Vasculitishttps://en.wikipedia.org/wiki/Vasculitis
Ko te Vasculitis he roopu o ngā māte e whakangaro ana i ngā ōko toto nā te mumura. Ka taea te whakarōpūhia te Vasculitis e te take, te wāhi, te momo ōko, te rahi rānei o te waka. Ko ngā whakamātautau taiwhanga me te koiora kiri ka hiahiatia kia kitea ngā take o raro. Ko ngā maimoatanga he tikanga ki te whakamutu i te mumura me te pehi i te pūnaha māte. Ko te tikanga, ka whakamahia ngā corticosteroids pērā i te prednisone.

Tataritanga
Ko te Vasculitis iti ki te kiri ka pai ake i te wā. Heoi anō, ka mahia ngā whakamātautau toto me te mimi kia kitea te māte o te pūnaha, te māte autoimmune rānei.

Maimoatanga - Rongoa OTC
Mēnā ka whāiti te vasculitis ki te kiri, kāore e pangia e ētahi atu whekau, ka taea te whakamahi i te hinu steroid.
#OTC steroid ointment
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  • Ko ētahi atu mate kino (mate autoimmune) e pā ana ki te vasculitis, me te whakakore.
  • He pikitia anamata o te vasculitis o te waewae. Ka taea te mahi whakamātautau mimi ki te tirotiro mō ngā māuiui o te mahi whatukuhu.
  • Livedo vasculopathy
  • Purpura
  • Henoch‑Schönlein purpura
References An aetiological & clinicopathological study on cutaneous vasculitis 22382191 
NIH
Of the 61 patients studied, hypersensitivity vasculitis (HSV) [23 (37.7%)] and Henoch Schonlein purpura (HSP) [16 (26.2%)] were the two most common forms. Systemic involvement was seen in 32 (52.45%) patients. Drugs were implicated in 12 (19.7%) cases, infections in 7 (11.4%) and connective tissue disorders in 4 (6.5%) cases. No association was seen between history of drug intake and tissue eosinophilia and also between histologically severe vasculitis and clinical severity.
 Leukocytoclastic Vasculitis 29489227 
NIH
Ko te Leukocytoclastic vasculitis he momo mumura kiri e pa ana ki nga oko toto iti i nga papa hohonu o te kiri. Ka puta mo te kore mohio he take, ka hono ranei ki nga mate, ki nga pukupuku, ki nga mate autoimmune, ki nga rongoa ranei. Ko nga tohu tohu ko nga waahi whero, papura ranei i runga i nga waewae, te urunga o nga oko iti, a, i roto i te 30 paiheneti o nga keehi, ka pangia etahi atu wahanga o te tinana. Ko te nuinga o nga keehi ka maarama ake i roto i etahi wiki ki nga marama. He rereke nga maimoatanga i runga i te kaha o te mate, mai i te whakaheke haere i nga corticosteroids-waha ki te whakamahi i etahi atu raau taero hei whakaheke i te mumura kaore he steroids.
Leukocytoclastic vasculitis is a cutaneous, small-vessel vasculitis of the dermal capillaries and venules. This condition can be idiopathic or can be associated with infections, neoplasms, autoimmune disorders, and drugs. Key clinical features of leukocytoclastic vasculitis include palpable purpura on the lower extremity, small vessel involvement, and, in about 30 percent of individuals, extracutaneous involvement. Most cases of idiopathic cutaneous, small vessel vasculitis are self-limited with 90 percent of cases resolving in weeks to months of onset. Otherwise, treatment depends on the severity of disease and can range from an oral corticosteroid taper to various steroid-sparing immunosuppressive agents.