Herpes simplex
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Herpes simplex pa milomo.
Herpes simplex ― Matenda a Herpes simplex pa zala amapezeka kwambiri mwa ana aang'ono kusiyana ndi akuluakulu.
References
Matenda a HSV-1 amapita patsogolo kudzera mu matenda oyamba a maselo a epithelial, otsatiridwa ndi latency, makamaka mu neurons, ndi kubwezeretsanso. HSV-1 nthawi zambiri imayambitsa kuphulika koyambirira komanso kobwerezabwereza kwa vesicular, makamaka pakamwa ndi mucosa yakumaliseche. Mawonetseredwe ake amachokera ku orolabial herpes kupita kuzinthu zosiyanasiyana monga herpetic folliculitis, matenda a pakhungu, kukhudzidwa kwa maso, ndi milandu yoopsa ngati nsungu encephalitis. Chithandizo cha antiviral chimathandizira kuthana ndi matenda a HSV.
Herpes simplex virus type 1 (HSV-1) is a member of the Alphaherpesviridae subfamily. Its structure is composed of linear dsDNA, an icosahedral capsid that is 100 to 110 nm in diameter, with a spikey envelope. In general, the pathogenesis of HSV-1 infection follows a cycle of primary infection of epithelial cells, latency primarily in neurons, and reactivation. HSV-1 is responsible for establishing primary and recurrent vesicular eruptions, primarily in the orolabial and genital mucosa. HSV-1 infection has a wide variety of presentations, including orolabial herpes, herpetic sycosis (HSV folliculitis), herpes gladiatorum, herpetic whitlow, ocular HSV infection, herpes encephalitis, Kaposi varicelliform eruption (eczema herpeticum), and severe or chronic HSV infection. Antiviral therapy limits the course of HSV infection.
Herpes simplex virus type 1 (HSV-1) is a member of the Alphaherpesviridae subfamily. Its structure is composed of linear dsDNA, an icosahedral capsid that is 100 to 110 nm in diameter, with a spikey envelope. In general, the pathogenesis of HSV-1 infection follows a cycle of primary infection of epithelial cells, latency primarily in neurons, and reactivation. HSV-1 is responsible for establishing primary and recurrent vesicular eruptions, primarily in the orolabial and genital mucosa. HSV-1 infection has a wide variety of presentations, including orolabial herpes, herpetic sycosis (HSV folliculitis), herpes gladiatorum, herpetic whitlow, ocular HSV infection, herpes encephalitis, Kaposi varicelliform eruption (eczema herpeticum), and severe or chronic HSV infection. Antiviral therapy limits the course of HSV infection.
Herpes simplex virus type 2 (HSV-2) ndi matenda ofala, okhudza pafupifupi 22% ya akuluakulu azaka 12 ndi kupitilira apo, okwana 45 miliyoni akulu ku United States. Ngakhale kuti HSV-1 nthawi zambiri imayambitsa zilonda zam'kamwa, imatha kuyambitsa zilonda zam'mimba. Komabe, pamene odwala ali ndi zotupa kumaliseche, HSV-2 nthawi zambiri imakhala yodetsa nkhawa kwambiri. Zizindikiro za kuphulika kwa HSV-2 nthawi zambiri zimakhala zosamveka bwino, monga kuyabwa ndi kukwiya kwa maliseche, zomwe zingachedwetse kuzindikira ndi kulandira chithandizo. Kuchedwa kumeneku kungayambitse kufalikira kwa anthu omwe alibe kachilomboka.
Herpes simplex virus type 2 (HSV-2) continues to be a common infection, affecting approximately 22% of adults ages 12 and older, representing 45 million adults in the United States alone. While HSV-1 often affects the perioral region and can be known to cause genital lesions, HSV-2 is more commonly the consideration when patients present with genital lesions. Despite this, most outbreaks of the infection will present with nonspecific symptoms such as genital itching, irritation, and excoriations, which may cause diagnosis and treatment to be delayed. As a result, further exposure to uninfected individuals may occur.
Herpes simplex virus type 2 (HSV-2) continues to be a common infection, affecting approximately 22% of adults ages 12 and older, representing 45 million adults in the United States alone. While HSV-1 often affects the perioral region and can be known to cause genital lesions, HSV-2 is more commonly the consideration when patients present with genital lesions. Despite this, most outbreaks of the infection will present with nonspecific symptoms such as genital itching, irritation, and excoriations, which may cause diagnosis and treatment to be delayed. As a result, further exposure to uninfected individuals may occur.
Herpes simplex virus (HSV) nthawi zambiri amayambitsa matenda monga maliseche ndi zilonda zozizira kwa achinyamata ndi akulu. Pamene HSV imayambitsa khanda mkati mwa masabata 4-6 a moyo, zimatha kudwala kwambiri ndi zotsatira zake zoopsa. Kuzindikira msanga matenda a HSV akhanda ndikofunikira kuti matendawa asapitirire, kuteteza matenda amitsempha (ngakhale imfa) .
Herpes simplex virus (HSV), a member of the Herpesviridae family, is a well-known cause of infections including genital herpes and herpes labialis in the adolescent and adult population. Transmission of HSV infection to an infant during the first 4-6 weeks of life can lead to devastating disease with the potential for poor outcomes. Early diagnosis is imperative when evaluating neonatal HSV infection in order to prevent further disease progression, neurological complications, and even death.
Herpes simplex virus (HSV), a member of the Herpesviridae family, is a well-known cause of infections including genital herpes and herpes labialis in the adolescent and adult population. Transmission of HSV infection to an infant during the first 4-6 weeks of life can lead to devastating disease with the potential for poor outcomes. Early diagnosis is imperative when evaluating neonatal HSV infection in order to prevent further disease progression, neurological complications, and even death.
Herpes simplex Matendawa amapezeka kwambiri ndipo amatha kupatsirana kuchokera kwa amayi oyembekezera kupita kwa ana awo. Kachilomboka kangayambitse matenda aakulu kapena imfa mwa ana obadwa kumene. Ngakhale kuti sizichitika kawirikawiri panthawi yomwe ali ndi pakati, nthawi zambiri zimachitika panthawi yobereka. Chiwopsezo chimakhala chachikulu ngati mayi atenga kachilombo pakadutsa nthawi yoyembekezera. Komabe, chiopsezochi chikhoza kuchepetsedwa pogwiritsa ntchito mankhwala oletsa tizilombo toyambitsa matenda kapena kusankha gawo la C nthawi zina.
Infection with herpes simplex is one of the most common sexually transmitted infections. Because the infection is common in women of reproductive age it can be contracted and transmitted to the fetus during pregnancy and the newborn. Herpes simplex virus is an important cause of neonatal infection, which can lead to death or long-term disabilities. Rarely in the uterus, it occurs frequently during the transmission delivery. The greatest risk of transmission to the fetus and the newborn occurs in case of an initial maternal infection contracted in the second half of pregnancy. The risk of transmission of maternal-fetal-neonatal herpes simplex can be decreased by performing a treatment with antiviral drugs or resorting to a caesarean section in some specific cases.
Infection with herpes simplex is one of the most common sexually transmitted infections. Because the infection is common in women of reproductive age it can be contracted and transmitted to the fetus during pregnancy and the newborn. Herpes simplex virus is an important cause of neonatal infection, which can lead to death or long-term disabilities. Rarely in the uterus, it occurs frequently during the transmission delivery. The greatest risk of transmission to the fetus and the newborn occurs in case of an initial maternal infection contracted in the second half of pregnancy. The risk of transmission of maternal-fetal-neonatal herpes simplex can be decreased by performing a treatment with antiviral drugs or resorting to a caesarean section in some specific cases.
Herpes simplex virus (HSV) mitundu 1 ndi 2 imakhudza anthu ambiri padziko lonse lapansi. Nthawi zambiri, kachilomboka kamakhala chete m'maselo a mitsempha pambuyo pa kupatsira pakhungu, koma imatha kuyambiranso pambuyo pake, kumayambitsa zilonda zozizira. Nthawi zina, zimabweretsa mavuto akulu monga matenda a maso, kutupa muubongo, kapena mikhalidwe yowopsa kwa ana obadwa kumene komanso anthu omwe ali ndi chitetezo chofooka. Ngakhale mankhwala omwe alipo amathandizira kuwongolera matenda, chiwopsezo cha kukana mankhwala ndi zotsatira zake chimakhalabe chodetsa nkhawa. Tikufuna mankhwala atsopano kuti tithane ndi kachilomboka bwino.
Infection with herpes simplex virus (HSV) types 1 and 2 is ubiquitous in the human population. Most commonly, virus replication is limited to the epithelia and establishes latency in enervating sensory neurons, reactivating periodically to produce localized recurrent lesions. However, these viruses can also cause severe disease such as recurrent keratitis leading potentially to blindness, as well as encephalitis, and systemic disease in neonates and immunocompromised patients. Although antiviral therapy has allowed continual and substantial improvement in the management of both primary and recurrent infections, resistance to currently available drugs and long-term toxicity pose a current and future threat that should be addressed through the development of new antiviral compounds directed against new targets.
Infection with herpes simplex virus (HSV) types 1 and 2 is ubiquitous in the human population. Most commonly, virus replication is limited to the epithelia and establishes latency in enervating sensory neurons, reactivating periodically to produce localized recurrent lesions. However, these viruses can also cause severe disease such as recurrent keratitis leading potentially to blindness, as well as encephalitis, and systemic disease in neonates and immunocompromised patients. Although antiviral therapy has allowed continual and substantial improvement in the management of both primary and recurrent infections, resistance to currently available drugs and long-term toxicity pose a current and future threat that should be addressed through the development of new antiviral compounds directed against new targets.
Pali mitundu iwiri ya kachilombo ka herpes simplex , mtundu 1 (HSV-1) ndi mtundu wachiwiri (HSV-2). HSV-1 nthawi zambiri imayambitsa matenda pakamwa pomwe HSV-2 nthawi zambiri imayambitsa matenda kumaliseche. Amapatsirana mwachindunji ndi munthu yemwe ali ndi kachilomboka. Genital herpes amatchulidwa ngati matenda opatsirana pogonana. Akhoza kufalikira kwa khanda panthawi yobereka. Pambuyo pa matenda, mavairasi amasamutsidwa m'mitsempha ya minyewa kupita ku ma cell a mitsempha, komwe amakhala moyo wonse. Zomwe zimayambitsa kubwereza zingaphatikizepo: kuchepa kwa chitetezo cha mthupi, kupsinjika maganizo, ndi kuwala kwa dzuwa.
Nthawi zambiri, mankhwala oletsa tizilombo toyambitsa matenda amatengedwa pokhapokha ngati zizindikiro zakhala zovuta kwambiri. Mankhwala oletsa tizilombo toyambitsa matenda tsiku ndi tsiku akhoza kuperekedwa kwa munthu amene ali ndi matenda afupipafupi. Palibe katemera wopezeka ndipo katemera wa shingles samateteza herpes simplex. Kuchiza ndi mankhwala oletsa tizilombo toyambitsa matenda monga aciclovir kapena valaciclovir kumatha kuchepetsa kuopsa kwa zizindikiro.
Padziko lonse lapansi, HSV-1 kapena HSV-2 ili pakati pa 60% ndi 95% mwa akulu. HSV-1 nthawi zambiri amadwala ali mwana. Pafupifupi anthu 536 miliyoni padziko lonse lapansi (16% ya anthu) adadwala HSV-2 pofika chaka cha 2003 ndi chiwopsezo chachikulu pakati pa amayi ndi omwe akutukuka kumene. Anthu ambiri omwe ali ndi HSV-2 samazindikira kuti ali ndi kachilomboka.
○ Chithandizo ― OTC Mankhwala
Pewani kukhudzana kulikonse, monga kupsompsona mwana, pamene matuza alipo, chifukwa kukhudzana kungathe kufalitsa matenda kwa anthu ena. Muyenera kupuma osamwa mowa.
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