Poikiloderma
https://en.wikipedia.org/wiki/Poikiloderma
☆ ໃນປີ 2022 Stiftung Warentest ຜົນໄດ້ຮັບຈາກເຢຍລະມັນ, ຄວາມພໍໃຈຂອງຜູ້ບໍລິໂພກກັບ ModelDerm ແມ່ນຕໍ່າກວ່າການປຶກສາຫາລືທາງດ້ານການປິ່ນປົວທາງໂທລະສັບເລັກນ້ອຍເທົ່ານັ້ນ. relevance score : -100.0%
References
Diagnosis and Differential Diagnosis of Poikiloderma of Civatte: A Dermoscopy Cohort Study 36892344 NIH
Poikiloderma of Civatte ເປັນສະພາບຜິວໜັງທົ່ວໄປທີ່ສ່ວນຫຼາຍຈະປາກົດຢູ່ຄໍ ແລະໃບໜ້າ, ໂດຍສະເພາບໃນຜູ່ຍິງທີ່ມີຜິວເນື້ອສີຂາວ (fair‑skinned individuals), ອາຍຸຫຼັງໝົດປະຈຳເດືອນ (postmenopausal women). ມັນສະແດງໃຫ້ເຫັນການປະສົມຂອງ telangiectasia, hyperpigmentation ແລະ atrophy ທີ່ມີຮູບແບບເຣຕິກູລາຣ (reticular pattern). ດ້ວຍປົກກະຕິ, ມັນມີຜົນກະທົບຕໍ່ພື້ນທີ່ທີ່ແສງແດດ (sun‑exposed areas) ເຊັ່ນ: ຄໍ, ຫນ້າເອິກ, ແລະ ບ່ອນທີ່ບໍ່ໄດ້ຮັບແສງ (non‑sun‑exposed areas). Poikiloderma of Civatte ສາມາດຈັດປະເພດໂດຍອີງຕາມລັກສະນະຕົ້ນຕໍ: erythemato‑telangiectatic, pigmented, ຫຼື mixed clinical types. ສາເຫດທີ່ແນ່ນອນບໍ່ແຈ້ງແນ່ນອນແຕ່ປະກອບດ້ວຍ: exposure to ultraviolet radiation, hormonal changes of menopause, contact sensitization to perfumes and cosmetics, ແລະ normal ageing. ການວິນິດສັບສາມາດກວດພິສູດດ້ວຍການກວດສອບພາບພາຍໃນ (histology) ແລະ ມັນມັກຈະຮ້າຍແຮງຂຶ້ນຊ້າໆຕາມເວລາ (slowly progressive).
Poikiloderma of Civatte (PC) is a rather common benign dermatosis of the neck and face, mainly affecting fair-skinned individuals, especially postmenopausal females. It is characterized by a combination of a reticular pattern of linear telangiectasia, mottled hyperpigmentation and superficial atrophy. Clinically, it involves symmetrically sun-exposed areas of the face, the neck, and the V-shaped area of the chest, invariably sparing the anatomically shaded areas. Depending on the prevalent clinical feature, PC can be classified into erythemato-telangiectatic, pigmented, and mixed clinical types. The etiopathogenesis of PC is incompletely understood. Exposure to ultraviolet radiation, hormonal changes of menopause, contact sensitization to perfumes and cosmetics, and normal ageing have been incriminated. The diagnosis is usually clinical and can be confirmed by histology, which is characteristic, but not pathognomonic. The course is slowly progressive and irreversible, often causing significant cosmetic disfigurement.