Herpes simplex
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Herpes simplex fuq ix-xoffa.
Herpes simplex ― L-infezzjoni tal-herpes simplex fis-swaba' hija aktar komuni fi tfal żgħar milli fl-adulti.
References
L-infezzjoni HSV-1 timxi 'l quddiem permezz ta' infezzjoni primarja ta 'ċelluli epiteljali, segwita minn latency, prinċipalment fin-newroni, u riattivazzjoni. HSV-1 komunement jikkawża eruzzjonijiet vesikulari inizjali u rikorrenti, prinċipalment fuq il-ħalq u l-mukoża ġenitali. Il-manifestazzjonijiet tiegħu jvarjaw minn herpes orolabjali għal kundizzjonijiet varji bħal follikulite erpetika, infezzjonijiet tal-ġilda, involviment okulari, u każijiet severi bħall-enċefalite tal-herpes. It-terapija antivirali tgħin biex timmaniġġja l-infezzjoni tal-HSV.
Herpes simplex virus type 1 (HSV-1) is a member of the Alphaherpesviridae subfamily. Its structure is composed of linear dsDNA, an icosahedral capsid that is 100 to 110 nm in diameter, with a spikey envelope. In general, the pathogenesis of HSV-1 infection follows a cycle of primary infection of epithelial cells, latency primarily in neurons, and reactivation. HSV-1 is responsible for establishing primary and recurrent vesicular eruptions, primarily in the orolabial and genital mucosa. HSV-1 infection has a wide variety of presentations, including orolabial herpes, herpetic sycosis (HSV folliculitis), herpes gladiatorum, herpetic whitlow, ocular HSV infection, herpes encephalitis, Kaposi varicelliform eruption (eczema herpeticum), and severe or chronic HSV infection. Antiviral therapy limits the course of HSV infection.
Herpes simplex virus type 1 (HSV-1) is a member of the Alphaherpesviridae subfamily. Its structure is composed of linear dsDNA, an icosahedral capsid that is 100 to 110 nm in diameter, with a spikey envelope. In general, the pathogenesis of HSV-1 infection follows a cycle of primary infection of epithelial cells, latency primarily in neurons, and reactivation. HSV-1 is responsible for establishing primary and recurrent vesicular eruptions, primarily in the orolabial and genital mucosa. HSV-1 infection has a wide variety of presentations, including orolabial herpes, herpetic sycosis (HSV folliculitis), herpes gladiatorum, herpetic whitlow, ocular HSV infection, herpes encephalitis, Kaposi varicelliform eruption (eczema herpeticum), and severe or chronic HSV infection. Antiviral therapy limits the course of HSV infection.
Herpes simplex virus type 2 (HSV-2) hija infezzjoni mifruxa, li taffettwa madwar 22% tal-adulti li għandhom 12-il sena 'l fuq, li jammontaw għal 45 miljun adult fl-Istati Uniti. Filwaqt li l-HSV-1 tipikament jikkawża feriti orali, jista 'jwassal ukoll għal leżjonijiet ġenitali. Madankollu, meta l-pazjenti jkollhom feriti ġenitali, HSV-2 normalment ikun it-tħassib ewlieni. Is-sintomi tat-tifqigħat tal-HSV-2 ħafna drabi huma vagi, bħal ħakk u irritazzjoni ġenitali, li jistgħu jdewmu d-dijanjosi u t-trattament. Dan id-dewmien jista' jirriżulta f'aktar trażmissjoni lil individwi mhux infettati.
Herpes simplex virus type 2 (HSV-2) continues to be a common infection, affecting approximately 22% of adults ages 12 and older, representing 45 million adults in the United States alone. While HSV-1 often affects the perioral region and can be known to cause genital lesions, HSV-2 is more commonly the consideration when patients present with genital lesions. Despite this, most outbreaks of the infection will present with nonspecific symptoms such as genital itching, irritation, and excoriations, which may cause diagnosis and treatment to be delayed. As a result, further exposure to uninfected individuals may occur.
Herpes simplex virus type 2 (HSV-2) continues to be a common infection, affecting approximately 22% of adults ages 12 and older, representing 45 million adults in the United States alone. While HSV-1 often affects the perioral region and can be known to cause genital lesions, HSV-2 is more commonly the consideration when patients present with genital lesions. Despite this, most outbreaks of the infection will present with nonspecific symptoms such as genital itching, irritation, and excoriations, which may cause diagnosis and treatment to be delayed. As a result, further exposure to uninfected individuals may occur.
Herpes simplex virus (HSV) komunement tikkawża infezzjonijiet bħal herpes ġenitali u feriti fl-żagħżagħ u fl-adulti. Meta l-HSV jinfetta tarbija fl-ewwel 4-6 ġimgħat tal-ħajja, jista' jirriżulta f'mard sever b'konsegwenzi serji. Id-dijanjosi ta 'malajr ta' infezzjoni ta 'HSV tat-twelid hija kruċjali biex twaqqaf il-marda milli tmur għall-agħar, u tevita kwistjonijiet newroloġiċi (anke mewt) .
Herpes simplex virus (HSV), a member of the Herpesviridae family, is a well-known cause of infections including genital herpes and herpes labialis in the adolescent and adult population. Transmission of HSV infection to an infant during the first 4-6 weeks of life can lead to devastating disease with the potential for poor outcomes. Early diagnosis is imperative when evaluating neonatal HSV infection in order to prevent further disease progression, neurological complications, and even death.
Herpes simplex virus (HSV), a member of the Herpesviridae family, is a well-known cause of infections including genital herpes and herpes labialis in the adolescent and adult population. Transmission of HSV infection to an infant during the first 4-6 weeks of life can lead to devastating disease with the potential for poor outcomes. Early diagnosis is imperative when evaluating neonatal HSV infection in order to prevent further disease progression, neurological complications, and even death.
Herpes simplex infezzjoni hija komuni ħafna u tista 'tgħaddi minn nisa tqal lit-trabi tagħhom. Dan il-virus jista 'jikkawża problemi serji ta' saħħa jew saħansitra mewt fi trabi tat-twelid. Filwaqt li huwa rari waqt it-tqala stess, ħafna drabi jiġri waqt it-twelid. Ir-riskju huwa l-ogħla jekk l-omm tiġi infettata fl-aħħar stadji tat-tqala. Madankollu, dan ir-riskju jista 'jitnaqqas bl-użu ta' mediċini antivirali jew billi tagħżel C-section f'ċerti sitwazzjonijiet.
Infection with herpes simplex is one of the most common sexually transmitted infections. Because the infection is common in women of reproductive age it can be contracted and transmitted to the fetus during pregnancy and the newborn. Herpes simplex virus is an important cause of neonatal infection, which can lead to death or long-term disabilities. Rarely in the uterus, it occurs frequently during the transmission delivery. The greatest risk of transmission to the fetus and the newborn occurs in case of an initial maternal infection contracted in the second half of pregnancy. The risk of transmission of maternal-fetal-neonatal herpes simplex can be decreased by performing a treatment with antiviral drugs or resorting to a caesarean section in some specific cases.
Infection with herpes simplex is one of the most common sexually transmitted infections. Because the infection is common in women of reproductive age it can be contracted and transmitted to the fetus during pregnancy and the newborn. Herpes simplex virus is an important cause of neonatal infection, which can lead to death or long-term disabilities. Rarely in the uterus, it occurs frequently during the transmission delivery. The greatest risk of transmission to the fetus and the newborn occurs in case of an initial maternal infection contracted in the second half of pregnancy. The risk of transmission of maternal-fetal-neonatal herpes simplex can be decreased by performing a treatment with antiviral drugs or resorting to a caesarean section in some specific cases.
Herpes simplex virus (HSV) tipi 1 u 2 jinfettaw ħafna nies madwar id-dinja. Normalment, il-virus jibqa’ kwiet fiċ-ċelloli tan-nervituri wara li jinfetta l-ġilda, iżda jista’ jerġa’ jattiva aktar tard, u jikkawża feriti. Xi drabi, iwassal għal problemi serji bħal infezzjonijiet fl-għajnejn, infjammazzjoni tal-moħħ, jew kundizzjonijiet perikolużi fi trabi tat-twelid u nies b'sistemi immuni dgħajfa. Filwaqt li l-mediċini attwali jgħinu biex jikkontrollaw l-infezzjonijiet, ir-riskju ta’ reżistenza għall-mediċini u l-effetti sekondarji jibqa’ ta’ tħassib. Għandna bżonn mediċini ġodda biex nimmiraw aħjar il-virus.
Infection with herpes simplex virus (HSV) types 1 and 2 is ubiquitous in the human population. Most commonly, virus replication is limited to the epithelia and establishes latency in enervating sensory neurons, reactivating periodically to produce localized recurrent lesions. However, these viruses can also cause severe disease such as recurrent keratitis leading potentially to blindness, as well as encephalitis, and systemic disease in neonates and immunocompromised patients. Although antiviral therapy has allowed continual and substantial improvement in the management of both primary and recurrent infections, resistance to currently available drugs and long-term toxicity pose a current and future threat that should be addressed through the development of new antiviral compounds directed against new targets.
Infection with herpes simplex virus (HSV) types 1 and 2 is ubiquitous in the human population. Most commonly, virus replication is limited to the epithelia and establishes latency in enervating sensory neurons, reactivating periodically to produce localized recurrent lesions. However, these viruses can also cause severe disease such as recurrent keratitis leading potentially to blindness, as well as encephalitis, and systemic disease in neonates and immunocompromised patients. Although antiviral therapy has allowed continual and substantial improvement in the management of both primary and recurrent infections, resistance to currently available drugs and long-term toxicity pose a current and future threat that should be addressed through the development of new antiviral compounds directed against new targets.
Hemm żewġ tipi ta' virus herpes simplex , tip 1 (HSV-1) u tip 2 (HSV-2). HSV-1 jikkawża aktar komunement infezzjonijiet madwar il-ħalq filwaqt li HSV-2 jikkawża aktar komunement infezzjonijiet ġenitali. Huma trażmessi b'kuntatt dirett mal-individwu infettat. L-herpes ġenitali huwa kklassifikat bħala infezzjoni trażmessa sesswalment. Jista 'jinfirex lil tarbija waqt il-ħlas. Wara l-infezzjoni, il-viruses jiġu ttrasportati tul in-nervituri sensorji lejn il-korpi taċ-ċelluli tan-nervituri, fejn jgħixu tul il-ħajja. Kawżi ta 'rikorrenza jistgħu jinkludu: tnaqqis fil-funzjoni immuni, stress, u espożizzjoni għad-dawl tax-xemx.
F'ħafna każijiet, mediċini antivirali jittieħdu biss meta s-sintomi jkunu severi. Medikazzjoni antivirali ta 'kuljum tista' tiġi preskritta lil xi ħadd li għandu infezzjoni frekwenti ħafna. M'hemm l-ebda vaċċin disponibbli u l-vaċċin ta 'ħruq ta' Sant'Antnin ma jipprevjenix l-herpes simplex. Trattamenti b'medikazzjoni antivirali bħal aciclovir jew valaciclovir jistgħu jnaqqsu s-severità tas-sintomi.
Ir-rati madwar id-dinja ta' jew HSV-1 jew HSV-2 huma bejn 60% u 95% fl-adulti. HSV-1 normalment ikun infettat matul it-tfulija. Huwa stmat li 536 miljun ruħ madwar id-dinja (16% tal-popolazzjoni) kienu infettati bl-HSV-2 mill-2003 b'rati akbar fost in-nisa u dawk fid-dinja li qed tiżviluppa. Ħafna nies b'HSV-2 ma jirrealizzawx li huma infettati.
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