Poikiloderma
Poikiloderma jẹ ipo awọ ara ti o ni awọn agbegbe ti hypopigmentation, hyperpigmentation, telangiectasias ati atrophy. Poikiloderma ni a maa n rii nigbagbogbo lori àyà tabi ọrun, ti a ṣe afihan pẹlu pigmenti awọ pupa lori awọ ara ti o wọpọ pẹlu ibajẹ oorun.
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References
Poikiloderma of Civatte jẹ ipo awọ ti o wọpọ ti o han ni ọrun ati oju, paapaa ni awọ-awọ, awọn obinrin lẹhin menopause. O ṣe afihan bi apapọ awọn ila pupa, awọn aaye dudu, ati awọ tinrin. Ni deede, o kan awọn agbegbe ti o farahan si oorun, bii oju, ọrun, ati àyà, ṣugbọn kii ṣe awọn agbegbe iboji. Poikiloderma of Civatte le jẹ tito lẹšẹšẹ da lori awọn ẹya akọkọ rẹ: pupa, awọn aaye dudu, tabi apapọ awọn mejeeji. Idi gangan ko ni oye ni kikun, ṣugbọn awọn okunfa bii ifihan oorun, awọn iyipada homonu, awọn aati si awọn turari tabi awọn ohun ikunra, ati ti ogbo ni a ro pe o ṣe ipa kan. Poikiloderma of Civatte duro lati buru si laiyara lori akoko.
Poikiloderma of Civatte (PC) is a rather common benign dermatosis of the neck and face, mainly affecting fair-skinned individuals, especially postmenopausal females. It is characterized by a combination of a reticular pattern of linear telangiectasia, mottled hyperpigmentation and superficial atrophy. Clinically, it involves symmetrically sun-exposed areas of the face, the neck, and the V-shaped area of the chest, invariably sparing the anatomically shaded areas. Depending on the prevalent clinical feature, PC can be classified into erythemato-telangiectatic, pigmented, and mixed clinical types. The etiopathogenesis of PC is incompletely understood. Exposure to ultraviolet radiation, hormonal changes of menopause, contact sensitization to perfumes and cosmetics, and normal ageing have been incriminated. The diagnosis is usually clinical and can be confirmed by histology, which is characteristic, but not pathognomonic. The course is slowly progressive and irreversible, often causing significant cosmetic disfigurement.
Poikiloderma of Civatte (PC) is a rather common benign dermatosis of the neck and face, mainly affecting fair-skinned individuals, especially postmenopausal females. It is characterized by a combination of a reticular pattern of linear telangiectasia, mottled hyperpigmentation and superficial atrophy. Clinically, it involves symmetrically sun-exposed areas of the face, the neck, and the V-shaped area of the chest, invariably sparing the anatomically shaded areas. Depending on the prevalent clinical feature, PC can be classified into erythemato-telangiectatic, pigmented, and mixed clinical types. The etiopathogenesis of PC is incompletely understood. Exposure to ultraviolet radiation, hormonal changes of menopause, contact sensitization to perfumes and cosmetics, and normal ageing have been incriminated. The diagnosis is usually clinical and can be confirmed by histology, which is characteristic, but not pathognomonic. The course is slowly progressive and irreversible, often causing significant cosmetic disfigurement.
