Staphylococcal scalded skin syndrome
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Mucheche ane Staphylococcal scalded skin syndrome
References
Staphylococcal Scalded Skin Syndrome imamiriro ekuti ganda rinodonha nekuda kwechepfu inogadzirwa nemamwe marudzi eStaphylococcus bacteria. Izvo hazviwanzo muvana vanopfuura makore matanhatu. Inogonawo kuitika kune vanhu vakuru vane simba rekudzivirira zvirwere kana matambudziko makuru eitsvo. Chinhu chikuru ndechekufumuka kweganda zvichitevera kuzvimba. Kuomarara kunobva kune mashoma mablister kusvika pakupararira kweganda kurasikirwa, izvo zvinogona kukonzera kudonha kwakanyanya kwekushisa kwemuviri uye kusagadzikana muropa.
Staphylococcal Scalded Skin Syndrome is a disease characterized by denudation of the skin caused by exotoxin producing strains of the Staphylococcus species, typically from a distant site. It usually presents 48 hours after birth and is rare in children older than six years. It may also present in immunocompromised adults or those with severe renal disease. The disorder is characterized by significant exfoliation of skin following cellulitis. The severity may vary from a few blisters to system exfoliation leading to marked hypothermia and hemodynamic instability.
Staphylococcal Scalded Skin Syndrome is a disease characterized by denudation of the skin caused by exotoxin producing strains of the Staphylococcus species, typically from a distant site. It usually presents 48 hours after birth and is rare in children older than six years. It may also present in immunocompromised adults or those with severe renal disease. The disorder is characterized by significant exfoliation of skin following cellulitis. The severity may vary from a few blisters to system exfoliation leading to marked hypothermia and hemodynamic instability.
Staphylococcal scalded skin syndrome (SSSS) and bullous impetigo are infections caused by Staphylococcus aureus. Bullous impetigo is due to the local release of these toxins and thus, often presents with localized skin findings, whereas SSSS is from the systemic spread of these toxins, resulting in a more generalized rash and severe presentation. Both conditions are treated with antibiotics that target S. aureus. These conditions can sometimes be confused with other conditions that result in superficial blistering.
Kakomana kane 10months kakauya kakarova mhino kasingadyi zvakanaka. Vanachiremba vakati chaive chirwere chepamusoro chekufema. Kwapera mazuva maviri, akadzoka nekuti akanga asiri kuita zviri nani uye akanga ava nezvinetso zvitsva zvakaita sechiso chakazvimba uye ganda rakatsvinda pamuromo. Mumazuva maviri akatevera, mukomana wacho akawedzera kuipa. Maoko nemakumbo akazvimba uye ganda rake rakatanga kubuda. Tadzokera kuchipatara, vanachiremba vakaona mapundu akatsvuka kumeso kwake uye pakapeta ganda rake, iro raipenya kana akabatwa. Vakamuvheneka ne staphylococcal scalded skin syndrome (SSSS) uye vakamutanga kushandisa mishonga inorwisa mabhakitiriya kuburikidza netsinga.
A 10-month-old male presented with rhinorrhea and decreased oral intake and was diagnosed with an upper respiratory infection. Two days later, he returned to the clinic due to a lack of improvement and the onset of new symptoms, including facial edema and perioral skin irritation. That evening, he became febrile at 100.4 °F and went to the emergency department at the local children's hospital. No further workup was done and the parents were instructed to continue with the current treatment regimen. Over the next 48 hours, the patient's symptoms worsened with the new onset of bilateral extremity edema and desquamation. The patient was returned to the emergency department. A physical exam was notable for a blanching, desquamating, erythematous rash on the face and creases of the arms, legs, and groin. A positive Nikolsky sign was reported. A clinical diagnosis of staphylococcal scalded skin syndrome (SSSS) was made, and the patient was started on intravenous clindamycin. This case illustrates a severe presentation of SSSS in a pediatric patient, demonstrating the challenges it poses to diagnosis and treatment.
A 10-month-old male presented with rhinorrhea and decreased oral intake and was diagnosed with an upper respiratory infection. Two days later, he returned to the clinic due to a lack of improvement and the onset of new symptoms, including facial edema and perioral skin irritation. That evening, he became febrile at 100.4 °F and went to the emergency department at the local children's hospital. No further workup was done and the parents were instructed to continue with the current treatment regimen. Over the next 48 hours, the patient's symptoms worsened with the new onset of bilateral extremity edema and desquamation. The patient was returned to the emergency department. A physical exam was notable for a blanching, desquamating, erythematous rash on the face and creases of the arms, legs, and groin. A positive Nikolsky sign was reported. A clinical diagnosis of staphylococcal scalded skin syndrome (SSSS) was made, and the patient was started on intravenous clindamycin. This case illustrates a severe presentation of SSSS in a pediatric patient, demonstrating the challenges it poses to diagnosis and treatment.
Mumwe musikana ane makore 2 okuberekwa akazviratidza aine mapundu muviri wake wose akanga aita maawa anopfuura makumi mana nemasere, zvichitevera kurumwa netumbuyu kumeso kwake nezuro wacho. Panguva yekuongororwa, aive nekuputika kwakapararira kwakagadzirwa nemapundu madiki akabatana pamwe chete, uye paakakwizwa zvishoma, ganda rake rairatidza zviratidzo zvechiratidzo cheNikolsky. Pakanga pasina zviratidzo zvepundu raibata mutezo wake. Kuongororwa kweropa rake hakuna kuratidza zviratidzo zvehutachiona.
A 2-year-old girl presented a generalised rash with 48 h of evolution, in the context of insect bites on the face on the day before. At observation, she had a generalised micropapular rash with confluent areas and Nikolsky sign. There was no mucosal area affected. Blood cultures were negative.
A 2-year-old girl presented a generalised rash with 48 h of evolution, in the context of insect bites on the face on the day before. At observation, she had a generalised micropapular rash with confluent areas and Nikolsky sign. There was no mucosal area affected. Blood cultures were negative.
Chirwere ichi chinokonzerwa ne epidermolytic exotoxins (exfoliatin)A uye B, iyo inobudiswa neS. aureus. Chirevo che staphylococcal scalded skin syndrome muvana chakanakisisa, nekugadzirisa kwakakwana mukati memazuva gumi ekurapwa, uye pasina mavanga makuru. Zvisinei, staphylococcal scalded skin syndrome inofanira kusiyaniswa zvakanyatsonaka kubva kune chepfu epidermal necrolysis, iyo inotakura kufungidzira kusina kunaka.
○ Kuongororwa uye Kurapwa
Misdiagnosis se eczema maronda akadai seatopic dermatitis, uye kushandiswa kwe steroid mafuta anowedzera maronda. Ndokumbirawo utsvage kurapwa nekukurumidza paunenge uchizora mishonga inorwisa mabhakitiriya.
#Bacitracin
#First-generation cephalosporins (e.g. Cefradine)
#Bacterial culture
#Third-generation cephalosporins (e.g. Cefditoren Pivoxil)