Syphilis
☆ Mune 2022 Stiftung Warentest mhedzisiro kubva kuGermany, kugutsikana kwevatengi neModelDerm kwakangodzikira zvishoma pane nekubhadharwa kwe telemedicine kubvunzana.
Chancres ― primary syphilitic infection neTreponema pallidum
References
Syphilis chirwere chepabonde chinokonzerwa neutachiona hweTreponema pallidum. Muchidzidzo ichi, tinotarisa kuvarwere makumi mashanu nevanomwe vane makore 18-68 vane syphilis yechipiri.
Venereal syphilis is a multi-stage, sexually transmitted disease caused by the spirochetal bacterium Treponema pallidum (Tp). Herein we describe a cohort of 57 patients (age 18-68 years) with secondary syphilis (SS) identified through a network of public sector primary health care providers in Cali, Colombia.
Venereal syphilis is a multi-stage, sexually transmitted disease caused by the spirochetal bacterium Treponema pallidum (Tp). Herein we describe a cohort of 57 patients (age 18-68 years) with secondary syphilis (SS) identified through a network of public sector primary health care providers in Cali, Colombia.
Syphilis chirwere chebhakitiriya chinokonzerwa neTreponema pallidum. Inogona kutevedzera zvirwere zvakasiyana-siyana, ichiwana zita remadunhurirwa great imitator. Syphilis ichiri kubata mamirioni pasi rose asi inogona kurapwa nemazvo nepenicillin.
Syphilis is a systemic bacterial infection caused by the spirochete Treponema pallidum. Due to its many protean clinical manifestations, it has been named the “great imitator and mimicker.” Syphilis remains a contemporary plague that continues to afflict millions of people worldwide. Luckily, the causative organism is still sensitive to penicillin.
Syphilis is a systemic bacterial infection caused by the spirochete Treponema pallidum. Due to its many protean clinical manifestations, it has been named the “great imitator and mimicker.” Syphilis remains a contemporary plague that continues to afflict millions of people worldwide. Luckily, the causative organism is still sensitive to penicillin.
Treponema pallidum inokonzeresa syphilis kuburikidza nekusangana pabonde kana kubva kuna amai kuenda kumwana panguva yekuzvitakura. Kunyangwe paine ongororo dzakareruka dzekuiongorora uye kurapwa nepfuti imwe yepenicillin yenguva refu inoshanda nemazvo, syphilis iri kuita dambudziko guru zvakare pasi rose. Izvi ndezvechokwadi kunyanya pakati pevarume vanoita zvepabonde nevarume (MSM) munyika dzine mari yepamusoro uye yepakati. Nepo dzimwe nyika dzine hupfumi hwepasi dzakazadzikisa zvinangwa zveWHO zvekumisa kubuda kwenjovhera kubva kuna amai ichienda kumwana, pane kuwedzera kuri kushungurudza kwenjovhera pakati peMSM vane HIV.
Treponema pallidum subspecies pallidum (T. pallidum) causes syphilis via sexual exposure or via vertical transmission during pregnancy. Despite the availability of simple diagnostic tests and the effectiveness of treatment with a single dose of long-acting penicillin, syphilis is re-emerging as a global public health problem, particularly among men who have sex with men (MSM) in high-income and middle-income countries. Although several low-income countries have achieved WHO targets for the elimination of congenital syphilis, an alarming increase in the prevalence of syphilis in HIV-infected MSM serves as a strong reminder of the tenacity of T. pallidum as a pathogen.
Treponema pallidum subspecies pallidum (T. pallidum) causes syphilis via sexual exposure or via vertical transmission during pregnancy. Despite the availability of simple diagnostic tests and the effectiveness of treatment with a single dose of long-acting penicillin, syphilis is re-emerging as a global public health problem, particularly among men who have sex with men (MSM) in high-income and middle-income countries. Although several low-income countries have achieved WHO targets for the elimination of congenital syphilis, an alarming increase in the prevalence of syphilis in HIV-infected MSM serves as a strong reminder of the tenacity of T. pallidum as a pathogen.
(1) Chiropa Chakawedzera: Izvi zvinowanzowanikwa uye zvinogona kuitika pamwe chete nespleen yakakura. Kuongorora chiropa biopsy pasi perima microscope inogona kuratidza kuvapo kwe spirochete. Zviyedzo zvekushanda kwechiropa zvinogona kuratidza zvisirizvo. (2) Yero YeGanda (Jaundice) : Kuti munhu anoratidza jaundice zvinoenderana nekuti chiropa chabatwa zvakadii. (3) Runny Nose: Kazhinji chimwe chezviratidzo zvekutanga, kazhinji mukati mevhiki yekutanga mushure mokuberekwa. (4) Kuzvimba Lymph Nodes: Kuzvimba kwakawanda kwemalymph nodes, kazhinji kusingarwadzi, kunowanzoitika. (5) Ganda Rash: Rash inowanzoitika vhiki imwe kusvika maviri mushure mekumhanya kwemhino. Iwe unogona kuona madiki madiki kana epingi mavara kumashure, matako, zvidya, uye pasi petsoka. Uku kuputika kunogona kufambira mberi kusvika pakupenengura uye kuputika.
(1) Hepatomegaly: This is the most common finding and may occur with splenomegaly. Biopsy of the liver followed by darkfield microscopy may reveal the spirochete. Liver function tests may be abnormal. (2) Jaundice: Jaundice may or may not be present depending on the extent of liver injury. (3) Rhinitis: One of the first clinical presentations, usually in the first week of life. Copious, persistent white discharge is noted, which contains spirochetes that can be visualized under darkfield microscopy. (4) Generalized Lymphadenopathy: Generalized, non-tender lymphadenopathy is also a common finding. (5) Rash: Rash usually appears one to two weeks after rhinitis. Small red or pink colored maculopapular lesions may be commonly seen on the back, buttocks, posterior thigh and soles of the feet. The rash progresses to desquamation and crusting.
(1) Hepatomegaly: This is the most common finding and may occur with splenomegaly. Biopsy of the liver followed by darkfield microscopy may reveal the spirochete. Liver function tests may be abnormal. (2) Jaundice: Jaundice may or may not be present depending on the extent of liver injury. (3) Rhinitis: One of the first clinical presentations, usually in the first week of life. Copious, persistent white discharge is noted, which contains spirochetes that can be visualized under darkfield microscopy. (4) Generalized Lymphadenopathy: Generalized, non-tender lymphadenopathy is also a common finding. (5) Rash: Rash usually appears one to two weeks after rhinitis. Small red or pink colored maculopapular lesions may be commonly seen on the back, buttocks, posterior thigh and soles of the feet. The rash progresses to desquamation and crusting.
○ Kuongororwa uye Kurapwa
VDRL neRPR inogona kushandiswa kusimbisa hutachiona huchangobva kuitika uye skrini yesyphilis. FTA-ABS bvunzo ndeyakanyanya kuyedza uye inogona kushandiswa kutarisa nhoroondo yehutachiona yapfuura. Penicillin inoshandiswa kurapa syphilis.