is ubiquitous in the human population. Most commonly, virus replication is limited to the epithelia and establishes latency in enervating sensory neurons, reactivating periodically to produce localized recurrent lesions. However, these viruses can also cause severe disease such as recurrent keratitis leading potentially to blindness, as well as encephalitis, and systemic disease in neonates and immunocompromised patients. Although antiviral therapy has allowed continual and substantial improvement in the management of both primary and recurrent infections, resistance to currently available drugs and long-term toxicity pose a current and future threat that should be addressed through the development of new antiviral compounds directed against new targets.
Infection with herpes simplex virus (HSV) types 1 and 2 is ubiquitous in the human population. Most commonly, virus replication is limited to the epithelia and establishes latency in enervating sensory neurons, reactivating periodically to produce localized recurrent lesions. However, these viruses can also cause severe disease such as recurrent keratitis leading potentially to blindness, as well as encephalitis, and systemic disease in neonates and immunocompromised patients. Although antiviral therapy has allowed continual and substantial improvement in the management of both primary and recurrent infections, resistance to currently available drugs and long-term toxicity pose a current and future threat that should be addressed through the development of new antiviral compounds directed against new targets.
Du cureyên vîrusa herpes simplex hene, cureya 1 (HSV-1) û cureya 2 (HSV-2). HSV-1 bi gelemperî sedema enfeksiyonên li dora devê dike, dema ku HSV-2 bi gelemperî sedema enfeksiyonên zayendî dike. Ew bi têkiliya rasterast bi kesê vegirtî re têne veguheztin. Herpesên zayendî wekî enfeksiyonek zayendî tê dabeş kirin. Dibe ku di dema zayînê de li pêşîkekê belav bibe. Piştî enfeksiyonê, vîrus bi nervên hestî ve tê veguheztin laşên hucreya nervê, li wir ew heya heyatê dimînin. Sedemên dûbarebûnê ev in: kêmbûna fonksiyona parastinê, stres, û ronahiya rojê.
Di pir rewşan de, dermanên antiviral tenê dema ku nîşanên giran in têne girtin. Dermanê antîvîrusê yê rojane dikare ji kesê ku pir caran enfeksiyonê pê re heye were destnîşan kirin. Derzîyek berdest tune û derzîya zozanê pêşî li herpes simplex nagire. Dermankirina bi dermanên antiviral ên wekî acyclovir an valacyclovir dikare giraniya nîşanan kêm bike.
Rêjeyên gerdûnî yên HSV-1 an jî HSV-2 di mezinan de di navbera 60 % û 95 % de ne. HSV-1 bi gelemperî di dema zaroktiyê de tê vegirtin. Nêzîkî 536 mîlyon mirov li cîhanê (16 % ji nifûsê) ji sala 2003‑an de bi HSV-2 vegirtî bûn; di nav jinan de û di cîhanê pêşkeftî de rêjeyek mezintir heye. Piraniya mirovên bi HSV-2 nizanin ku ew vegirtî ne.
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Xwe ji têkiliyên laşî dûr bixin, wek ramûsandina zarokekî, dema ku bilbil hene, ji ber ku têkiliya wan dikare enfeksiyonê li mirovên din belav bike. Divê hûn bêyî vexwarina alkolê bihêlin.
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