Erythema multiforme - Erythema Multiform
☆ AI Dermatology — Free Service
Mune 2022 Stiftung Warentest mhedzisiro kubva kuGermany, kugutsikana kwevatengi neModelDerm kwakangodzikira zvishoma pane nekubhadharwa kwe telemedicine kubvunzana.
Mune 2022 Stiftung Warentest mhedzisiro kubva kuGermany, kugutsikana kwevatengi neModelDerm kwakangodzikira zvishoma pane nekubhadharwa kwe telemedicine kubvunzana.
Erythema multiforme minor ― Ziva kuti nzvimbo dzemaronda dzinogona kuputika.
Urticaria inogonawo kutorwa seyakasiyana pakuongororwa.
relevance score : -100.0%
ReferencesErythema multiforme (EM) imamiriro ezvinhu apo mapundu akasiyana‑siyana anowoneka paganda uye pamukobvu (mucous membranes) nekuda kwekupindira kwemaitiro ekudzivirira. Kunyange zvazvo kazhinji ichikonzerwa nehutachiona, kunyanya herpes simplex virus (HSV), kana nemimwe mishonga, chikonzero chinoramba chisingazivikanwi muzviitiko zvakawanda. Kurapa acute EM kunotarisa kuderedza zviratidzo nekushandisa makirimu ane steroid kana antihistamines. Kurapa EM inodzokororwa kunonyanya kushanda kana kwakagadzirirwa murwere wega. Nzira dzekutanga dzinosanganisira kurapwa kwemumuromo uye kwepamusoro, izvo zvinosanganisira corticosteroids uye antiviral mishonga. Kurapwa kwepamusoro kunosanganisira steroid creams dzine simba uye kugadzirisa mucous membranes dzakakuvadzwa. Kune varwere vasingade mishonga inorwisa mavhairasi, sarudzo dzemutsara wechipiri dzinosanganisira immunosuppressants, antibiotics, anthelmintics, uye antimalarials.
Erythema multiforme (EM) is an immune-mediated condition that classically presents with discrete targetoid lesions and can involve both mucosal and cutaneous sites. While EM is typically preceded by viral infections, most notably herpes simplex virus (HSV), and certain medications, a large portion of cases are due to an unidentifiable cause. Treatment for acute EM is focused on relieving symptoms with topical steroids or antihistamines. Treatment for recurrent EM is most successful when tailored to individual patients. First line treatment for recurrent EM includes both systemic and topical therapies. Systemic therapies include corticosteroid therapy and antiviral prophylaxis. Topical therapies include high-potency corticosteroids, and antiseptic or anesthetic solutions for mucosal involvement. Second-line therapies for patients who do not respond to antiviral medications include immunosuppressive agents, antibiotics, anthelmintics, and antimalarials
Erythema multiforme (EM) is an immune-mediated condition that classically presents with discrete targetoid lesions and can involve both mucosal and cutaneous sites. While EM is typically preceded by viral infections, most notably herpes simplex virus (HSV), and certain medications, a large portion of cases are due to an unidentifiable cause. Treatment for acute EM is focused on relieving symptoms with topical steroids or antihistamines. Treatment for recurrent EM is most successful when tailored to individual patients. First line treatment for recurrent EM includes both systemic and topical therapies. Systemic therapies include corticosteroid therapy and antiviral prophylaxis. Topical therapies include high-potency corticosteroids, and antiseptic or anesthetic solutions for mucosal involvement. Second-line therapies for patients who do not respond to antiviral medications include immunosuppressive agents, antibiotics, anthelmintics, and antimalarials
Muzviitiko zvakawanda, erythema multiforme inopera yega mukati memavhiki maviri kusvika mana. Stevens‑Johnson syndrome, chirwere chakakomba chinokanganisa mucous membranes, chinogona kuramba chiripo kusvika kumavhiki matanhatu. Steroids haiwanzo kurudzirwa pakurapa zvinyoro. Kana steroids ichifanira kushandiswa pakurapa erythema multiforme yakanyanya, hazvina kusimbiswa sezvo pasina humbowo hwakajeka kubva kumiedzo yakarongeka inoratidza kuti ndiani anobatsirwa nekurapwa uku.
In most cases, mild erythema multiforme is self-limited and resolves in 2 to 4 weeks. Stevens-Johnson syndrome is a serious disease that involves the mucous membranes and lasts up to 6 weeks. There is no indication for using steroids for the mild form. Use of steroids for erythema multiforme major is debatable because no randomized studies clearly indicate which children will benefit from this treatment.
In most cases, mild erythema multiforme is self-limited and resolves in 2 to 4 weeks. Stevens-Johnson syndrome is a serious disease that involves the mucous membranes and lasts up to 6 weeks. There is no indication for using steroids for the mild form. Use of steroids for erythema multiforme major is debatable because no randomized studies clearly indicate which children will benefit from this treatment.
Isu tinopa nyaya ye erythema multiforme (EM) yemumuromo yakakonzerwa ne TMP/SMX, inoratidza maronda emumuromo nemuviri pasina maronda eganda. Izvi zvinosimbisa kukosha kwekuisiyanisa kubva kune mamwe ma‑oral ulcerative kusagadzikana. Murwere akawana kurapwa kwechiratidzo pamwe nemapiritsi e prednisolone, izvo zvakabatsira kuti pave nekuvandudzwa mushure mekumisa TMP/SMX therapy.
We report a case of oral erythema multiforme (EM) secondary to TMP/SMX that presented with oral and lip ulcerations typical of EM without any skin lesions and highlights the importance of distinguishing them from other ulcerative disorders involving oral cavity. The patient was treated symptomatically and given tablet prednisolone. The condition improved with stoppage of TMP/SMX therapy.
We report a case of oral erythema multiforme (EM) secondary to TMP/SMX that presented with oral and lip ulcerations typical of EM without any skin lesions and highlights the importance of distinguishing them from other ulcerative disorders involving oral cavity. The patient was treated symptomatically and given tablet prednisolone. The condition improved with stoppage of TMP/SMX therapy.
Erythema multiforme inobata ganda uye dzimwe nguva mucosa, ichikonzerwa ne immune system. Kazhinji, inoratidzika semaronda anovavarira, anogona kuva akasiyana‑siyana, kudzoka, kana kuramba. Maronda aya anowanzo kukanganisa magumo eganda zvakaenzana, kunyanya panzvimbo dzemukati. Zvikonzero zvikuru zvinosanganisira hutachiona hweherpes simplex virus ne Mycoplasma pneumoniae, pamwe nemimwe mishonga, majekiseni, uye autoimmune zvirwere. Kusiyanisa erythema multiforme kubva ku urticaria kunoenderana nenguva yemaronda; maronda e erythema multiforme anoramba achienderera kwemazuva manomwe, nepo maronda e urticaria achiwanzopera mukati mezuva rimwe. Kunyangwe zvichifanana, zvakakosha kusiyanisa erythema multiforme kubva kuStevens‑Johnson syndrome, iyo inowanzo ratidza macules erythematous kana purpuric ane mablister. Kurapa erythema multiforme kunosanganisira kuzorodza zviratidzo ne topical steroids kana antihistamines uye kugadzirisa chikonzero. Muzviitiko zvakabatana neherpes simplex virus, antiviral prophylaxis inorudzirwa. Kana mucosal ichibatanidzwa zvakanyanya, zvinodikanwa kuenda kuchipatara kuti ape intravenous fluids uye kutsiva ma‑electrolyte.
Erythema multiforme is a reaction involving the skin and sometimes the mucosa, triggered by the immune system. Typically, it manifests as target-like lesions, which may appear isolated, recur, or persist. These lesions usually symmetrically affect the extremities, particularly their outer surfaces. The main causes include infections like herpes simplex virus and Mycoplasma pneumoniae, as well as certain medications, immunizations, and autoimmune diseases. Distinguishing erythema multiforme from urticaria relies on the duration of lesions; erythema multiforme lesions remain fixed for at least seven days, while urticarial lesions often vanish within a day. Although similar, it's crucial to differentiate erythema multiforme from the more severe Stevens-Johnson syndrome, which typically presents widespread erythematous or purpuric macules with blisters. Managing erythema multiforme involves symptomatic relief with topical steroids or antihistamines and addressing the underlying cause. For recurrent cases associated with herpes simplex virus, prophylactic antiviral therapy is recommended. Severe mucosal involvement may necessitate hospitalization for intravenous fluids and electrolyte replacement.
Erythema multiforme is a reaction involving the skin and sometimes the mucosa, triggered by the immune system. Typically, it manifests as target-like lesions, which may appear isolated, recur, or persist. These lesions usually symmetrically affect the extremities, particularly their outer surfaces. The main causes include infections like herpes simplex virus and Mycoplasma pneumoniae, as well as certain medications, immunizations, and autoimmune diseases. Distinguishing erythema multiforme from urticaria relies on the duration of lesions; erythema multiforme lesions remain fixed for at least seven days, while urticarial lesions often vanish within a day. Although similar, it's crucial to differentiate erythema multiforme from the more severe Stevens-Johnson syndrome, which typically presents widespread erythematous or purpuric macules with blisters. Managing erythema multiforme involves symptomatic relief with topical steroids or antihistamines and addressing the underlying cause. For recurrent cases associated with herpes simplex virus, prophylactic antiviral therapy is recommended. Severe mucosal involvement may necessitate hospitalization for intravenous fluids and electrolyte replacement.
Mamiriro acho anogona kusiyana kubva pakupfava, kuzvimisikidza kusvika pakunetsa, uye inotyisidzira hupenyu kana ikava erythema multiforme huru iyo inosanganisirawo mucous membranes. Kupinda kwemukasi membrane kana kuvapo kwemabhuru ndizvo zviratidzo zvakakosha zvekuomarara.
- Erythema multiforme minor: zvinowanzoitika kana kusimudzwa, edematous papules akagoverwa acrally. Chimiro chakapfava chinowanzo ratidza nekukwenya zvinyoro (asi kukwenya kunogona kuve kwakanyanya), mabhururu epingi‑tsvuku, akarongeka, uye anowanikwa pakupera. Kupora kunowanzotora mazuva 7‑10.
- Erythema multiforme major: zvinowanzoitika kana kusimudzwa, edematous papules akagoverwa acrally pamwe nekubatana kwemamwe kana akawanda mucous membranes. Epidermal detachment inosanganisira isingasviki 10 % yenzvimbo yese yemuviri.
○ Kurapa ― OTC Zvinodhaka
Kana chirwere chichiperekedzwa nefivha (kupisa muviri), zvinokurudzirwa kushanyira chipatara nokukurumidza.
Mishonga inofungidzirwa kuti inofanira kuregerwa (semuenzaniso maantibiotic, non‑steroidal anti‑inflammatory drugs).
Oral antihistamines yakadai se‑cetirizine uye loratadine inogona kubatsira pakukwenya.
#Cetirizine [Zytec]
#LevoCetirizine [Xyzal]
#Loratadine [Claritin]