Hidradenitis suppurativa
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Hidradenitis suppurativa (waahanga I) i roto i te keokeo. He keehi tino ngawari tenei o Hidradenitis suppurativa.
relevance score : -100.0%
ReferencesKo te Hidradenitis suppurativa he mate kiri e mau tonu ana, ka hoki mai ano, ka tino pa ki tō oranga. Na te mumura i roto i ngā nodules, he maha ngā wā ka pā ki ngā mate huakita. Ko te nuinga o ngā wā ka tirohia e ngā taakuta mā te titiro ki ngā momo tuwhenua kei a koe (pēnei i te nodules, abscesses, sinus tracts rānei), kei hea (te nuinga o ngā kopa kiri), me te maha o te hokinga mai me te roa o te piri.
Hidradenitis suppurativa is a chronic, recurrent, and debilitating skin condition. It is an inflammatory disorder of the follicular epithelium, but secondary bacterial infection can often occur. The diagnosis is made clinically based on typical lesions (nodules, abscesses, sinus tracts), locations (skin folds), and nature of relapses and chronicity.
Hidradenitis suppurativa is a chronic, recurrent, and debilitating skin condition. It is an inflammatory disorder of the follicular epithelium, but secondary bacterial infection can often occur. The diagnosis is made clinically based on typical lesions (nodules, abscesses, sinus tracts), locations (skin folds), and nature of relapses and chronicity.
Ko ngā rāhui kōmaka (antibiotics) me ngā rāhui kore‑kōmaka e whakamahia ana mō te mate ngawari ka taea te whakakotahi me te rongoā kōmaka me te pokanga mō te mate ngawari ki te mate kino. Ngā rangahau tata nei ka whakaatu i ētahi atu taunakitanga mō te whakamahi corticosteroids i werohia tika ki roto i ngā whiu mō te Hidradenitis suppurativa (HS) flare‑ups me ngā whiu rohe. I tua atu, he taunakitanga e kī ana ko te whakamahi i ngā tetracyclines anake ka rite te whai hua ki te whakakotahi i te clindamycin me te rifampicin.
Non-biologic and non-procedural treatments are often used as monotherapy for mild disease and can be used in conjunction with biologic therapy and surgery for moderate to severe disease. Recent studies highlighted in this review add support for the use of intralesional corticosteroids for HS flares and localized lesions, and there is evidence that monotherapy with tetracyclines may be as effective as the clindamycin/rifampicin combination.
Non-biologic and non-procedural treatments are often used as monotherapy for mild disease and can be used in conjunction with biologic therapy and surgery for moderate to severe disease. Recent studies highlighted in this review add support for the use of intralesional corticosteroids for HS flares and localized lesions, and there is evidence that monotherapy with tetracyclines may be as effective as the clindamycin/rifampicin combination.
He maha ngā rongoā e whakamahia ana mō te hidradenitis suppurativa, tae atu ki ngā antibiotika (antibiotics), retinoids, antiandrogens, rongoā immunosuppressive (immunosuppressive medications), rongoā anti‑inflammatory (anti‑inflammatory medications), me te radiotherapy mō ngā whiu (skin lesions). Ko ngā rongoā tino pai ko te adalimumab me te laser therapy. Ko te surgery, ko te simple excision, ko te complete local excision rānei me te skin grafting, te mea pai mō ngā keehi tino nui (severe, advanced cases), kāore e aro pai ki etahi atu maimoatanga (does not respond well to other treatments).
Many treatments are used for hidradenitis suppurativa, including antibiotics, retinoids, antiandrogens, immune-suppressing drugs, anti-inflammatory medications, and radiotherapy for early lesions. The top recommended treatments are adalimumab and laser therapy. Surgery, either simple excision or complete local excision with skin grafting, is the preferred option for severe, advanced cases that don't respond well to other treatments.
Many treatments are used for hidradenitis suppurativa, including antibiotics, retinoids, antiandrogens, immune-suppressing drugs, anti-inflammatory medications, and radiotherapy for early lesions. The top recommended treatments are adalimumab and laser therapy. Surgery, either simple excision or complete local excision with skin grafting, is the preferred option for severe, advanced cases that don't respond well to other treatments.
Ko te tino take kaore i te marama, engari e whakaponohia ana he huinga o nga ahuatanga ira me te taiao. Tata ki te hautoru o nga tangata e mate ana he mema whanau kua pa. Ko etahi atu mea morearea ko te momona me te kai paipa. Ehara i te mea he mate kōkare (infection), he kino te akuaku.
Kaore he rongoa e mohiotia ana. Ko te tapahi i nga whiu kia pai ai te rere kaore he painga nui. Ahakoa e whakamahia nuitia ana nga pāhiri (antibiotics), he iti noa nga taunakitanga mo te whakamahi. Ka taea hoki te whakamatautau i nga rongoā whakaiti pūmua (immunosuppressive medications). I te hunga e mate kino atu ana, ka taea te rongoā rererangi (laser therapy), te rongoā whakakore (surgery) ranei hei tango i te kiri kua pangia. He onge, ka puta he mate kanikani kiri (skin cancer).
Mena ka whakauruhia nga whakawhāiti (lesions) ngawari o hidradenitis suppurativa, ko te whakatau tata o tona kaimahi (prevalence) mai i te 1‑4 % o te taupori. Ko nga wahine e toru nga wa ka nui ake te mate i nga tane. Ko te nuinga o nga wa ka timata te ahua i te taiohi.