Poikilodermahttps://en.wikipedia.org/wiki/Poikiloderma
Poikiloderma ndi khungu lomwe limapangidwa ndi madera a hypopigmentation, hyperpigmentation, telangiectasias ndi atrophy. Poikiloderma imawoneka kwambiri pachifuwa kapena pakhosi, yomwe imadziwika ndi utoto wofiyira pakhungu womwe umayenderana ndi kuwonongeka kwa dzuwa.

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      References Diagnosis and Differential Diagnosis of Poikiloderma of Civatte: A Dermoscopy Cohort Study 36892344 
      NIH
      Poikiloderma of Civatte ndi khungu lodziwika bwino lomwe limawonekera kwambiri pakhosi ndi kumaso, makamaka kwa amayi akhungu loyera, omwe ali ndi vuto losiya kusamba. Imawonetsa ngati kusakaniza kwa mizere yofiira, mawanga akuda, ndi khungu lopyapyala. Nthawi zambiri, zimakhudza madera omwe ali ndi dzuwa, monga nkhope, khosi, ndi chifuwa, koma osati madera amthunzi. Poikiloderma of Civatte ikhoza kugawidwa m'magulu ake akuluakulu: kufiira, madontho akuda, kapena kusakaniza zonsezi. Choyambitsa chenicheni sichidziwika bwino, koma zinthu monga kutenthedwa ndi dzuwa, kusintha kwa mahomoni, machitidwe a mafuta onunkhira kapena zodzoladzola, ndi ukalamba zimaganiziridwa kuti zimakhudza. Poikiloderma of Civatte imayamba kuipira pang'onopang'ono pakapita nthawi.
      Poikiloderma of Civatte (PC) is a rather common benign dermatosis of the neck and face, mainly affecting fair-skinned individuals, especially postmenopausal females. It is characterized by a combination of a reticular pattern of linear telangiectasia, mottled hyperpigmentation and superficial atrophy. Clinically, it involves symmetrically sun-exposed areas of the face, the neck, and the V-shaped area of the chest, invariably sparing the anatomically shaded areas. Depending on the prevalent clinical feature, PC can be classified into erythemato-telangiectatic, pigmented, and mixed clinical types. The etiopathogenesis of PC is incompletely understood. Exposure to ultraviolet radiation, hormonal changes of menopause, contact sensitization to perfumes and cosmetics, and normal ageing have been incriminated. The diagnosis is usually clinical and can be confirmed by histology, which is characteristic, but not pathognomonic. The course is slowly progressive and irreversible, often causing significant cosmetic disfigurement.