Cellulitishttps://yo.wikipedia.org/wiki/Cellulitis
Cellulitis jẹ akóràn kokoro‑àrùn tí ó kan àwọn ìpele inú ti awọ ara. Gẹ́gẹ́ bí àmi àìsàn, agbègbè tí ó ń rọ̀rùn àti pupa ń pọ̀ sí i ní ọ̀pọ̀ ọjọ́ díẹ̀. Ààlà agbègbè pupa kò ṣe kedere, ó sì lè di wiwu. Agbègbè ikọlu máa ń ní irora. Ẹniyan lè ní ibà àti myalgia.

Ẹsẹ àti ojú ni àwọn agbègbè tí ó wọ́pọ̀ jùlọ tí ó kan. Àwọn àǹfààní ewu ní í ṣe pẹ̀lú isanraju, wiwu ẹsẹ, àti ọjọ‑ori. Àwọn kokoro arun tí ó wọ́pọ̀ jùlọ ni streptococci àti Staphylococcus aureus.

Itọju máa ń jẹ́ pẹ̀lú àwọn egboogi tí a ń mu ní ẹnu, bíi cephalexin, amoxicillin tàbí cloxacillin. Ní àìmọ̀ 95 % àwọn ènìyàn ń gba imularada lẹ́yìn ọjọ́ 7 sí 10 tí wọ́n ti ń gba itọju. Àwọn tó ní àtọgbẹ, ṣùgbọ́n, máa ń ní àbájáde tó burú jùlọ.

Cellulitis jẹ́ arun tí ó wọ́pọ̀, ó sì jẹ́ ìdí fún 1.6 % ti ìbáwọ̀lé sí ilé‑iwosan ní United Kingdom. Ní ọdún 2015, cellulitis yọrí sí ikú tó tó 16,900 ní agbáyé.

Itọju – Oògùn OTC
Cellulitis tó ti ń gòkè lè ní ìtòsí pẹ̀lú àwọn oogun aporo tí dókítà pàṣẹ nìkan. Bí àrùn náà bá ń yára sí i, tí ibà àti ìtúra bá wáyé, ó dájú kí o kan sí dókítà ní kánkán.
Lílo ohun èlò àìlera lórí‑ìtòsí lè rànlọwọ. Bí epo àìlera bá rọ̀rùn jù, ó lè má ṣiṣẹ́.
#Polysporin
#Bacitracin
#Betadine

Lo àwọn olùtìràn‑ìrora OTC bí acetaminophen láti dín ìrora kù.
#Ibuprofen
#Naproxen
#Acetaminophen

Ṣe àtìlẹ́yìn kí ẹsẹ má bàjẹ́, kí o sì tọju àìlera ẹsẹ, nítorí pé àìlera ẹsẹ ń pọ̀ sí i eewu cellulitis.

Itọju
#First‑generation cephalosporins (e.g. Cefradine)
#Bacterial culture
#Third‑generation cephalosporins (e.g. Cefditoren Pivoxil)
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  • Ẹsẹ jẹ́ ibi tí ó wọ́pọ̀ fún Cellulitis.
  • No Yoruba text was provided for correction. Please supply the material you would like edited.
  • Cellulitis – Ipele Iwosan
  • Cellulitis lori ẹsẹ osi. Ti o ba tan kaakiri, ìbà gíga le waye, tí yóò sì yọrí sí àìlera tó lewu.
  • Nigbati ọgbẹ naa ba ń yọ̀, hyperpigmentation lẹ́yìn iredodo le hàn.
  • Ó lè ṣẹlẹ̀ nípasẹ̀ egbo lórí ọpẹ.
  • Aṣoju ọ̀ràn
  • Ti alemo erythematous tutu ba tan kaakiri, o yẹ ki o fura si Cellulitis.
  • Bi Cellulitis ṣe n dara si, awọn sẹẹli awọ ara ti o ku le yọ kuro, ti o sì le di yun.
  • Pupọ̀ Cellulitis wà pẹ̀lú wíwú àti erythema ní agbègbè tó kan. Ṣùgbọ́n, nínú ọ̀ràn aláìlẹ̀gbẹ́ yìí tí kò ní erythema agbègbè, carcinoma cell squamous yẹ kí a tún fura sí.
  • Aṣoju ọ̀ràn
  • iwosan alakoso.
References Cellulitis 31747177 
NIH
Cellulitis jẹ akoran awọ ara ti o fa nipasẹ kokoro arun ti o wọpọ. O kan eniyan miliọnu 14 lọdọọdun ni Amẹrika. O nyorisi si bii $3.7 bilionu ni awọn idiyele itọju alaisan ati awọn ile‑iwosan to 650,000 ni ọdun kọọkan. Ni deede, cellulitis maa n farahan gẹgẹbi igbona, agbegbe pupa lori awọ ara pẹlu wiwu ati rirọ. O ṣẹlẹ nipasẹ ikolu kokoro‑arun lojiji ti o nfa igbona ninu awọn ipele awọ‑ara ti o jinlẹ ati àkúnya to wa nitosi. Ko si àkúnya tàbí pus pẹlu ikolu yii. Beta‑hemolytic streptococci, paapaa ẹgbẹ A streptococcus (Streptococcus pyogenes), jẹ awọn ọlọjẹ deede, ti a tún le ri methicillin‑sensitive Staphylococcus aureus.
Cellulitis is a common bacterial skin infection, with over 14 million cases occurring in the United States annually. It accounts for approximately 3.7 billion dollars in ambulatory care costs and 650000 hospitalizations annually. Cellulitis typically presents as a poorly demarcated, warm, erythematous area with associated edema and tenderness to palpation. It is an acute bacterial infection causing inflammation of the deep dermis and surrounding subcutaneous tissue. The infection is without an abscess or purulent discharge. Beta-hemolytic streptococci typically cause cellulitis, generally group A streptococcus (i.e., Streptococcus pyogenes), followed by methicillin-sensitive Staphylococcus aureus. Patients who are immunocompromised, colonized with methicillin-resistant Staphylococcus aureus, bitten by animals, or have comorbidities such as diabetes mellitus may become infected with other bacteria.
 Cellulitis: current insights into pathophysiology and clinical management 29219814
Cellulitis jẹ́ àkóràn kokoro arun tó ń kan awọ ara àti àwọn tísọ̀ rírò. Ó ṣẹlẹ̀ nígbà tí àwọn ìpò bá wà pẹ̀lú ìdènà ààbò awọ ara, ètò àjẹ́ṣàra, tàbí sísàn ẹ̀jẹ̀. Àtọgbẹ, isanraju, àti ọjọ́-ọdún ń pọ̀ sí i àǹfààní cellulitis nípa fífi ipa kàn àwọn àgbègbè wọ̀nyí. A tún wo àwọn àwárí àìpẹ̀ nípa ìwádìí cellulitis, tí ń ṣàfihàn pàtàkì ìwádìí àìsàn tó péye níwọ̀n bí àwọn ìpò bí aipe iṣọn-ẹ̀jẹ̀, àléfọ, thrombosis iṣọn jinlẹ̀, àti gout ṣe máa ń dákẹ́ àìlera pẹ̀lú cellulitis. Àwọn ègboogi tí a lò láti tọju cellulitis ni a yàn pẹ̀kipẹki láti dojú kọ́ àwọn kokoro arun tó wọ́pọ̀ láì fà àìlera àìlera àpòro. A tún sọ̀rọ̀ nípa àwọn ègboogi tuntun tí a fọwọ́sí fún cellulitis. Cellulitis máa ń tún padà wá nígbà tí àwọn àǹfààní eewu ń tẹ̀síwájú àti ibajẹ sí eto lymphatic.
Cellulitis is a bacterial infection of the skin and soft tissues. It happens when there are issues with the skin's protective barrier, the immune system, or blood circulation. Diabetes, obesity, and old age increase the chances of cellulitis by affecting these areas. We also look at recent findings on diagnosing cellulitis, highlighting the importance of accurate diagnosis since conditions like venous insufficiency, eczema, deep vein thrombosis, and gout are often confused with cellulitis. Antibiotics used to treat cellulitis are chosen carefully to target common bacteria without encouraging antibiotic resistance. We also talk about new antibiotics approved for cellulitis. Cellulitis often comes back because of ongoing risk factors and damage to the lymphatic system..
 Current Treatment Options for Acute Skin and Skin-structure Infections 30957166 
NIH
Ọ̀pọ̀ ènìyàn ń lọ sí ilé‑ìwòsàn tàbí yàrá pajawiri fún àkóràn àwọ̀ ara tó ń bọ́ láìpé. Staphylococcus aureus ni àkọ́kọ́ gérèmì tí ń fa àwọn àkóràn wọ̀nyí, ó sì ń ṣòro láti tọju nítorí pé díẹ̀ lára àwọn ìgárá rẹ̀ ń kọ́ àwọ̀n egboogi tó wọ́pọ̀.
Acute bacterial skin and skin-structure infections are a common reason for seeking care at acute healthcare facilities, including emergency departments. Staphylococcus aureus is the most common organism associated with these infections, and the emergence of community-associated methicillin-resistant Staphylococcus aureus (MRSA) has represented a considerable challenge in their treatment.
 Prevalence and Therapies of Antibiotic-Resistance in Staphylococcus aureus 32257966 
NIH
Staphylococcus aureus le pin si oriṣi meji, da lori bi wọn ṣe dahun si awọn oogun apakokoro: methicillin-sensitive Staphylococcus aureus (MSSA) ati methicillin-resistant Staphylococcus aureus (MRSA). Ni awọn ọdun diẹ sẹhin, itankalẹ kokoro arun ati ilokulo awọn oogun apakokoro ti fa ilodisi S. Aureus si awọn oogun, ti o yori si ilosoke agbaye ninu awọn oṣuwọn ikolu MRSA.
According to the sensitivity to antibiotic drugs, S. aureus can be divided into methicillin-sensitive Staphylococcus aureus (MSSA) and methicillin-resistant Staphylococcus aureus (MRSA). In recent decades, due to the evolution of bacteria and the abuse of antibiotics, the drug resistance of S. aureus has gradually increased, the infection rate of MRSA has increased worldwide.
 Treatment of severe skin and soft tissue infections: a review 29278528 
NIH
To review the salient features of the management of severe skin and soft tissue infections (SSTIs), including toxic shock syndrome, myonecrosis/gas gangrene, and necrotizing fasciitis.