Acanthosis nigricanshttps://en.wikipedia.org/wiki/Acanthosis_nigricans
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Black pigmentation and rugs in both alis suadeant Acanthosis nigricans.
relevance score : -100.0%
References Acanthosis Nigricans 28613711 NIH
Acanthosis nigricans Est manifestatio cutaneae condicionis subiectae. Saepe apparet in plicas cutis sicut collum, alis, et inguina, apparentes sicut in marginibus obscuris obscuris velutinis. Haec conditio fere cum diabete et resistentia insulinae coniungitur, sed raro, ut cancer intra corpus monstrare posset. Posset etiam ostendere ob quaestiones hormonum vel medicamentum specificum sumere sicut steroids et pilulas partus.
Acanthosis nigricans is a cutaneous manifestation of an underlying condition. It usually develops in skin folds, such as the back of the neck, axilla, and groin, where it presents as velvety hyper-pigmented patches with poorly defined borders. Acanthosis nigricans is most commonly associated with diabetes and insulin resistance, but rarely it can be a sign of internal malignancy. It can also occur with hormone disorders or with the use of certain medications like systemic glucocorticoids and oral contraceptives.
Current treatment options for acanthosis nigricans 30122971 NIH
Acanthosis nigricans (AN) Est communis cutis conditio variis exitibus sanitatis coniuncta sicut resistentia insulina, diabete, dolore magna, carcinomata quaedam, problemata hormonalia et motus ad medicamenta. Agitur de AN versari in quaestionibus salutis subiectae alloquens. Initio, medici reprehendo signa syndrome insulinae resistentiae, quae includit ADIPS, altum cholesterolum, altum sanguinem pressuram, et typus 2 diabete. Medici saepe praecipiunt topical retinoids ut electionem primam curationi, quam adiuvant cum crasso cute. Sed oratio non plene obscuratio. Aliae optiones curationis (salicylic acid, podophyllin, urea, calcipotriol) etiam frequenti applicatione indigent.
Acanthosis nigricans (AN) is a common dermatologic manifestation of systemic disease that is associated with insulin resistance, diabetes mellitus, obesity, internal malignancy, endocrine disorders, and drug reactions. Treatment of AN primarily focuses on resolution of the underlying disease processes causing the velvety, hyperpigmented, hyperkeratotic plaques found on the skin. Initial considerations for the AN workup include evaluating patients for insulin resistance syndrome characterized by obesity, dyslipidemia, hypertension, and diabetes mellitus type II. For cosmetic treatment, topical retinoids are considered the first-line therapy for insulin-resistant AN by modifying keratinization rate. However, topical tretinoin requires application for long durations and improves hyperkeratosis, but not hyperpigmentation. Topical salicylic acid, podophyllin, urea, and calcipotriol also require frequent application, while TCA peels may provide a faster and less time-intense burden.
○ Causae
Plerumque accidit in hominibus quam ante annos XL minores, genere hereditari potest et cum obesitate vel endocrinopathiis coniungitur, ut hypothyroidismus, acromegaly, morbus ovarii polycystici, insulino-repugnans diabete vel morbo Cushing.
○ Curatio OTC Medicamenta
#40% urea cream