Amyloidosis
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Fitur raray klasik tina Amyloidosis
References
Hiji awéwé 26 taun datang ka klinik kami complaining ngeunaan hiji baruntus itchy dina suku nya nu manehna kukituna ngalaman keur 10 taun. Sanajan ngagunakeun krim stéroid jeung krim tazarotene, baruntus teu meunang hadé. Anjeunna henteu ngagaduhan riwayat kulawarga anu relevan. Nalika urang nalungtik dirina, urang kapanggih diangkat, patch kasar dina hareupeun suku nya, nu cocog kasakit disebut lichen amyloidosis.
A 26-year-old woman presented to our clinic with an itchy rash on her legs that had persisted for 10 years. The rash had previously been treated with topical steroids and tazarotene cream, with no improvement. The patient’s family history was noncontributory. A physical examination showed discrete and coalescing hyperkeratotic tan-brown papules on the pretibial surfaces, consistent with lichen amyloidosis.
A 26-year-old woman presented to our clinic with an itchy rash on her legs that had persisted for 10 years. The rash had previously been treated with topical steroids and tazarotene cream, with no improvement. The patient’s family history was noncontributory. A physical examination showed discrete and coalescing hyperkeratotic tan-brown papules on the pretibial surfaces, consistent with lichen amyloidosis.
Lichen Amyloidosis mangrupikeun kaayaan kulit anu jarang dihubungkeun sareng gatal-gatal anu terus-terusan sabab anu teu dipikanyaho. Biasana muncul salaku gugusan bintik-bintik anu digedékeun, discolored dina permukaan luar kulit. Lichen Amyloidosis biasana muncul dina jalma yuswa 50 dugi ka 60 sareng hanjakalna, teu aya ubar pikeun éta. Pangobatan anu sayogi ayeuna biasana henteu tiasa dianggo saé.
Lichen Amyloidosis (LA) is an uncommon, primary cutaneous amyloidosis associated with chronic, idiopathic pruritus. Clinical presentation of LA includes skin colored to hyperpigmented, papules coalescing into plaques with a rippled appearance on the extensors.1 LA most commonly presents in the fifth to sixth decade of life and has no curative treatments. Overall response to current therapies is poor.
Lichen Amyloidosis (LA) is an uncommon, primary cutaneous amyloidosis associated with chronic, idiopathic pruritus. Clinical presentation of LA includes skin colored to hyperpigmented, papules coalescing into plaques with a rippled appearance on the extensors.1 LA most commonly presents in the fifth to sixth decade of life and has no curative treatments. Overall response to current therapies is poor.
Lichen amyloidosis mangrupikeun kaayaan kulit gatal anu berkepanjangan. Ieu dipikawanoh pikeun klaster nabrak thickened utamana kapanggih dina tonggong, tulang tonggong, pingping, jeung leungeun. Nalika ditalungtik dina mikroskop, Lichen amyloidosis nunjukkeun akumulasi amiloid dina lapisan luhur kulit sareng penebalan sareng pembesaran lapisan kulit luar. Sanaos panyabab pasti tina Lichen amyloidosis teu acan kahartos sapinuhna, panilitian sateuacana parantos ngaitkeun kana faktor sapertos gosok atanapi gesekan dina kulit, maot sél, inféksi virus, sareng anu sanésna. Lichen amyloidosis sigana dikaitkeun sareng sababaraha kaayaan kulit sanés (atopic dermatitis, lichen planus, mycosis fungoides) .
Lichen amyloidosis (LA) is a chronic pruritic skin disorder characterized by multiple grouped hyperkeratotic papules, predominantly located on the back, shins, thighs, and arms. Histological analysis of LA shows amyloid deposition in the papillary dermis and hyperkeratosis and acanthosis of the epidermis. The exact pathogenesis of LA has not yet been elucidated; however, prior reports have implicated frictional epidermal damage, apoptosis, viral infection, and many other triggers. LA is reportedly associated with several skin disorders, including atopic dermatitis (AD), lichen planus, and mycosis fungoides.
Lichen amyloidosis (LA) is a chronic pruritic skin disorder characterized by multiple grouped hyperkeratotic papules, predominantly located on the back, shins, thighs, and arms. Histological analysis of LA shows amyloid deposition in the papillary dermis and hyperkeratosis and acanthosis of the epidermis. The exact pathogenesis of LA has not yet been elucidated; however, prior reports have implicated frictional epidermal damage, apoptosis, viral infection, and many other triggers. LA is reportedly associated with several skin disorders, including atopic dermatitis (AD), lichen planus, and mycosis fungoides.
○ Diagnosis sareng Perawatan
#Electrophoresis of blood or urine
#Skin biopsy