Syphilis
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Chancres ― chirwere che syphilis chekutanga chinokonzerwa neTreponema pallidum
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ReferencesSyphilis chirwere chepabonde chinokonzerwa nehutachiona hweTreponema pallidum. Muchidzidzo ichi, tinotarisa varwere makumi mashanu nevanomwe vane makore 18–68 vane syphilis yechipiri.
Venereal syphilis is a multi-stage, sexually transmitted disease caused by the spirochetal bacterium Treponema pallidum (Tp). Herein we describe a cohort of 57 patients (age 18-68 years) with secondary syphilis (SS) identified through a network of public sector primary health care providers in Cali, Colombia.
Venereal syphilis is a multi-stage, sexually transmitted disease caused by the spirochetal bacterium Treponema pallidum (Tp). Herein we describe a cohort of 57 patients (age 18-68 years) with secondary syphilis (SS) identified through a network of public sector primary health care providers in Cali, Colombia.
Syphilis chirwere chebhakitiriya chinokonzerwa neTreponema pallidum. Inogona kutevedzera zvirwere zvakasiyana-siyana, ichinzi great imitator. Syphilis ichiri kukanganisa mamiriyoni evanhu pasi rose, asi inogona kurapwa nemazvo nepenicillin.
Syphilis is a systemic bacterial infection caused by the spirochete Treponema pallidum. Due to its many protean clinical manifestations, it has been named the “great imitator and mimicker.” Syphilis remains a contemporary plague that continues to afflict millions of people worldwide. Luckily, the causative organism is still sensitive to penicillin.
Syphilis is a systemic bacterial infection caused by the spirochete Treponema pallidum. Due to its many protean clinical manifestations, it has been named the “great imitator and mimicker.” Syphilis remains a contemporary plague that continues to afflict millions of people worldwide. Luckily, the causative organism is still sensitive to penicillin.
Treponema pallidum inokonzeresa syphilis kuburikidza nekusangana pabonde kana kubva kuna amai kuenda kumwana panguva yekuzvitakura. Kunyangwe paine ongororo dzakareruka dzekuiongorora uye kurapwa ne‑dose imwe ye‑penicillin yenguva refu inoshanda nemazvo, syphilis iri kuita dambudziko guru pasi rose. Izvi ndezvechokwadi kunyanya pakati pevarume vanobata zvepabonde nevarume (MSM) munyika dzine mari yepamusoro neyakati. Nepo dzimwe nyika dzine hupfumi hwepasi dzakazadzikisa zvinangwa zveWHO zvekumisa kubuda kwenjova kubva kuna amai ichienda kumwana, pane kuwedzera kunoshungurudza kwenjova pakati peMSM vane HIV.
Treponema pallidum subspecies pallidum (T. pallidum) causes syphilis via sexual exposure or via vertical transmission during pregnancy. Despite the availability of simple diagnostic tests and the effectiveness of treatment with a single dose of long-acting penicillin, syphilis is re-emerging as a global public health problem, particularly among men who have sex with men (MSM) in high-income and middle-income countries. Although several low-income countries have achieved WHO targets for the elimination of congenital syphilis, an alarming increase in the prevalence of syphilis in HIV-infected MSM serves as a strong reminder of the tenacity of T. pallidum as a pathogen.
Treponema pallidum subspecies pallidum (T. pallidum) causes syphilis via sexual exposure or via vertical transmission during pregnancy. Despite the availability of simple diagnostic tests and the effectiveness of treatment with a single dose of long-acting penicillin, syphilis is re-emerging as a global public health problem, particularly among men who have sex with men (MSM) in high-income and middle-income countries. Although several low-income countries have achieved WHO targets for the elimination of congenital syphilis, an alarming increase in the prevalence of syphilis in HIV-infected MSM serves as a strong reminder of the tenacity of T. pallidum as a pathogen.
(1) Chiropa chakawedzera: Izvi zvinowanzowanikwa uye zvinogona kuitika pamwe chete ne spleen yakakura. Kuongorora chiropa ne biopsy pasi perima microscope kunogona kuratidza kuvapo kwe‑spirochete. Zviyedzo zvekushanda kwechiropa zvinogona kuratidza zvisiri izvo. (2) Yero yeganda (Jaundice): Kuti munhu anoratidza jaundice zvinoenderana nekuti chiropa chabatwa zvakadii. (3) Runny nose: Kazhinji chimwe chezviratidzo zvekutanga, chinowanikwa mukati mevhiki yekutanga mushure mekuberekwa. (4) Kuzvimba kwema lymph nodes: Kuzvimba kwakawanda kwema lymph nodes, kazhinji kusingarwadzi, kunowanzoitika. (5) Rash yeganda: Rash inowanzoitika vhiki imwe kusvika maviri mushure mekufamba kwemhino. Unogona kuona madiki madiki kana mapundu emavara kumashure, pamabvi, pazvidya, uye pasi petsoka. Kuputika uku kunogona kufambira mberi kusvika pakupfava uye kuputika.
(1) Hepatomegaly: This is the most common finding and may occur with splenomegaly. Biopsy of the liver followed by darkfield microscopy may reveal the spirochete. Liver function tests may be abnormal. (2) Jaundice: Jaundice may or may not be present depending on the extent of liver injury. (3) Rhinitis: One of the first clinical presentations, usually in the first week of life. Copious, persistent white discharge is noted, which contains spirochetes that can be visualized under darkfield microscopy. (4) Generalized Lymphadenopathy: Generalized, non-tender lymphadenopathy is also a common finding. (5) Rash: Rash usually appears one to two weeks after rhinitis. Small red or pink colored maculopapular lesions may be commonly seen on the back, buttocks, posterior thigh and soles of the feet. The rash progresses to desquamation and crusting.
(1) Hepatomegaly: This is the most common finding and may occur with splenomegaly. Biopsy of the liver followed by darkfield microscopy may reveal the spirochete. Liver function tests may be abnormal. (2) Jaundice: Jaundice may or may not be present depending on the extent of liver injury. (3) Rhinitis: One of the first clinical presentations, usually in the first week of life. Copious, persistent white discharge is noted, which contains spirochetes that can be visualized under darkfield microscopy. (4) Generalized Lymphadenopathy: Generalized, non-tender lymphadenopathy is also a common finding. (5) Rash: Rash usually appears one to two weeks after rhinitis. Small red or pink colored maculopapular lesions may be commonly seen on the back, buttocks, posterior thigh and soles of the feet. The rash progresses to desquamation and crusting.
○ Kuongororwa uye Kurapwa
VDRL neRPR zvinogona kushandiswa kusimbisa hutachiona huchangobva kuitika uye sekirini yesyphilis. FTA‑ABS bvunzo ndiyo inonyanya kushandiswa uye inogona kushandiswa kutarisa nhoroondo yehutachiona yapfuura. Penicillin inoshandiswa kurapa syphilis.