Staphylococcal scalded skin syndrome
☆ Di encamên 2022-an de Stiftung Warentest ji Almanyayê, razîbûna xerîdar bi ModelDerm tenê ji şêwirmendên telemedicine drav hindik hindiktir bû.
Pitikek bi Staphylococcal scalded skin syndrome
References
Staphylococcal Scalded Skin Syndrome rewşek e ku çerm ji ber toksînên ku ji hêla hin celeb bakteriyên Staphylococcus ve têne hilberandin dirijîne. Di zarokên ji şeş salî mezintir de ne asayî ye. Di heman demê de ew dikare di mezinên xwedan pergalên parastinê yên qels an pirsgirêkên giran ên gurçikê de jî çêbibe. Taybetmendiya sereke pilingbûna berfireh a çerm piştî iltîhaba ye. Zehmetî ji çend bilbilan bigire heya windabûna çerm a berbelav, ku dikare bibe sedema daketinên giran di germahiya laş û bêîstîqrara tansiyona xwînê de.
Staphylococcal Scalded Skin Syndrome is a disease characterized by denudation of the skin caused by exotoxin producing strains of the Staphylococcus species, typically from a distant site. It usually presents 48 hours after birth and is rare in children older than six years. It may also present in immunocompromised adults or those with severe renal disease. The disorder is characterized by significant exfoliation of skin following cellulitis. The severity may vary from a few blisters to system exfoliation leading to marked hypothermia and hemodynamic instability.
Staphylococcal Scalded Skin Syndrome is a disease characterized by denudation of the skin caused by exotoxin producing strains of the Staphylococcus species, typically from a distant site. It usually presents 48 hours after birth and is rare in children older than six years. It may also present in immunocompromised adults or those with severe renal disease. The disorder is characterized by significant exfoliation of skin following cellulitis. The severity may vary from a few blisters to system exfoliation leading to marked hypothermia and hemodynamic instability.
Staphylococcal scalded skin syndrome (SSSS) and bullous impetigo are infections caused by Staphylococcus aureus. Bullous impetigo is due to the local release of these toxins and thus, often presents with localized skin findings, whereas SSSS is from the systemic spread of these toxins, resulting in a more generalized rash and severe presentation. Both conditions are treated with antibiotics that target S. aureus. These conditions can sometimes be confused with other conditions that result in superficial blistering.
Zarokek 10 mehî ku pozê xwe diherikî û baş nedixwar hat hundur. Bijîşkan gotin ku ew enfeksiyonek nefesa jorîn bû. Du roj şûnda, ew vegeriya ji ber ku ew baş nebû û pirsgirêkên wî yên nû mîna rûyê werimî û çermê devê devê hêrsbûyî hebûn. Di du rojên pêş de, kurik xirabtir bû. Dest û lingên wî werimîbûn û çermê wî dest pê kir. Vegere nexweşxaneyê, bijîjkan li ser rûyê wî û di pelikên çermê wî de xişokek sor dît, ku gava dest lê dixist ew spî dibû. Wan bi staphylococcal scalded skin syndrome (SSSS) teşhîsa wî dan û bi rehê wî dest bi antîbiyotîkan kirin.
A 10-month-old male presented with rhinorrhea and decreased oral intake and was diagnosed with an upper respiratory infection. Two days later, he returned to the clinic due to a lack of improvement and the onset of new symptoms, including facial edema and perioral skin irritation. That evening, he became febrile at 100.4 °F and went to the emergency department at the local children's hospital. No further workup was done and the parents were instructed to continue with the current treatment regimen. Over the next 48 hours, the patient's symptoms worsened with the new onset of bilateral extremity edema and desquamation. The patient was returned to the emergency department. A physical exam was notable for a blanching, desquamating, erythematous rash on the face and creases of the arms, legs, and groin. A positive Nikolsky sign was reported. A clinical diagnosis of staphylococcal scalded skin syndrome (SSSS) was made, and the patient was started on intravenous clindamycin. This case illustrates a severe presentation of SSSS in a pediatric patient, demonstrating the challenges it poses to diagnosis and treatment.
A 10-month-old male presented with rhinorrhea and decreased oral intake and was diagnosed with an upper respiratory infection. Two days later, he returned to the clinic due to a lack of improvement and the onset of new symptoms, including facial edema and perioral skin irritation. That evening, he became febrile at 100.4 °F and went to the emergency department at the local children's hospital. No further workup was done and the parents were instructed to continue with the current treatment regimen. Over the next 48 hours, the patient's symptoms worsened with the new onset of bilateral extremity edema and desquamation. The patient was returned to the emergency department. A physical exam was notable for a blanching, desquamating, erythematous rash on the face and creases of the arms, legs, and groin. A positive Nikolsky sign was reported. A clinical diagnosis of staphylococcal scalded skin syndrome (SSSS) was made, and the patient was started on intravenous clindamycin. This case illustrates a severe presentation of SSSS in a pediatric patient, demonstrating the challenges it poses to diagnosis and treatment.
Keçeke 2-salî li seranserê laşê wê 48 saetan de xirecir xuya bû, li dû kêzikên kêzikan li ser rûyê wê rojek berê. Di dema muayeneyê de, pişkek wê ya berbelav bû ku ji gûzên piçûk ên ku bi hev re digihêjin hev, û dema ku bi sivikî tê şûştin, çermê wê nîşanên nîşana Nikolsky nîşan dide. Ti nîşanekan tunebû ku raşê bandorê li mûkozên wê bike. Testên li ser xwîna wê ti nîşanên enfeksiyonê nedan.
A 2-year-old girl presented a generalised rash with 48 h of evolution, in the context of insect bites on the face on the day before. At observation, she had a generalised micropapular rash with confluent areas and Nikolsky sign. There was no mucosal area affected. Blood cultures were negative.
A 2-year-old girl presented a generalised rash with 48 h of evolution, in the context of insect bites on the face on the day before. At observation, she had a generalised micropapular rash with confluent areas and Nikolsky sign. There was no mucosal area affected. Blood cultures were negative.
Sendrom ji hêla egzotoksînên epîdermolîtîk (exfoliatin) A û B, ku ji hêla S. aureus ve têne derxistin, çêdibe. Pêşbîniya staphylococcal scalded skin syndrome di zarokan de xweş e, di nav 10 rojên dermankirinê de bi tevahî çareser dibe, û bêyî şopên girîng. Lêbelê, staphylococcal scalded skin syndrome divê bi baldarî ji nekrolîza epîdermal a toksîk, ku pêşgotinek nebaş hildigire, were cûda kirin.
○ Nîşanî û dermankirin
Teşhîskirina xelet wekî birînên eczema yên wekî dermatitis atopîk, û serîlêdana rûnê steroîdan birînan zêde dike. Ji kerema xwe dema ku rûnê antîbiyotîk bikar tînin, di zûtirîn dem de li bal bijîşkî bigerin.
#Bacitracin
#First-generation cephalosporins (e.g. Cefradine)
#Bacterial culture
#Third-generation cephalosporins (e.g. Cefditoren Pivoxil)