Mamwe mashoko ― Shona

Of the 61 patients studied, hypersensitivity vasculitis (HSV) [23 (37.7%)] and Henoch Schonlein purpura (HSP) [16 (26.2%)] were the two most common forms. Systemic involvement was seen in 32 (52.45%) patients. Drugs were implicated in 12 (19.7%) cases, infections in 7 (11.4%) and connective tissue disorders in 4 (6.5%) cases. No association was seen between history of drug intake and tissue eosinophilia and also between histologically severe vasculitis and clinical severity.

Leukocytoclastic vasculitis imhando yekuzvimba kweganda kunobata tsinga diki dzeropa dziri pakadzika peganda. Zvinogona kuitika pasina chikonzero chinozivikanwa kana kubatanidzwa nehutachiona, tumors, autoimmune zvirwere, kana mishonga. Zviratidzo zvinowanzobatanidza mavara matsvuku kana epepuru pamakumbo, kubatanidzwa kwemidziyo miduku, uye mune inenge 30 muzana yezviitiko, dzimwe nhengo dzemuviri dzinokanganiswawo. Zviitiko zvakawanda zvinopera zvega mukati memavhiki mashoma kusvika kumwedzi. Kurapa kunosiyana zvichienderana nekuoma kwazvakaita, kubva zvishoma nezvishoma kuderedza oral corticosteroids kusvika kushandisa mamwe madhiragi anoderedza kuzvimba pasina steroid.
Leukocytoclastic vasculitis is a cutaneous, small-vessel vasculitis of the dermal capillaries and venules. This condition can be idiopathic or can be associated with infections, neoplasms, autoimmune disorders, and drugs. Key clinical features of leukocytoclastic vasculitis include palpable purpura on the lower extremity, small vessel involvement, and, in about 30 percent of individuals, extracutaneous involvement. Most cases of idiopathic cutaneous, small vessel vasculitis are self-limited with 90 percent of cases resolving in weeks to months of onset. Otherwise, treatment depends on the severity of disease and can range from an oral corticosteroid taper to various steroid-sparing immunosuppressive agents.