Impetigo
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Ninu awọn abajade 2022 Stiftung Warentest lati Jẹmánì, itẹlọrun alabara pẹlu ModelDerm jẹ kekere diẹ ju pẹlu awọn ijumọsọrọ telemedicine isanwo.
Ninu awọn abajade 2022 Stiftung Warentest lati Jẹmánì, itẹlọrun alabara pẹlu ModelDerm jẹ kekere diẹ ju pẹlu awọn ijumọsọrọ telemedicine isanwo.
Impetigo jẹ́ àìlera tó ń hàn lórí awọ̀. Impetigo yẹ kí a fura sí i tí ọmọ kékeré kò bá ní ìtànjẹ́ ìpalára, ṣùgbọ́n ó máa ń hàn bí ọ̀gbẹ́ tó ń tan.
Ko dabi atopic dermatitis, impetigo nilo itọju apakokoro, ati pe o le buru si pẹlu lilo awọn sitẹriọdu.
relevance score : -100.0%
ReferencesImpetigo, akoran awọ ara kokoro arun ti o wọpọ julọ ni awọn ọmọde ti o wa ni ọdun meji si marun, wa ni awọn oriṣi akọkọ meji: nonbulous (70% awọn iṣẹlẹ) ati bullous (30% awọn iṣẹlẹ). Impetigo ti kii ṣe akọbulọsi jẹ deede nipasẹ Staphylococcus aureus tabi Streptococcus pyogenes. O jẹ idanimọ nipasẹ awọn erunrun awọ oyin lori oju ati awọn ẹsẹ, ati ni pataki ti o fojusi awọ ara, tabi o le ṣe akoran awọn buje kokoro, àléfọ, tabi awọn egbo herpetic. Bullous impetigo, ti o ṣẹlẹ nikan nipasẹ S. aureus, nyorisi si awọn bullae tobi, flaccid, tí ó sì máa kan àwọn àgbègbè tí ara rubs pọ̀. Awọn oriṣi mejeeji maa n yọkuro laarin ọsẹ meji si mẹta laisi aleebu, ati awọn ilolu jẹ ṣọwọn, pẹlu poststreptococcal glomerulonephritis tí ó lè jẹ́ àìlera tó pọ̀jù. Itoju jẹ́ pẹ̀lú àwọn egboogi ti agbegbe (mupirocin, retapamulin, fusidic acid). Awọn egboogi ti ẹnu le jẹ́ pataki fún impetigo tí ó ní bullae ńlá tàbí nígbà tí itọju agbegbe kò ṣeé ṣe. Lakọ̀ọ́kọ̀ tí ọ̀pọ̀lọpọ̀ oogun apako ẹnu (amoxicillin/clavulanate, dicloxacillin, cephalexin, clindamycin, doxycycline, minocycline, trimethoprim/sulfamethoxazole, macrolides) jẹ́ àwọn aṣàyàn, penicillin kò ní ipa. Awọn apakokoro ti agbegbe kò dára bí oogun apako, ó sì yẹ kí a yàgò fún wọn. Fusidic acid, mupirocin, retapamulin jẹ́ munadoko lodi si methicillin‑ailagbara S. aureus àti àwọn akóràn streptococcal. Clindamycin wúlò fún ifura methicillin‑resistant S. aureus àkóràn. Trimethoprim/sulfamethoxazole ṣiṣẹ́ lodi si methicillin‑sooro S. aureus, ṣùgbọ́n kò tó fún ikọlu streptococcal.
Impetigo, the most common bacterial skin infection in children aged two to five, comes in two main types: nonbullous (70% of cases) and bullous (30% of cases). Nonbullous impetigo is typically caused by Staphylococcus aureus or Streptococcus pyogenes. It's recognized by honey-colored crusts on the face and limbs and mainly targets the skin or can infect insect bites, eczema, or herpetic lesions. Bullous impetigo, caused solely by S. aureus, leads to large, flaccid bullae and often affects areas where skin rubs together. Both types usually clear up within two to three weeks without scarring, and complications are rare, with poststreptococcal glomerulonephritis being the most severe. Treatment involves topical antibiotics (mupirocin, retapamulin, fusidic acid). Oral antibiotics might be necessary for impetigo with large bullae or when topical treatment isn't feasible. While several oral antibiotics (amoxicillin/clavulanate, dicloxacillin, cephalexin, clindamycin, doxycycline, minocycline, trimethoprim/sulfamethoxazole, macrolides) are options, penicillin isn't effective. Topical disinfectants aren't as good as antibiotics and should be avoided. Fusidic acid, mupirocin, retapamulin are effective against methicillin-susceptible S. aureus and streptococcal infections. Clindamycin is useful for suspected methicillin-resistant S. aureus infections. Trimethoprim/sulfamethoxazole works against methicillin-resistant S. aureus, but isn't enough for streptococcal infection.
Impetigo, the most common bacterial skin infection in children aged two to five, comes in two main types: nonbullous (70% of cases) and bullous (30% of cases). Nonbullous impetigo is typically caused by Staphylococcus aureus or Streptococcus pyogenes. It's recognized by honey-colored crusts on the face and limbs and mainly targets the skin or can infect insect bites, eczema, or herpetic lesions. Bullous impetigo, caused solely by S. aureus, leads to large, flaccid bullae and often affects areas where skin rubs together. Both types usually clear up within two to three weeks without scarring, and complications are rare, with poststreptococcal glomerulonephritis being the most severe. Treatment involves topical antibiotics (mupirocin, retapamulin, fusidic acid). Oral antibiotics might be necessary for impetigo with large bullae or when topical treatment isn't feasible. While several oral antibiotics (amoxicillin/clavulanate, dicloxacillin, cephalexin, clindamycin, doxycycline, minocycline, trimethoprim/sulfamethoxazole, macrolides) are options, penicillin isn't effective. Topical disinfectants aren't as good as antibiotics and should be avoided. Fusidic acid, mupirocin, retapamulin are effective against methicillin-susceptible S. aureus and streptococcal infections. Clindamycin is useful for suspected methicillin-resistant S. aureus infections. Trimethoprim/sulfamethoxazole works against methicillin-resistant S. aureus, but isn't enough for streptococcal infection.
Impetigo jẹ ikolu awọ ara ti o wọpọ ti o fa nipasẹ awọn kokoro arun kan, ti o tan kaakiri nipasẹ olubasọrọ. O maa n farahan gẹgẹ bi awọn abulẹ pupa ti a bo pelu erunrun ofeefee kan, ti o le fa nyún tàbí irora. Ikolu yii wọpọ julọ ní àwọn ọmọde tí ń gbé ní àgbègbè tó gbóná, tó ní ọ̀rinrin. Ó lè hàn bí rọ́rọ̀ tàbí láìsí erun. Bí ó tilẹ̀ jẹ́ pé ó máa ń kan oju, ó lè ṣẹlẹ̀ ní ibikíbi tí ìfarapa bá wà lórí awọ ara. Ayẹwo àkọ́kọ́ dá lórí àwọn ààmì àìsàn àti bí ó ṣe ń hàn. Itọju máa ń jẹ́ pẹ̀lú àwọn egboogi, mejeeji ti agbegbe àti ti ẹnu, pẹ̀lú iṣakoso ààmì àìsàn.
Impetigo is a common infection of the superficial layers of the epidermis that is highly contagious and most commonly caused by gram-positive bacteria. It most commonly presents as erythematous plaques with a yellow crust and may be itchy or painful. The lesions are highly contagious and spread easily. Impetigo is a disease of children who reside in hot humid climates. The infection may be bullous or nonbullous. The infection typically affects the face but can also occur in any other part of the body that has an abrasion, laceration, insect bite or other trauma. Diagnosis is typically based on the symptoms and clinical manifestations alone. Treatment involves topical and oral antibiotics and symptomatic care.
Impetigo is a common infection of the superficial layers of the epidermis that is highly contagious and most commonly caused by gram-positive bacteria. It most commonly presents as erythematous plaques with a yellow crust and may be itchy or painful. The lesions are highly contagious and spread easily. Impetigo is a disease of children who reside in hot humid climates. The infection may be bullous or nonbullous. The infection typically affects the face but can also occur in any other part of the body that has an abrasion, laceration, insect bite or other trauma. Diagnosis is typically based on the symptoms and clinical manifestations alone. Treatment involves topical and oral antibiotics and symptomatic care.
Impetigo máa ńfa nípasẹ̀ Staphylococcus aureus tàbí Streptococcus pyogenes. Pẹ̀lú ìfọwọ́kàn, ó lè tan kaakiri láàárín àwọn ènìyàn. Nínú àwọn ọmọde, ó lè tan sí àwọn arákùnrin wọn.
Itọju sábà máa ń lo àwọn ohun‑èlò apakokoro bíi mupirocin tàbí fusidic acid. Àwọn egboogi tí a gba ní ẹnu, gẹ́gẹ́ bí cefalexin, lè ṣee lo tí àgbègbè tó ń kan bá tóbi.
Impetigo kan nípa 140 millionu ènìyàn (2 % ti àwọn olùgbé àgbáyé) ní ọdún 2010. Ó lè hàn ní gbogbo ọjọ‑ori, ṣùgbọ́n ó wọ́pọ̀ jùlọ láàárín àwọn ọmọde. Àwọn ilolu lè ní cellulitis tàbí poststreptococcal glomerulonephritis.
○ Itọju - Oògùn OTC
* Nítorí pé impetigo jẹ́ arun aarun, àwọn ikunra sitẹriọdu kò yẹ kí a lo. Bí o bá nira láti yàtọ̀ egbo impetigo kúrò ní àléfọ, jọ̀wọ́ lo antihistamine OTC láì lo àwọn ikunra sitẹriọdu.
#OTC antihistamine
* Jọ̀wọ́ lo ikunra aporo OTC sí ọ̀gbẹ́ náà.
#Bacitracin
#Polysporin