Amyloidosis
☆ In anno 2022 Stiftung Warentest ex Germania provenit, satisfactio consumptoria cum ModelDerm paulo minus fuit quam cum consultationibus telemedicinis solutis.
Ordo vultus of Amyloidosis
References
Mulier XXVI annorum venit ad nostram inhaero querentem de SCABROSUS temerarius in cruribus suis quas iam per X annos habebat. Quamvis per cremorem steroidei et cremor tazarotene, temerarius melius non est. Historiam familiarem aliquam non habuit. Cum eam examinavimus, in fronte crurum eius asperae inaequalitates invenimus, quae morbus nomine lichen amyloidosis compositus est.
A 26-year-old woman presented to our clinic with an itchy rash on her legs that had persisted for 10 years. The rash had previously been treated with topical steroids and tazarotene cream, with no improvement. The patient’s family history was noncontributory. A physical examination showed discrete and coalescing hyperkeratotic tan-brown papules on the pretibial surfaces, consistent with lichen amyloidosis.
A 26-year-old woman presented to our clinic with an itchy rash on her legs that had persisted for 10 years. The rash had previously been treated with topical steroids and tazarotene cream, with no improvement. The patient’s family history was noncontributory. A physical examination showed discrete and coalescing hyperkeratotic tan-brown papules on the pretibial surfaces, consistent with lichen amyloidosis.
Lichen Amyloidosis Rara est conditio cutis cum pertinaci pruritu ignotae causae coniuncta. Plerumque ostendit racemos elevatos, inaequaliter decoloratos in superficie cutis exterioris. Lichen Amyloidosis Solet in hominibus aetatis 50 ad 60 apparet et pro dolor, nulla cura est. Curationes nunc praesto non bene operari solent.
Lichen Amyloidosis (LA) is an uncommon, primary cutaneous amyloidosis associated with chronic, idiopathic pruritus. Clinical presentation of LA includes skin colored to hyperpigmented, papules coalescing into plaques with a rippled appearance on the extensors.1 LA most commonly presents in the fifth to sixth decade of life and has no curative treatments. Overall response to current therapies is poor.
Lichen Amyloidosis (LA) is an uncommon, primary cutaneous amyloidosis associated with chronic, idiopathic pruritus. Clinical presentation of LA includes skin colored to hyperpigmented, papules coalescing into plaques with a rippled appearance on the extensors.1 LA most commonly presents in the fifth to sixth decade of life and has no curative treatments. Overall response to current therapies is poor.
Lichen amyloidosis SCABROSUS cutis diuturna est conditio. Notum est propter racemos labefecit incrassatos maxime in dorso, tibiis, femoribus, et armis repertis. Cum sub microscopio examinatus est, Lichen amyloidosis ostendit fabricam amyloideae in summo strato cutis cum crasso et dilatatione cutis exterioris. Quamquam causa exacta Lichen amyloidosis nondum plene cognita est, studia priora eam cum factoribus quasi frictione vel frictione in cute, mortem cellae, infectiones virales, inter alia coniunxerunt. Lichen amyloidosis cum pluribus aliis condicionibus cutis coniunctum esse videtur (atopic dermatitis, lichen planus, mycosis fungoides) .
Lichen amyloidosis (LA) is a chronic pruritic skin disorder characterized by multiple grouped hyperkeratotic papules, predominantly located on the back, shins, thighs, and arms. Histological analysis of LA shows amyloid deposition in the papillary dermis and hyperkeratosis and acanthosis of the epidermis. The exact pathogenesis of LA has not yet been elucidated; however, prior reports have implicated frictional epidermal damage, apoptosis, viral infection, and many other triggers. LA is reportedly associated with several skin disorders, including atopic dermatitis (AD), lichen planus, and mycosis fungoides.
Lichen amyloidosis (LA) is a chronic pruritic skin disorder characterized by multiple grouped hyperkeratotic papules, predominantly located on the back, shins, thighs, and arms. Histological analysis of LA shows amyloid deposition in the papillary dermis and hyperkeratosis and acanthosis of the epidermis. The exact pathogenesis of LA has not yet been elucidated; however, prior reports have implicated frictional epidermal damage, apoptosis, viral infection, and many other triggers. LA is reportedly associated with several skin disorders, including atopic dermatitis (AD), lichen planus, and mycosis fungoides.
○ Diagnosis et curatio
#Electrophoresis of blood or urine
#Skin biopsy