Postinflammatory hyperpigmentation - Hiperpigmentasi Postinflammatory
Hiperpigmentasi Postinflammatory (Postinflammatory hyperpigmentation) ialah keadaan kulit yang dicirikan oleh peningkatan pigmen dalam kulit berikutan keradangan kulit. hiperpigmentasi postinflammatory (postinflammatory hyperpigmentation) boleh disebabkan oleh pendedahan matahari yang berpanjangan, keradangan atau kecederaan kulit yang lain, termasuk yang berkaitan dengan jerawat. Orang yang mempunyai warna kulit yang lebih gelap biasanya lebih terdedah kepada hiperpigmentasi dengan pendedahan matahari yang berlebihan.
☆ Dalam keputusan Stiftung Warentest 2022 dari Jerman, kepuasan pengguna terhadap ModelDerm hanya lebih rendah sedikit berbanding dengan perundingan teleperubatan berbayar.
References
Postinflammatory hyperpigmentation (PIH) adalah masalah kulit yang kerap berlaku selepas keradangan atau kecederaan kulit. Ia cenderung untuk bertahan lama dan lebih teruk pada orang yang mempunyai warna kulit yang lebih gelap (Fitzpatrick skin types III–VI) . Walaupun ia sering menjadi lebih baik dengan sendirinya, ini boleh mengambil sedikit masa, jadi rawatan sering diperlukan untuk masa yang lama. Menggabungkan rawatan yang berbeza berkesan.
Postinflammatory hyperpigmentation (PIH) is a common acquired cutaneous disorder occurring after skin inflammation or injury. It is chronic and is more common and severe in darker-skinned individuals (Fitzpatrick skin types III–VI). While the condition typically improves spontaneously, this process can take months to years, necessitating prolonged treatment. Combination therapy is the most effective.
Postinflammatory hyperpigmentation (PIH) is a common acquired cutaneous disorder occurring after skin inflammation or injury. It is chronic and is more common and severe in darker-skinned individuals (Fitzpatrick skin types III–VI). While the condition typically improves spontaneously, this process can take months to years, necessitating prolonged treatment. Combination therapy is the most effective.
Postinflammatory hyperpigmentation adalah sekuela biasa keradangan kulit. Ia cenderung untuk menjejaskan individu berkulit gelap dengan lebih teruk dan kerap. Kajian menunjukkan bahawa isu seperti postinflammatory hyperpigmentation adalah antara sebab utama mengapa orang yang mempunyai warna kulit yang lebih gelap mendapatkan rawatan dermatologi. Rawatan awal adalah penting untuk menyelesaikan postinflammatory hyperpigmentation dan biasanya bermula dengan menguruskan keadaan keradangan awal. Baris pertama rawatan biasanya melibatkan penggunaan agen topikal yang mencerahkan kulit bersama-sama dengan pelindung matahari untuk perlindungan. Ejen ini, seperti hydroquinone, azelaic acid, kojic acid, arbutin, licorice extracts , boleh mengurangkan pigmentasi berlebihan secara berkesan. Selain itu, retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, soy juga digunakan sebagai agen depigmentasi, dengan rawatan baru muncul. Walaupun rawatan topikal biasanya berkesan untuk hiperpigmentasi paras permukaan, prosedur (laser, chemical peel) mungkin diperlukan untuk kes degil. Adalah penting untuk berhati-hati dengan rawatan ini untuk mengelakkan kerengsaan dan kemerosotan postinflammatory hyperpigmentation.
Postinflammatory hyperpigmentation is a common sequelae of inflammatory dermatoses that tends to affect darker skinned patients with greater frequency and severity. Epidemiological studies show that dyschromias, including postinflammatory hyperpigmentation, are among the most common reasons darker racial/ethnic groups seek the care of a dermatologist. The treatment of postinflammatory hyperpigmentation should be started early to help hasten its resolution and begins with management of the initial inflammatory condition. First-line therapy typically consists of topical depigmenting agents in addition to photoprotection including a sunscreen. Topical tyrosinase inhibitors, such as hydroquinone, azelaic acid, kojic acid, arbutin, and certain licorice extracts, can effectively lighten areas of hypermelanosis. Other depigmenting agents include retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, and soy with a number of emerging therapies on the horizon. Topical therapy is typically effective for epidermal postinflammatory hyperpigmentation; however, certain procedures, such as chemical peeling and laser therapy, may help treat recalcitrant hyperpigmentation. It is also important to use caution with all of the above treatments to prevent irritation and worsening of postinflammatory hyperpigmentation.
Postinflammatory hyperpigmentation is a common sequelae of inflammatory dermatoses that tends to affect darker skinned patients with greater frequency and severity. Epidemiological studies show that dyschromias, including postinflammatory hyperpigmentation, are among the most common reasons darker racial/ethnic groups seek the care of a dermatologist. The treatment of postinflammatory hyperpigmentation should be started early to help hasten its resolution and begins with management of the initial inflammatory condition. First-line therapy typically consists of topical depigmenting agents in addition to photoprotection including a sunscreen. Topical tyrosinase inhibitors, such as hydroquinone, azelaic acid, kojic acid, arbutin, and certain licorice extracts, can effectively lighten areas of hypermelanosis. Other depigmenting agents include retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, and soy with a number of emerging therapies on the horizon. Topical therapy is typically effective for epidermal postinflammatory hyperpigmentation; however, certain procedures, such as chemical peeling and laser therapy, may help treat recalcitrant hyperpigmentation. It is also important to use caution with all of the above treatments to prevent irritation and worsening of postinflammatory hyperpigmentation.
