Postinflammatory hyperpigmentation
Postinflammatory hyperpigmentation waa xaalad maqaarka ah oo lagu garto midabka maqaarka oo kordhay ka dib bararka maqaarka. Postinflammatory hyperpigmentation waxa sababi kara soo-gaadhista qorraxda dheer, bararka, ama dhaawacyo kale oo maqaarka ah, oo ay ku jiraan kuwa la xidhiidha finanka. Dadka leh midabada madaw waxay inta badan aad ugu nugul yihiin midabka midabka leh oo leh soo-gaadhista cadceedda ee xad-dhaafka ah.
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References
Postinflammatory hyperpigmentation (PIH) waa dhibaato maqaarka oo soo noqnoqota oo dhacda kadib caabuq maqaarka ama dhaawac. Waxay u egtahay inay sii socoto waqti dheer waxayna aad uga xun tahay dadka leh midab madow (Fitzpatrick skin types III–VI) . Inkasta oo ay inta badan iskeed u soo roonaato, tani waxay qaadan kartaa wakhti, sidaas darteed daawaynta ayaa badanaa loo baahan yahay wakhti dheer. Isku darka daaweynta kala duwan ayaa si fiican u shaqeysa.
Postinflammatory hyperpigmentation (PIH) is a common acquired cutaneous disorder occurring after skin inflammation or injury. It is chronic and is more common and severe in darker-skinned individuals (Fitzpatrick skin types III–VI). While the condition typically improves spontaneously, this process can take months to years, necessitating prolonged treatment. Combination therapy is the most effective.
Postinflammatory hyperpigmentation (PIH) is a common acquired cutaneous disorder occurring after skin inflammation or injury. It is chronic and is more common and severe in darker-skinned individuals (Fitzpatrick skin types III–VI). While the condition typically improves spontaneously, this process can take months to years, necessitating prolonged treatment. Combination therapy is the most effective.
Postinflammatory hyperpigmentation waa daba-galka caadiga ah ee caabuqa maqaarka. Waxay u egtahay inay si daran oo joogto ah u saamayso shakhsiyaadka maqaarka madow. Daraasaduhu waxay muujinayaan in arrimaha sida postinflammatory hyperpigmentation ay ka mid yihiin sababaha ugu waaweyn ee dadka midabada madaw ay u raadsadaan daryeelka maqaarka. Daawaynta hore waxay muhiim u tahay xalinta postinflammatory hyperpigmentation waxayna badanaa ku bilaabataa maaraynta xaalada caabuqa ee bilowga ah. Daawaynta kowaad waxay caadi ahaan ku lug leedahay isticmaalka walxaha jirka la mariyo ee fududeeya maqaarka oo ay weheliyaan muraayadaha qorraxda si looga hortago. Wakiiladan, sida hydroquinone, azelaic acid, kojic acid, arbutin, licorice extracts , waxay si wax ku ool ah u yareeyaan midabaynta xad dhaafka ah. Intaa waxaa dheer, retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, soy sidoo kale waxaa loo isticmaalaa sidii wakiilada midabaynta, oo ay soo baxayaan daaweyno cusub. Iyadoo daawaynta la mariyo ay caadi ahaan waxtar u leedahay heerka midabka sare ee midabka, hababka (laser, chemical peel) ayaa laga yaabaa inay lagama maarmaan u noqoto kiisaska madax-adayg. Waxaa muhiim ah in si taxadar leh loo isticmaalo daawayntan si looga fogaado xanaaqa iyo ka sii dara postinflammatory hyperpigmentation.
Postinflammatory hyperpigmentation is a common sequelae of inflammatory dermatoses that tends to affect darker skinned patients with greater frequency and severity. Epidemiological studies show that dyschromias, including postinflammatory hyperpigmentation, are among the most common reasons darker racial/ethnic groups seek the care of a dermatologist. The treatment of postinflammatory hyperpigmentation should be started early to help hasten its resolution and begins with management of the initial inflammatory condition. First-line therapy typically consists of topical depigmenting agents in addition to photoprotection including a sunscreen. Topical tyrosinase inhibitors, such as hydroquinone, azelaic acid, kojic acid, arbutin, and certain licorice extracts, can effectively lighten areas of hypermelanosis. Other depigmenting agents include retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, and soy with a number of emerging therapies on the horizon. Topical therapy is typically effective for epidermal postinflammatory hyperpigmentation; however, certain procedures, such as chemical peeling and laser therapy, may help treat recalcitrant hyperpigmentation. It is also important to use caution with all of the above treatments to prevent irritation and worsening of postinflammatory hyperpigmentation.
Postinflammatory hyperpigmentation is a common sequelae of inflammatory dermatoses that tends to affect darker skinned patients with greater frequency and severity. Epidemiological studies show that dyschromias, including postinflammatory hyperpigmentation, are among the most common reasons darker racial/ethnic groups seek the care of a dermatologist. The treatment of postinflammatory hyperpigmentation should be started early to help hasten its resolution and begins with management of the initial inflammatory condition. First-line therapy typically consists of topical depigmenting agents in addition to photoprotection including a sunscreen. Topical tyrosinase inhibitors, such as hydroquinone, azelaic acid, kojic acid, arbutin, and certain licorice extracts, can effectively lighten areas of hypermelanosis. Other depigmenting agents include retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, and soy with a number of emerging therapies on the horizon. Topical therapy is typically effective for epidermal postinflammatory hyperpigmentation; however, certain procedures, such as chemical peeling and laser therapy, may help treat recalcitrant hyperpigmentation. It is also important to use caution with all of the above treatments to prevent irritation and worsening of postinflammatory hyperpigmentation.
