Postinflammatory hyperpigmentation - 發炎後的皮膚色素增生https://en.wikipedia.org/wiki/Hyperpigmentation
發炎後的皮膚色素增生 (Postinflammatory hyperpigmentation) 是一種皮膚狀況,特徵為發炎後皮膚色素增多。發炎後的皮膚色素增生 (Postinflammatory hyperpigmentation) 可能由長期日曬、發炎或其他皮膚損傷(包括痤瘡相關的損傷)所致。膚色較深者通常因過度日曬更易出現色素沉著。

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      References Postinflammatory Hyperpigmentation 32644576 
      NIH
      Postinflammatory hyperpigmentation (PIH) 是皮膚發炎或受傷後常見的皮膚問題。它往往會持續很長時間,且對膚色較深的人(Fitzpatrick skin types III–VI)情況更為嚴重。雖然它通常會自行好轉,但可能需要相當長的時間,因此常需進行較長時間的治療。結合不同的治療方式可獲得最佳效果。
      Postinflammatory hyperpigmentation (PIH) is a common acquired cutaneous disorder occurring after skin inflammation or injury. It is chronic and is more common and severe in darker-skinned individuals (Fitzpatrick skin types III–VI). While the condition typically improves spontaneously, this process can take months to years, necessitating prolonged treatment. Combination therapy is the most effective.
       Postinflammatory hyperpigmentation: a review of the epidemiology, clinical features, and treatment options in skin of color 20725554 
      NIH
      Postinflammatory hyperpigmentation 是皮膚發炎常見的後遺症,對深色皮膚的人影響更嚴重且更頻繁。研究顯示,像 postinflammatory hyperpigmentation 這類問題是膚色較深者尋求皮膚科護理的主要原因之一。早期治療對於改善 postinflammatory hyperpigmentation 至關重要,通常先控制最初的發炎狀況。第一線治療多採用美白外用製劑搭配防曬霜,以保護皮膚。這些藥劑與 hydroquinone、azelaic acid、kojic acid、arbutin、licorice extracts 等同樣能有效減少過度色素沉澱。除此之外,retinoids、mequinol、ascorbic acid、niacinamide、N‑acetyl glucosamine、soy 等也常被用作脫色劑,新的治療方法不斷湧現。雖然局部治療對表面色素沉著通常有效,但對於頑固病例可能需要手術(laser、chemical peel)。因此,使用這些治療時必須謹慎,以免刺激或加重 postinflammatory hyperpigmentation。
      Postinflammatory hyperpigmentation is a common sequelae of inflammatory dermatoses that tends to affect darker skinned patients with greater frequency and severity. Epidemiological studies show that dyschromias, including postinflammatory hyperpigmentation, are among the most common reasons darker racial/ethnic groups seek the care of a dermatologist. The treatment of postinflammatory hyperpigmentation should be started early to help hasten its resolution and begins with management of the initial inflammatory condition. First-line therapy typically consists of topical depigmenting agents in addition to photoprotection including a sunscreen. Topical tyrosinase inhibitors, such as hydroquinone, azelaic acid, kojic acid, arbutin, and certain licorice extracts, can effectively lighten areas of hypermelanosis. Other depigmenting agents include retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, and soy with a number of emerging therapies on the horizon. Topical therapy is typically effective for epidermal postinflammatory hyperpigmentation; however, certain procedures, such as chemical peeling and laser therapy, may help treat recalcitrant hyperpigmentation. It is also important to use caution with all of the above treatments to prevent irritation and worsening of postinflammatory hyperpigmentation.