Amyloidosis
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Faiga masani o le foliga o le Amyloidosis
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ReferencesNa sau se fafine e 26 tausaga le matua i le matou falemaʻi ma faito i le mageso o ona vae na maua i le 10 tausaga. E ui i le fa'aaogaina o kulimi steroid ma le kulimi tazarotene, e le'i toe lelei le mageso. E le'i i ai sana tala'aga talafeagai o lona aiga. Ina ua matou su'esu'eina o ia, na matou mauaina i luga o ona vae ua si'i i luga ma talatala, e fetaui ma se ma'i e ta'ua o le lichen amyloidosis.
A 26-year-old woman presented to our clinic with an itchy rash on her legs that had persisted for 10 years. The rash had previously been treated with topical steroids and tazarotene cream, with no improvement. The patient’s family history was noncontributory. A physical examination showed discrete and coalescing hyperkeratotic tan-brown papules on the pretibial surfaces, consistent with lichen amyloidosis.
A 26-year-old woman presented to our clinic with an itchy rash on her legs that had persisted for 10 years. The rash had previously been treated with topical steroids and tazarotene cream, with no improvement. The patient’s family history was noncontributory. A physical examination showed discrete and coalescing hyperkeratotic tan-brown papules on the pretibial surfaces, consistent with lichen amyloidosis.
Lichen Amyloidosis o se ma'i e masani ona maua i le pa'u e feso'ota'i i mageso faifai pea ona mafua'aga. E masani ona aliali ai ni fuifui o pa'i fa'atui'i, fa'ailoga lanu i le pito i fafo o le pa'u. Lichen Amyloidosis e masani ona aliali i tagata i tausaga 50 i le 60, ma o le mea e leaga ai, e leai se fofo. O togafitiga o lo'o avanoa nei e le masani ona lelei.
Lichen Amyloidosis (LA) is an uncommon, primary cutaneous amyloidosis associated with chronic, idiopathic pruritus. Clinical presentation of LA includes skin colored to hyperpigmented, papules coalescing into plaques with a rippled appearance on the extensors.1 LA most commonly presents in the fifth to sixth decade of life and has no curative treatments. Overall response to current therapies is poor.
Lichen Amyloidosis (LA) is an uncommon, primary cutaneous amyloidosis associated with chronic, idiopathic pruritus. Clinical presentation of LA includes skin colored to hyperpigmented, papules coalescing into plaques with a rippled appearance on the extensors.1 LA most commonly presents in the fifth to sixth decade of life and has no curative treatments. Overall response to current therapies is poor.
Lichen amyloidosis o se ma'i mageso umi-umi. E lauiloa i fuifui o patupatu mafiafia, e masani ona maua i tua, ivi, ogavae, ma lima. Pe a su'esu'eina i lalo o le microscope, Lichen amyloidosis o lo'o fa'aalia ai le fa'aputuina o le amyloid i le pito i luga o le pa'u, fa'atasi ai ma le mafiafia ma le fa'ateleina o le pa'u pito i fafo. E ui o le mafua'aga moni o le Lichen amyloidosis e le o malamalama atoatoa i le taimi nei, o suʻesuʻega talu ai ua fesoʻotaʻi i mea e pei o le olo, le fete'enaʻi i luga o le paʻu, le mali o sela (cell death), maʻi viral, ma isi. Lichen amyloidosis e foliga mai e feso'ota'i ma le tele o isi tulaga pa'u (atopic dermatitis, lichen planus, mycosis fungoides).
Lichen amyloidosis (LA) is a chronic pruritic skin disorder characterized by multiple grouped hyperkeratotic papules, predominantly located on the back, shins, thighs, and arms. Histological analysis of LA shows amyloid deposition in the papillary dermis and hyperkeratosis and acanthosis of the epidermis. The exact pathogenesis of LA has not yet been elucidated; however, prior reports have implicated frictional epidermal damage, apoptosis, viral infection, and many other triggers. LA is reportedly associated with several skin disorders, including atopic dermatitis (AD), lichen planus, and mycosis fungoides.
Lichen amyloidosis (LA) is a chronic pruritic skin disorder characterized by multiple grouped hyperkeratotic papules, predominantly located on the back, shins, thighs, and arms. Histological analysis of LA shows amyloid deposition in the papillary dermis and hyperkeratosis and acanthosis of the epidermis. The exact pathogenesis of LA has not yet been elucidated; however, prior reports have implicated frictional epidermal damage, apoptosis, viral infection, and many other triggers. LA is reportedly associated with several skin disorders, including atopic dermatitis (AD), lichen planus, and mycosis fungoides.
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#Electrophoresis of blood or urine
#Skin biopsy