Psoriasishttps://en.wikipedia.org/wiki/Psoriasis
Psoriasis mangrupikeun panyakit autoimun anu kronis, henteu inféksi, anu dicirikeun ku lesi kulit anu teu normal. Wewengkon ieu beureum, atawa wungu dina sababaraha jalma, sareng kulit langkung poék, garing, gatel, sarta bersisik. Tatu kana kulit bisa memicu parobahan kulit psoriatic di titik éta, nu katelah “fenomena Koebner”.

Rupa‑rupa pangobatan tiasa ngabantosan ngontrol gejala. Pangobatan ieu kalebet krim steroid, krim vitamin D3, sinar ultraviolét, sareng obat imunosupresif, sapertos methotrexate. Sakitar 75 % tina keterlibatan kulit ningkat ku krim sorangan. Rupa‑rupa agen imunologis biologis geus dimekarkeun pikeun pengobatan psoriasis.

Psoriasis mangrupikeun gangguan umum anu mangaruhan 2‑4 % populasi. Lalaki sareng awéwé kapangaruhan ku frékuénsi anu sami. Panyakit ieu tiasa muncul dina sagala umur, tapi ilaharna dimimitian dina dewasa. Psoriatic arthritis mangaruhan nepi ka 30 % jalma kalayan psoriasis.

○ Perawatan – Obat OTC
Cahaya panonpoé tiasa ngabantosan psoriasis sabab paparan panonpoé ngabalukarkeun parobahan imunologis dina penderita psoriasis. Salep hydrocortisone hampang tiasa ngabantosan pikeun ngubaran sababaraha lesi leutik psoriasis.
#OTC steroid ointment

○ Perawatan
Psoriasis mangrupikeun panyakit kronis sareng aya seueur agen pangobatan anu dipaké. Agen biologis paling efektif, tapi mahal pisan.
#High potency steroid ointment
#Calcipotriol cream
#Phototherapy
#Biologics (e.g. infliximab, adalimumab, secukinumab, ustekinumab)
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  • Deui jeung leungeun hiji jalma anu ngidap psoriasis
  • Psoriasis has
  • Guttate Psoriasis: ieu sering lumangsung sanggeus gejala tiis.
  • Guttate Psoriasis
  • Plak sisik kandel jeung erythema téh mangrupa manifestasi khas psoriasis.
  • Psoriasis dina palém. Upami éta lumangsung dina dampal leungeun, bisul tiasa kabentuk.
  • Parah psoriasis pustular.
  • Guttate Psoriasis (Psoriasis Guttate)
References Psoriasis 28846344 
NIH
 Phototherapy 33085287 
NIH
 Tumor Necrosis Factor Inhibitors 29494032 
NIH
Tumor necrosis factor (TNF)-alpha inhibitors, including etanercept (E), infliximab (I), adalimumab (A), certolizumab pegol (C), and golimumab (G), are biologic agents which are FDA-approved to treat ankylosing spondylitis (E, I, A, C, and G), Crohn disease (I, A and C), hidradenitis suppurativa (A), juvenile idiopathic arthritis (A), plaque psoriasis (E, I and A), polyarticular juvenile idiopathic arthritis (E), psoriatic arthritis (E, I, A, C, and G), rheumatoid arthritis (E, I, A, C, and G), ulcerative colitis (I, A and G), and uveitis (A).