Herpes simplex
☆ Mune 2022 Stiftung Warentest mhedzisiro kubva kuGermany, kugutsikana kwevatengi neModelDerm kwakangodzikira zvishoma pane nekubhadharwa kwe telemedicine kubvunzana.
Herpes simplex pamuromo.
Herpes simplex ― Utachiona hweHerpes simplex muminwe hunowanikwa muvana vadiki pane vakuru.
References
HSV-1 hutachiwana hunofambira mberi kuburikidza nehutachiona hwekutanga hwemasero epithelial, hunoteverwa ne latency, kunyanya mueuroni, uye reactivation. HSV-1 inowanzokonzera kuputika kwe vesicular kwekutanga uye kunodzokororwa, kunyanya pamuromo uye mucosa yesikarudzi. Kuratidzwa kwayo kunobva kuorolabial herpes kune akasiyana mamiriro akadai seherpetic folliculitis, ganda utachiona, kubatanidzwa kwemaziso, uye zviitiko zvakakomba senge herpes encephalitis. Antiviral therapy inobatsira kubata HSV utachiona.
Herpes simplex virus type 1 (HSV-1) is a member of the Alphaherpesviridae subfamily. Its structure is composed of linear dsDNA, an icosahedral capsid that is 100 to 110 nm in diameter, with a spikey envelope. In general, the pathogenesis of HSV-1 infection follows a cycle of primary infection of epithelial cells, latency primarily in neurons, and reactivation. HSV-1 is responsible for establishing primary and recurrent vesicular eruptions, primarily in the orolabial and genital mucosa. HSV-1 infection has a wide variety of presentations, including orolabial herpes, herpetic sycosis (HSV folliculitis), herpes gladiatorum, herpetic whitlow, ocular HSV infection, herpes encephalitis, Kaposi varicelliform eruption (eczema herpeticum), and severe or chronic HSV infection. Antiviral therapy limits the course of HSV infection.
Herpes simplex virus type 1 (HSV-1) is a member of the Alphaherpesviridae subfamily. Its structure is composed of linear dsDNA, an icosahedral capsid that is 100 to 110 nm in diameter, with a spikey envelope. In general, the pathogenesis of HSV-1 infection follows a cycle of primary infection of epithelial cells, latency primarily in neurons, and reactivation. HSV-1 is responsible for establishing primary and recurrent vesicular eruptions, primarily in the orolabial and genital mucosa. HSV-1 infection has a wide variety of presentations, including orolabial herpes, herpetic sycosis (HSV folliculitis), herpes gladiatorum, herpetic whitlow, ocular HSV infection, herpes encephalitis, Kaposi varicelliform eruption (eczema herpeticum), and severe or chronic HSV infection. Antiviral therapy limits the course of HSV infection.
Herpes simplex virus type 2 (HSV-2) chirwere chakapararira, chinokanganisa 22% yevanhu vakuru vane makore gumi nemaviri uye pamusoro, vanosvika mamiriyoni makumi mana nemashanu evanhu vakuru muUnited States. Nepo HSV-1 ichiwanzo konzera maronda emukanwa, inogona zvakare kutungamirira kumaronda enhengo dzesikarudzi. Zvisinei, apo varwere vane maronda epanhengo yesikarudzi, HSV-2 kazhinji ndiyo inonyanya kunetseka. Zviratidzo zveHSV-2 kubuda kazhinji hazvina kujeka, senge kukwenya kwenhengo dzesikarudzi uye kutsamwa, izvo zvinogona kunonoka kuongororwa uye kurapwa. Kunonoka uku kunogona kukonzera kutapuriranwa kune vanhu vasina utachiona.
Herpes simplex virus type 2 (HSV-2) continues to be a common infection, affecting approximately 22% of adults ages 12 and older, representing 45 million adults in the United States alone. While HSV-1 often affects the perioral region and can be known to cause genital lesions, HSV-2 is more commonly the consideration when patients present with genital lesions. Despite this, most outbreaks of the infection will present with nonspecific symptoms such as genital itching, irritation, and excoriations, which may cause diagnosis and treatment to be delayed. As a result, further exposure to uninfected individuals may occur.
Herpes simplex virus type 2 (HSV-2) continues to be a common infection, affecting approximately 22% of adults ages 12 and older, representing 45 million adults in the United States alone. While HSV-1 often affects the perioral region and can be known to cause genital lesions, HSV-2 is more commonly the consideration when patients present with genital lesions. Despite this, most outbreaks of the infection will present with nonspecific symptoms such as genital itching, irritation, and excoriations, which may cause diagnosis and treatment to be delayed. As a result, further exposure to uninfected individuals may occur.
Herpes simplex virus (HSV) kazhinji inokonzeresa hutachiona senge genital herpes uye maronda echando muvechidiki nevakuru. Kana HSV inobata mucheche mukati memavhiki ekutanga e4-6 ehupenyu, zvinogona kukonzera kurwara kwakanyanya nemigumisiro yakakomba. Kukurumidza kuona hutachiona hweHSV hwevacheche hwakakosha kumisa chirwere ichi kuti chisawedzera, kudzivirira nyaya dzetsinga (kunyangwe rufu) .
Herpes simplex virus (HSV), a member of the Herpesviridae family, is a well-known cause of infections including genital herpes and herpes labialis in the adolescent and adult population. Transmission of HSV infection to an infant during the first 4-6 weeks of life can lead to devastating disease with the potential for poor outcomes. Early diagnosis is imperative when evaluating neonatal HSV infection in order to prevent further disease progression, neurological complications, and even death.
Herpes simplex virus (HSV), a member of the Herpesviridae family, is a well-known cause of infections including genital herpes and herpes labialis in the adolescent and adult population. Transmission of HSV infection to an infant during the first 4-6 weeks of life can lead to devastating disease with the potential for poor outcomes. Early diagnosis is imperative when evaluating neonatal HSV infection in order to prevent further disease progression, neurological complications, and even death.
Herpes simplex Utachiona huzhinji uye hunogona kutapurirwa kubva kumadzimai akazvitakura kuenda kuvacheche vavo. Hutachiona uhu hunogona kukonzera matambudziko akakomba ehutano kana kufa kwevacheche. Kunyange zvazvo zvisingawanzoitiki panguva yekuzvitakura pachayo, zvinowanzoitika panguva yekusununguka. Ngozi yakakura zvikuru kana amai vakatapukirwa munguva dzepashure penhumbu. Nekudaro, njodzi iyi inogona kuderedzwa nekushandisa antiviral zvinodhaka kana kusarudza C-chikamu mune mamwe mamiriro.
Infection with herpes simplex is one of the most common sexually transmitted infections. Because the infection is common in women of reproductive age it can be contracted and transmitted to the fetus during pregnancy and the newborn. Herpes simplex virus is an important cause of neonatal infection, which can lead to death or long-term disabilities. Rarely in the uterus, it occurs frequently during the transmission delivery. The greatest risk of transmission to the fetus and the newborn occurs in case of an initial maternal infection contracted in the second half of pregnancy. The risk of transmission of maternal-fetal-neonatal herpes simplex can be decreased by performing a treatment with antiviral drugs or resorting to a caesarean section in some specific cases.
Infection with herpes simplex is one of the most common sexually transmitted infections. Because the infection is common in women of reproductive age it can be contracted and transmitted to the fetus during pregnancy and the newborn. Herpes simplex virus is an important cause of neonatal infection, which can lead to death or long-term disabilities. Rarely in the uterus, it occurs frequently during the transmission delivery. The greatest risk of transmission to the fetus and the newborn occurs in case of an initial maternal infection contracted in the second half of pregnancy. The risk of transmission of maternal-fetal-neonatal herpes simplex can be decreased by performing a treatment with antiviral drugs or resorting to a caesarean section in some specific cases.
Herpes simplex virus (HSV) mhando 1 uye 2 inotapurira vanhu vazhinji pasi rese. Kazhinji, hutachiona hunoramba hwakanyarara mumaseru etsinga mushure mekutapurira ganda, asi hunogona kuitazve pave paye, huchikonzera maronda anotonhora. Dzimwe nguva, zvinotungamira kumatambudziko akakomba senge kutapukira kwemaziso, kuzvimba kwehuropi, kana mamiriro ane njodzi muvachangozvarwa uye vanhu vane immune immune system. Kunyange zvazvo mishonga yemazuva ano ichibatsira kudzora utachiona, njodzi yekuramba zvinodhaka nemishonga inoramba ichinetsa. Tinoda mishonga mitsva yekunangisa utachiona zviri nani.
Infection with herpes simplex virus (HSV) types 1 and 2 is ubiquitous in the human population. Most commonly, virus replication is limited to the epithelia and establishes latency in enervating sensory neurons, reactivating periodically to produce localized recurrent lesions. However, these viruses can also cause severe disease such as recurrent keratitis leading potentially to blindness, as well as encephalitis, and systemic disease in neonates and immunocompromised patients. Although antiviral therapy has allowed continual and substantial improvement in the management of both primary and recurrent infections, resistance to currently available drugs and long-term toxicity pose a current and future threat that should be addressed through the development of new antiviral compounds directed against new targets.
Infection with herpes simplex virus (HSV) types 1 and 2 is ubiquitous in the human population. Most commonly, virus replication is limited to the epithelia and establishes latency in enervating sensory neurons, reactivating periodically to produce localized recurrent lesions. However, these viruses can also cause severe disease such as recurrent keratitis leading potentially to blindness, as well as encephalitis, and systemic disease in neonates and immunocompromised patients. Although antiviral therapy has allowed continual and substantial improvement in the management of both primary and recurrent infections, resistance to currently available drugs and long-term toxicity pose a current and future threat that should be addressed through the development of new antiviral compounds directed against new targets.
Kune mhando mbiri dze herpes simplex virus, type 1 (HSV-1) ne type 2 (HSV-2). HSV-1 kazhinji inokonzeresa hutachiona pamuromo nepo HSV-2 ichinyanya kukonzera kutapukira kunhengo dzakavandika. Zvinotapurirwa nekusangana kwakananga nemunhu ane utachiona. Genital herpes inorondedzerwa sechirwere chepabonde. Inogona kupararira kumucheche panguva yekusununguka. Mushure mekutapukirwa, mavhairasi aya anotakurwa achitevedza tsinga dzekunzwa kuenda kumasero etsinga, kwaanogara hupenyu hwese. Zvikonzero zvekudzokazve zvinogona kusanganisira: kuderera kwesimba rekudzivirira, kushushikana, uye kubuda kwezuva.
Kazhinji, mishonga inorwisa mavhairasi inotorwa chete kana zviratidzo zvanyanya. Mushonga wemazuva ese weantiviral unogona kunyorerwa kune munhu ane hutachiona hwenguva zhinji. Iko hakuna yekudzivirira inowanikwa uye iyo yekudzivirira shingles haidziviriri herpes simplex. Kurapa nemishonga inorwisa utachiona senge aciclovir kana valaciclovir kunogona kuderedza kuoma kwezviratidzo.
Pasi rose mitengo yeHSV-1 kana HSV-2 iri pakati pe60% ne95% muvakuru. HSV-1 inowanzobatwa nehutachiona munguva yehuduku. Vanhu vanosvika 536 miriyoni pasi rose (16% yevagari vemo) vakabatwa neHSV-2 kubva muna 2003 nehuwandu hwehuwandu pakati pevakadzi nevari munyika dzichiri kusimukira. Vanhu vazhinji vane HSV-2 havazivi kuti vane utachiona.
○ Kurapa ― OTC Zvinodhaka
Dzivisa kubata chero kupi zvako kwemuviri, sekutsvoda mwana, nepo mablister aripo, sezvo kubata kunogona kuparadzira hutachiona kune vamwe vanhu. Unofanira kuzorora usinganwi doro.
#Acyclovir cream