Poikilodermahttps://en.wikipedia.org/wiki/Poikiloderma
Poikiloderma chirwere cheganda chine nzvimbo dze hypopigmentation, hyperpigmentation, telangiectasias uye atrophy. Poikiloderma inowanzoonekwa pachipfuva kana muhuro, inoratidzwa neruvara rutsvuku paganda rinowanzobatanidzwa nekukuvadzwa nezuva.

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      References Diagnosis and Differential Diagnosis of Poikiloderma of Civatte: A Dermoscopy Cohort Study 36892344 
      NIH
      Poikiloderma of Civatte ichirwere cheganda chinowanzoitika pachifuva nepakumbo, kunyanya muvakadzi vane ganda rakatsetseka, vakapfuura menopause. Inoratidza semusanganiswa wetelangiectasia – mitsara yakatsvuka, hypopigmentation – kusvibira ganda, hyperpigmentation – mavara akasviba, uye atrophy – ganda rakatetepa. Kazhinji, inobata nzvimbo dzakaratidzwa kuzuva, sechiso, mutsipa, uye chipfuva, asi kwete nzvimbo dzine mumvuri. Poikiloderma of Civatte inogona kukamurwa zvichienderana neayo makuru maficha: erythemato‑telangiectatic, pigmented, kana mixed. Chikonzero chaicho hachinyatsonzwisiswi, asi zvinhu zvakaita sekubuda kwe ultraviolet radiation, kuchinja kwe hormonal changes of menopause, contact sensitization to perfumes and cosmetics, uye normal ageing zvinofungidzirwa kuti zvine basa. Poikiloderma of Civatte inowanzoita slowly progressive uye irreversible nekufamba kwenguva.
      Poikiloderma of Civatte (PC) is a rather common benign dermatosis of the neck and face, mainly affecting fair-skinned individuals, especially postmenopausal females. It is characterized by a combination of a reticular pattern of linear telangiectasia, mottled hyperpigmentation and superficial atrophy. Clinically, it involves symmetrically sun-exposed areas of the face, the neck, and the V-shaped area of the chest, invariably sparing the anatomically shaded areas. Depending on the prevalent clinical feature, PC can be classified into erythemato-telangiectatic, pigmented, and mixed clinical types. The etiopathogenesis of PC is incompletely understood. Exposure to ultraviolet radiation, hormonal changes of menopause, contact sensitization to perfumes and cosmetics, and normal ageing have been incriminated. The diagnosis is usually clinical and can be confirmed by histology, which is characteristic, but not pathognomonic. The course is slowly progressive and irreversible, often causing significant cosmetic disfigurement.