Staphylococcal scalded skin syndrome
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Se pepe ma Staphylococcal scalded skin syndrome
References
Staphylococcal Scalded Skin Syndrome ose tulaga e masa'a ai le pa'u ona o mea oona e gaosia e nisi ituaiga siama Staphylococcus. E le masani ai i tamaiti e sili atu i le ono. E mafai foi ona tupu i tagata matutua e vaivai le puipuiga o le tino poʻo faʻafitauli ogaoga fatugaʻo. O le uiga autu o le pa'u tele o le pa'u pe a uma le mumū. O le ogaoga e amata mai i nai ma'i pa'u i le salalau o le pa'u o le pa'u, lea e mafai ona mafua ai le pa'u ogaoga o le vevela o le tino ma le le mautu o le toto.
Staphylococcal Scalded Skin Syndrome is a disease characterized by denudation of the skin caused by exotoxin producing strains of the Staphylococcus species, typically from a distant site. It usually presents 48 hours after birth and is rare in children older than six years. It may also present in immunocompromised adults or those with severe renal disease. The disorder is characterized by significant exfoliation of skin following cellulitis. The severity may vary from a few blisters to system exfoliation leading to marked hypothermia and hemodynamic instability.
Staphylococcal Scalded Skin Syndrome is a disease characterized by denudation of the skin caused by exotoxin producing strains of the Staphylococcus species, typically from a distant site. It usually presents 48 hours after birth and is rare in children older than six years. It may also present in immunocompromised adults or those with severe renal disease. The disorder is characterized by significant exfoliation of skin following cellulitis. The severity may vary from a few blisters to system exfoliation leading to marked hypothermia and hemodynamic instability.
Staphylococcal scalded skin syndrome (SSSS) and bullous impetigo are infections caused by Staphylococcus aureus. Bullous impetigo is due to the local release of these toxins and thus, often presents with localized skin findings, whereas SSSS is from the systemic spread of these toxins, resulting in a more generalized rash and severe presentation. Both conditions are treated with antibiotics that target S. aureus. These conditions can sometimes be confused with other conditions that result in superficial blistering.
Na sau se tamaititi e 10 masina le matua ma le susu o lona isu ma ua le lelei lana 'ai. Fai mai fomaʻi o se faʻamaʻi manava pito i luga. I le lua aso mulimuli ane, na toe foʻi mai ona e leʻi malosi ma ua iai ni faʻafitauli fou e pei o le fulafula o foliga ma le paʻu ita i le gutu. I le isi lua aso na sosoo ai, na atili ai ona leaga le tama. Ua fulafula ona lima ma vae ma ua amata ona alu ese lona paʻu. I tua i le falemaʻi, na iloa ai e fomaʻi le mumu mumu i ona foliga ma i totonu o le gaugau o lona paʻu, e paʻu pe a tago i ai. Na latou maua o ia i le staphylococcal scalded skin syndrome (SSSS) ma amata ai o ia i vailaau faʻamaʻi e ala i se vein.
A 10-month-old male presented with rhinorrhea and decreased oral intake and was diagnosed with an upper respiratory infection. Two days later, he returned to the clinic due to a lack of improvement and the onset of new symptoms, including facial edema and perioral skin irritation. That evening, he became febrile at 100.4 °F and went to the emergency department at the local children's hospital. No further workup was done and the parents were instructed to continue with the current treatment regimen. Over the next 48 hours, the patient's symptoms worsened with the new onset of bilateral extremity edema and desquamation. The patient was returned to the emergency department. A physical exam was notable for a blanching, desquamating, erythematous rash on the face and creases of the arms, legs, and groin. A positive Nikolsky sign was reported. A clinical diagnosis of staphylococcal scalded skin syndrome (SSSS) was made, and the patient was started on intravenous clindamycin. This case illustrates a severe presentation of SSSS in a pediatric patient, demonstrating the challenges it poses to diagnosis and treatment.
A 10-month-old male presented with rhinorrhea and decreased oral intake and was diagnosed with an upper respiratory infection. Two days later, he returned to the clinic due to a lack of improvement and the onset of new symptoms, including facial edema and perioral skin irritation. That evening, he became febrile at 100.4 °F and went to the emergency department at the local children's hospital. No further workup was done and the parents were instructed to continue with the current treatment regimen. Over the next 48 hours, the patient's symptoms worsened with the new onset of bilateral extremity edema and desquamation. The patient was returned to the emergency department. A physical exam was notable for a blanching, desquamating, erythematous rash on the face and creases of the arms, legs, and groin. A positive Nikolsky sign was reported. A clinical diagnosis of staphylococcal scalded skin syndrome (SSSS) was made, and the patient was started on intravenous clindamycin. This case illustrates a severe presentation of SSSS in a pediatric patient, demonstrating the challenges it poses to diagnosis and treatment.
O se teineititi e 2-tausaga le matua na aliali mai ma se mageso i lona tino atoa na tupu i luga o le 48 itula, ina ua uma ona utia e iniseti ona foliga i le aso na muamua atu. I le taimi o su'esu'ega, sa tele lona ma'itama'i na faia i tama'i patupatu e fefiloi fa'atasi, ma a mimilo mama, o lona pa'u na fa'aalia ai ni fa'ailoga o le Nikolsky. E leai ni fa'ailoga o le mageso na a'afia ai ona mucous membrane. O su'ega i lona toto e le'i fa'aalia ai ni fa'ailoga o fa'ama'i.
A 2-year-old girl presented a generalised rash with 48 h of evolution, in the context of insect bites on the face on the day before. At observation, she had a generalised micropapular rash with confluent areas and Nikolsky sign. There was no mucosal area affected. Blood cultures were negative.
A 2-year-old girl presented a generalised rash with 48 h of evolution, in the context of insect bites on the face on the day before. At observation, she had a generalised micropapular rash with confluent areas and Nikolsky sign. There was no mucosal area affected. Blood cultures were negative.
O le ma'i e fa'aosoina e le epidermolytic exotoxins (exfoliatin) A ma le B, lea e fa'asa'olotoina e S. aureus. O le fa'ama'i o le staphylococcal scalded skin syndrome i tamaiti e lelei tele, fa'atasi ai ma le fa'ai'uga atoatoa i totonu o le 10 aso o togafitiga, ma e leai se ma'ila tele. Ae ui i lea, staphylococcal scalded skin syndrome e tatau ona va'ava'ai ma le fa'aeteete mai le necrolysis epidermal o'ona, lea e maua ai se fa'ailoga leaga.
○ Suiga ma Togafiti
O le fa'ailoa sese o manu'a eczema e pei ole atopic dermatitis, ma le fa'aogaina ole su'u steroid e fa'ateteleina ai fa'ama'i. Fa'amolemole saili vave togafitiga faafoma'i a'o fa'aogaina le suau'u antibiotic.
#Bacitracin
#First-generation cephalosporins (e.g. Cefradine)
#Bacterial culture
#Third-generation cephalosporins (e.g. Cefditoren Pivoxil)